miR-155 regulates high glucose-induced cardiac fibrosis via the TGF-β signaling pathway.
about
Mir-155 is overexpressed in systemic sclerosis fibroblasts and is required for NLRP3 inflammasome-mediated collagen synthesis during fibrosis.miRNAs in Normal and Malignant Hematopoiesis.Glycosides from Stevia rebaudiana Bertoni Possess Insulin-Mimetic and Antioxidant Activities in Rat Cardiac FibroblastsMicroRNA as a Therapeutic Target in Cardiac Remodeling.MicroRNA-495 inhibits the high glucose-induced inflammation, differentiation and extracellular matrix accumulation of cardiac fibroblasts through downregulation of NOD1.
P2860
miR-155 regulates high glucose-induced cardiac fibrosis via the TGF-β signaling pathway.
description
2016 nî lūn-bûn
@nan
2016年の論文
@ja
2016年学术文章
@wuu
2016年学术文章
@zh-cn
2016年学术文章
@zh-hans
2016年学术文章
@zh-my
2016年学术文章
@zh-sg
2016年學術文章
@yue
2016年學術文章
@zh
2016年學術文章
@zh-hant
name
miR-155 regulates high glucose-induced cardiac fibrosis via the TGF-β signaling pathway.
@en
miR-155 regulates high glucose-induced cardiac fibrosis via the TGF-β signaling pathway.
@nl
type
label
miR-155 regulates high glucose-induced cardiac fibrosis via the TGF-β signaling pathway.
@en
miR-155 regulates high glucose-induced cardiac fibrosis via the TGF-β signaling pathway.
@nl
prefLabel
miR-155 regulates high glucose-induced cardiac fibrosis via the TGF-β signaling pathway.
@en
miR-155 regulates high glucose-induced cardiac fibrosis via the TGF-β signaling pathway.
@nl
P2093
P2860
P356
P1433
P1476
miR-155 regulates high glucose-induced cardiac fibrosis via the TGF-β signaling pathway.
@en
P2093
P2860
P304
P356
10.1039/C6MB00649C
P50
P577
2016-12-07T00:00:00Z