MiR-221 promotes cardiac hypertrophy in vitro through the modulation of p27 expression.
about
Overview of MicroRNAs in Cardiac Hypertrophy, Fibrosis, and ApoptosisHuman miR-221/222 in Physiological and Atherosclerotic Vascular RemodelingMicroRNAs in heart failure: Small molecules with major impactmiR-185 plays an anti-hypertrophic role in the heart via multiple targets in the calcium-signaling pathwaysMiR-451 is decreased in hypertrophic cardiomyopathy and regulates autophagy by targeting TSC1.MicroRNA signatures differentiate preserved from reduced ejection fraction heart failureMicroRNA-221 inhibits autophagy and promotes heart failure by modulating the p27/CDK2/mTOR axis.MiRNA inhibition in tissue engineering and regenerative medicine.The altered expression profile of microRNAs in cardiopulmonary bypass canine models and the effects of mir-499 on myocardial ischemic reperfusion injury.ANGPTL2 activity in cardiac pathologies accelerates heart failure by perturbing cardiac function and energy metabolism.Comparative mRNA and MicroRNA Profiling during Acute Myocardial Infarction Induced by Coronary Occlusion and Ablation Radio-Frequency Currents.Role of microRNAs in Vascular Remodeling.The therapeutic potential of miRNAs regulated in settings of physiological cardiac hypertrophy.Therapeutic targeting of autophagy in myocardial infarction and heart failure.miR-30a downregulation aggravates pressure overload-induced cardiomyocyte hypertrophy.MicroRNA regulation in heart and skeletal muscle over the freeze-thaw cycle in the freeze tolerant wood frog.Hydrogen sulfide alleviates myocardial fibrosis in mice with alcoholic cardiomyopathy by downregulating autophagy.Exacerbation of diabetic cardiac hypertrophy in OVE26 mice by angiotensin II is associated with JNK/c-Jun/miR-221-mediated autophagy inhibition.Identification and analysis of a key long non-coding RNAs (lncRNAs)-associated module reveal functional lncRNAs in cardiac hypertrophy.Gender and cardiovascular disease: are sex-biased microRNA networks a driving force behind heart failure with preserved ejection fraction in women?Differential microRNA expression profiles and bioinformatics analysis between young and aging spontaneously hypertensive rats.The effects of subacute inhaled multi-walled carbon nanotube exposure on signaling pathways associated with cholesterol transport and inflammatory markers in the vasculature of wild-type miceCommentary: MicroRNA-221/222 Family Counteracts Myocardial Fibrosis in Pressure Overload-Induced Heart Failure
P2860
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P2860
MiR-221 promotes cardiac hypertrophy in vitro through the modulation of p27 expression.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年学术文章
@wuu
2012年学术文章
@zh
2012年学术文章
@zh-cn
2012年学术文章
@zh-hans
2012年学术文章
@zh-my
2012年学术文章
@zh-sg
2012年學術文章
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2012年學術文章
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name
MiR-221 promotes cardiac hypertrophy in vitro through the modulation of p27 expression.
@en
MiR-221 promotes cardiac hypertrophy in vitro through the modulation of p27 expression.
@nl
type
label
MiR-221 promotes cardiac hypertrophy in vitro through the modulation of p27 expression.
@en
MiR-221 promotes cardiac hypertrophy in vitro through the modulation of p27 expression.
@nl
prefLabel
MiR-221 promotes cardiac hypertrophy in vitro through the modulation of p27 expression.
@en
MiR-221 promotes cardiac hypertrophy in vitro through the modulation of p27 expression.
@nl
P2093
P2860
P356
P1476
MiR-221 promotes cardiac hypertrophy in vitro through the modulation of p27 expression.
@en
P2093
Changxin Wang
Jizheng Wang
Shuiyun Wang
Xiaojian Wang
P2860
P304
P356
10.1002/JCB.24075
P577
2012-06-01T00:00:00Z