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Interferon-α improves phosphoantigen-induced Vγ9Vδ2 T-cells interferon-γ production during chronic HCV infectionDifferent features of Vδ2 T and NK cells in fatal and non-fatal human Ebola infections.Human macrophage gamma interferon decreases gene expression but not replication of Mycobacterium tuberculosis: analysis of the host-pathogen reciprocal influence on transcription in a comparison of strains H37Rv and CMT97.HIV infection of monocytes-derived dendritic cells inhibits Vγ9Vδ2 T cells functionsGranulocytic myeloid derived suppressor cells expansion during active pulmonary tuberculosis is associated with high nitric oxide plasma level.Characterization of transglutaminase type II role in dendritic cell differentiation and function.Primary and Chronic HIV Infection Differently Modulates Mucosal Vδ1 and Vδ2 T-Cells Differentiation Profile and Effector Functions.Vγ9Vδ2 T-Cell Polyfunctionality Is Differently Modulated in HAART-Treated HIV Patients according to CD4 T-Cell Count.Longitudinal characterization of dysfunctional T cell-activation during human acute Ebola infection.Modulation of Phenotype and Function of Human CD4+CD25+ T Regulatory Lymphocytes Mediated by cAMP-Elevating AgentsHuman Zika infection induces a reduction of IFN-γ producing CD4 T-cells and a parallel expansion of effector Vδ2 T-cells.HIV impairs CD34+-derived monocytic precursor differentiation into functional dendritic cells.Bone Marrow CD34+ Progenitor Cells from HIV-Infected Patients Show an Impaired T Cell Differentiation Potential Related to Proinflammatory Cytokines.Dendritic cells activation is associated with sustained virological response to telaprevir treatment of HCV-infected patients.HIV-Specific CD8 T Cells Producing CCL-4 Are Associated With Worse Immune Reconstitution During Chronic Infection.Granulocytic Myeloid-Derived Suppressor Cells Increased in Early Phases of Primary HIV Infection Depending on TRAIL Plasma Level.In HIV-positive patients, myeloid-derived suppressor cells induce T-cell anergy by suppressing CD3ζ expression through ELF-1 inhibition.Chronic HIV-infected patients show an impaired dendritic cells differentiation of bone marrow CD34⁺ cells.An abnormal phenotype of lung Vγ9Vδ2 T cells impairs their responsiveness in tuberculosis patients.The basal activation state of DC subsets correlates with anti-HCV treatment outcome in HCV/HIV co-infected patients.Activation of interferon response genes and of plasmacytoid dendritic cells in HIV-1 positive subjects with GB virus C co-infection.GB virus type C-driven protection in HIV/HCV coinfection: possible role of interferon gamma and dendritic cell activation.In HIV/HCV coinfected patients dendritic cell activation state is not associated with IL28B genotype.Co-stimulatory molecule CD80 expression may correlate with anti-HCV treatment outcome.GB-virus type C effect on HIV infection, interferon system, and dendritric cells.Early ART in primary HIV infection may also preserve lymphopoiesis capability in circulating haematopoietic progenitor cells: a case report.Cyclic adenosine 5'-monophosphate and calcium induce CD152 (CTLA-4) up-regulation in resting CD4+ T lymphocytes.Cutting edge: TGF-beta1 and IL-15 Induce FOXP3+ gammadelta regulatory T cells in the presence of antigen stimulation.Bacterial infections promote T cell recognition of self-glycolipids.Zoledronic acid and interleukin-2 treatment improves immunocompetence in HIV-infected persons by activating Vgamma9Vdelta2 T cells.CD3zeta down-modulation may explain Vgamma9Vdelta2 T lymphocyte anergy in HIV-infected patients.Complementary function of gamma delta T-lymphocytes and dendritic cells in the response to isopentenyl-pyrophosphate and lipopolysaccharide antigens.Differentiation of monocytes into CD1a- dendritic cells correlates with disease progression in HIV-infected patients.Dendritic cells derived from BCG-infected precursors induce Th2-like immune response.Intrahepatic Vγ9Vδ2 T-cells from HCV-infected patients show an exhausted phenotype but can inhibit HCV replication.IL-18 and Stem Cell Factor affect hematopoietic progenitor cells in HIV-infected patients treated during primary HIV infection.Modulation of polyfunctional HIV-specific CD8 T cells in patients responding differently to antiretroviral therapy.Inhibition of T cell proliferation by cholera toxin involves the modulation of costimulatory molecules CTLA-4 and CD28.An IL-15 dependent CD8 T cell response to selected HIV epitopes is related to viral control in early-treated HIV-infected subjects.In HIV/HCV co-infected patients T regulatory and myeloid-derived suppressor cells persist after successful treatment with directly acting antivirals.
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P50
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onderzoeker
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Alessandra Sacchi
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Alessandra Sacchi
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A Sacchi
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Alessandra Sacchi
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P1053
K-7782-2016
P106
P1153
23019544600
P21
P31
P3829
P496
0000-0003-4964-386X