Hybrid spreading mechanisms and T cell activation shape the dynamics of HIV-1 infection - Wikidata - awgldk - TriplyDB

Hybrid spreading mechanisms and T cell activation shape the dynamics of HIV-1 infection

Hybrid spreading mechanisms and T cell activation shape the dynamics of HIV-1 infection

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Spatiotemporal Dynamics of Virus Infection Spreading in Tissues HIV-1-Induced Small T Cell Syncytia Can Transfer Virus Particles to Target Cells through Transient Contacts Multiscale modeling of virus replication and spread.Modeling Viral Spread.All-Round Manipulation of the Actin Cytoskeleton by HIV.Accounting for Space—Quantification of Cell-To-Cell Transmission Kinetics Using Virus Dynamics Models.

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Hybrid spreading mechanisms and T cell activation shape the dynamics of HIV-1 infection

http://www.wikidata.org/entity/Q21092519

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2015 nî lūn-bûn

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2015 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2015 թվականի ապրիլին հրատարակված գիտական հոդված

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年论文

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name

Hybrid spreading mechanisms and T cell activation shape the dynamics of HIV-1 infection

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Hybrid spreading mechanisms and T cell activation shape the dynamics of HIV-1 infection

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Hybrid spreading mechanisms and T cell activation shape the dynamics of HIV-1 infection

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Hybrid spreading mechanisms and T cell activation shape the dynamics of HIV-1 infection

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Hybrid spreading mechanisms and T cell activation shape the dynamics of HIV-1 infection

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Hybrid spreading mechanisms and T cell activation shape the dynamics of HIV-1 infection

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Hybrid spreading mechanisms and T cell activation shape the dynamics of HIV-1 infection

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Hybrid spreading mechanisms and T cell activation shape the dynamics of HIV-1 infection

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Hybrid spreading mechanisms and T cell activation shape the dynamics of HIV-1 infection

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Hybrid spreading mechanisms and T cell activation shape the dynamics of HIV-1 infection

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Hybrid spreading mechanisms and T cell activation shape the dynamics of HIV-1 infection

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Hybrid spreading mechanisms and T cell activation shape the dynamics of HIV-1 infection

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JOURNAL.PCBI.1004179

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PLOS Computational Biology

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Hybrid spreading mechanisms and T cell activation shape the dynamics of HIV-1 infection

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Changwang Zhang

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Ian Williams

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Pierre Pellegrino

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P2860

Modeling the within-host dynamics of HIV infection

High multiplicity HIV-1 cell-to-cell transmission from macrophages to CD4+ T cells limits antiretroviral efficacy

CD4+ T cell depletion during all stages of HIV disease occurs predominantly in the gastrointestinal tract

HIV-1 cell to cell transfer across an Env-induced, actin-dependent synapse

Avoiding the void: cell-to-cell spread of human viruses

In vivo imaging of virological synapses.

Cell death by pyroptosis drives CD4 T-cell depletion in HIV-1 infection

Cell-to-cell spread of HIV permits ongoing replication despite antiretroviral therapy

Modelling the course of an HIV infection: insights from ecology and evolution

Consolidated Guidelines on the Use of Antiretroviral Drugs for Treating and Preventing HIV Infection: Recommendations for a Public Health Approach

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e1004179

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Persephone Borrow

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274390201

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1503.08992

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