about
DEAD/H-box helicase 11Timeless circadian regulatorForkhead box M1O-6-methylguanine-DNA methyltransferaseNudix hydrolase 16Scaffold protein involved in DNA repairSpire type actin nucleation factor 1Spire type actin nucleation factor 2Formin 2SMC5-SMC6 complex localization factor 1SMC5-SMC6 complex localization factor 2SMC5-SMC6 complex localization factor 2SMC5-SMC6 complex localization factor 1Formin 2DEAD/H (Asp-Glu-Ala-Asp/His) box helicase 11Scaffolding protein involved in DNA repairForkhead box protein M1O-6-methylguanine-DNA methyltransferaseNudix (nucleoside diphosphate linked moiety X)-type motif 16Spire type actin nucleation factor 1Spire type actin nucleation factor 2Timeless circadian clock 1O-6-methylguanine-DNA methyltransferaseWD repeat containing, antisense to TP53Timeless circadian regulatorSMC5-SMC6 complex localization factor 1Spire-type actin nucleation factor 1aSMC5-SMC6 complex localization factor 1Timeless circadian regulator
P682
NUDT16 is a (deoxy)inosine diphosphatase, and its deficiency induces accumulation of single-strand breaks in nuclear DNA and growth arrestChk2 mediates stabilization of the FoxM1 transcription factor to stimulate expression of DNA repair genesScaffolding protein SPIDR/KIAA0146 connects the Bloom syndrome helicase with homologous recombination repairMiR-221/222 target the DNA methyltransferase MGMT in glioma cellsRoles of ChlR1 DNA helicase in replication recovery from DNA damageDNA damage induces nuclear actin filament assembly by Formin -2 and Spire-½ that promotes efficient DNA repair. [corrected].Phosphorylation of the Cajal body protein WRAP53β by ATM promotes its involvement in the DNA damage response.
P921
Q21102307-26ECFDE0-D80F-4D52-B3F1-AFB08035B293Q21102311-7F0B3D87-CC3D-4B35-9992-6FE712350414Q21108344-5EABD42D-B834-4E6A-9052-564E54BC8099Q21118735-80EA561F-B6F3-4D0E-B882-5C5A7BD4C0EDQ21120178-0D1688E4-67C2-4472-85F1-C0537E54B1E1Q21121425-C47A3336-89D3-48E4-8790-EDFF66CA9A7EQ21124556-4EB375C3-2BD6-4AA6-BEAE-F1B2716C8A76Q21124561-5AB35AD7-A9B6-4DF2-BD69-2DB8A289887BQ21132238-9CD6BB62-2938-4926-9869-AABC58C02182Q21418912-CF049424-B7D1-40BC-8180-F3B5BE73F3E2Q21423628-15B65A9C-60DD-49FD-A1F7-0507F5F35A23Q21484006-9C990C4E-BB41-4013-B58C-06398D83E9C6Q21484007-4B0DE527-4ACE-427B-8EE0-FB601F2F5554Q21495742-1EEE080C-5015-469F-8940-EB42BA7060FBQ21495742-569035EA-9F8D-4429-8B03-388C7B3BADCEQ21498273-8CA607C4-58F8-4180-9983-7DC681816127Q21498273-B9B246A9-8ECD-4565-9CAE-7708D54D1BEFQ21980200-4790EA82-5C3E-4C80-BEC0-B85998C9F19BQ21983966-0E13B49E-82B3-4CCF-982F-61A2492E585EQ21985779-390C5A03-D5F1-44B6-A32E-158806F5ED31Q21987778-F3B62F2B-DD1F-44C4-A397-0150191F00D0Q21988574-A1C1267D-7715-488A-8B1B-F08ABEF044BAQ21988580-0640F861-F6FB-4415-9FB6-391ECB79851AQ21988580-107EDDAC-C438-4745-A346-50145C4E84DAQ21989360-98326187-8198-4CE5-AA13-3B8A08CB6C50Q28556746-F5A184C8-BF51-450D-9CFA-BFE98FAB63C1Q28558281-4D1E4C0F-AEBA-45F8-831B-EE20C6D064B8Q28562525-9F9B4079-5C0A-41F1-ADAD-0CE455B8CDB3Q29522614-0EC24F18-2037-4049-9E71-6750413B5B20Q29832151-868A3B14-C658-4DB8-A2DC-D9C34BBD01C3Q55198462-7DADD48C-0AAD-4946-860C-C674F8A5AC29Q55200472-29ADD308-592A-4D42-8DE7-0B3050A774FC
P682
Q24306490-15F5850C-D8B1-405A-98CE-2CF26110C63CQ24314901-381B0C60-FE26-4098-9600-EEAE63A7D68DQ24336963-AFB8C8B5-6442-40A8-B2CC-38DF4590C4C4Q28118866-1ED43A06-007E-48D1-98CE-69807000656FQ28119141-EBA441F9-9E7F-4793-A0F0-A8D16389D889Q36080669-70CAA70E-1C79-4AF1-9327-42AEE652F878Q41072533-96039FD7-CD56-46A2-A12C-D5320F45013C
P921
description
Any process that activates or ...... of double-strand break repair.
@en
biologisch proces
@nl
name
positive regulation of double-strand break repair
@en
type
label
positive regulation of double-strand break repair
@en
prefLabel
positive regulation of double-strand break repair
@en
P2888
P686
GO:2000781