Urinary copper elevation in a mouse model of Wilson's disease is a regulated process to specifically decrease the hepatic copper load

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2012 nî lūn-bûn

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2012 թուականին հրատարակուած գիտական յօդուած

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2012 թվականին հրատարակված գիտական հոդված

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

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name

Urinary copper elevation in a ...... crease the hepatic copper load

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Urinary copper elevation in a ...... crease the hepatic copper load

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Urinary copper elevation in a ...... crease the hepatic copper load

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Urinary copper elevation in a ...... crease the hepatic copper load

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Urinary copper elevation in a ...... crease the hepatic copper load

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Urinary copper elevation in a ...... crease the hepatic copper load

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Urinary copper elevation in a ...... crease the hepatic copper load

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Urinary copper elevation in a ...... crease the hepatic copper load

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Urinary Copper Elevation in a ...... crease the Hepatic Copper Load

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Urinary copper elevation in a ...... crease the hepatic copper load

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Urinary copper elevation in a ...... crease the hepatic copper load

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Urinary copper elevation in a ...... crease the hepatic copper load

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Urinary copper elevation in a ...... crease the hepatic copper load

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P1476

Urinary copper elevation in a ...... crease the hepatic copper load

@en

P2093

Abigael Muchenditsi

http://www.w3.org/2001/XMLSchema#string

Jaekwon Lee

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Lawrence W Gray

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Otto Muzik

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Shannon A Molloy

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Svetlana Lutsenko

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Venkata S Pendyala

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P2860

Mitochondrial matrix copper complex used in metallation of cytochrome oxidase and superoxide dismutase

The Wilson disease gene is a copper transporting ATPase with homology to the Menkes disease gene

Copper-stimulated endocytosis and degradation of the human copper transporter, hCtr1

The copper-transporting ATPases, menkes and wilson disease proteins, have distinct roles in adult and developing cerebellum

Null mutation of the murine ATP7B (Wilson disease) gene results in intracellular copper accumulation and late-onset hepatic nodular transformation

Consequences of copper accumulation in the livers of the Atp7b-/- (Wilson disease gene) knockout mice

Cardiac copper deficiency activates a systemic signaling mechanism that communicates with the copper acquisition and storage organs.

Wilson disease at a single cell level: intracellular copper trafficking activates compartment-specific responses in hepatocytes.

Ctr1 is an apical copper transporter in mammalian intestinal epithelial cells in vivo that is controlled at the level of protein stability

The Wilson disease gene is a putative copper transporting P-type ATPase similar to the Menkes gene.

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e38327

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scholarly article

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4279515

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10.1371/JOURNAL.PONE.0038327

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English

P433

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P478

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P50

Fangyu Peng

Jack H. Kaplan

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2012-01-01T00:00:00Z

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P5875

229157726

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22802922

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3390108

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