MAnorm: a robust model for quantitative comparison of ChIP-Seq data sets - Wikidata - awgldk - TriplyDB

MAnorm: a robust model for quantitative comparison of ChIP-Seq data sets

MAnorm: a robust model for quantitative comparison of ChIP-Seq data sets

http://www.wikidata.org/entity/Q21184004

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Practical guidelines for the comprehensive analysis of ChIP-seq data Systematic identification of gene family regulators in mouse and human embryonic stem cells Neuronal Kmt2a/Mll1 histone methyltransferase is essential for prefrontal synaptic plasticity and working memory Higher-order unfolding of satellite heterochromatin is a consistent and early event in cell senescence Integrative analysis of C. elegans modENCODE ChIP-seq data sets to infer gene regulatory interactions.Accounting for immunoprecipitation efficiencies in the statistical analysis of ChIP-seq data.MMDiff: quantitative testing for shape changes in ChIP-Seq data sets An integrated model of multiple-condition ChIP-Seq data reveals predeterminants of Cdx2 binding.Myotonic dystrophy type 1 patient-derived iPSCs for the investigation of CTG repeat instability.Nonparametric Tests for Differential Histone Enrichment with ChIP-Seq Data.CARMO: a comprehensive annotation platform for functional exploration of rice multi-omics data.BioWardrobe: an integrated platform for analysis of epigenomics and transcriptomics data.Variable reproducibility in genome-scale public data: A case study using ENCODE ChIP sequencing resource.Statistical challenges in analyzing methylation and long-range chromosomal interaction data A ChIP-Seq Data Analysis Pipeline Based on Bioconductor Packages Transcriptome-wide N 6 -methyladenosine methylome profiling of porcine muscle and adipose tissues reveals a potential mechanism for transcriptional regulation and differential methylation pattern Epimetheus - a multi-profile normalizer for epigenomic sequencing data.dCLIP: a computational approach for comparative CLIP-seq analyses.Loss of Asxl2 leads to myeloid malignancies in mice Chromatin module inference on cellular trajectories identifies key transition points and poised epigenetic states in diverse developmental processes.Ethylene induces combinatorial effects of histone H3 acetylation in gene expression in Arabidopsis.Chloroquine eliminates cancer stem cells through deregulation of Jak2 and DNMT1.Identification of in vivo DNA-binding mechanisms of Pax6 and reconstruction of Pax6-dependent gene regulatory networks during forebrain and lens development.Embryonic transcription is controlled by maternally defined chromatin state.Modelling Fanconi anemia pathogenesis and therapeutics using integration-free patient-derived iPSCs Widespread misinterpretable ChIP-seq bias in yeast.histoneHMM: Differential analysis of histone modifications with broad genomic footprints.dCaP: detecting differential binding events in multiple conditions and proteins Identifying differential transcription factor binding in ChIP-seq.Selective coregulator function and restriction of steroid receptor chromatin occupancy by Hic-5.DNA methylation and chromatin organization in insects: insights from the Ant Camponotus floridanus.Identifying TF-MiRNA Regulatory Relationships Using Multiple Features.Detection of differentially methylated regions from whole-genome bisulfite sequencing data without replicates.Dynamics of MBD2 deposition across methylated DNA regions during malignant transformation of human mammary epithelial cells Arabidopsis Flower and Embryo Developmental Genes are Repressed in Seedlings by Different Combinations of Polycomb Group Proteins in Association with Distinct Sets of Cis-regulatory Elements.A novel statistical method for quantitative comparison of multiple ChIP-seq datasets Cross Talk Between GH-Regulated Transcription Factors HNF6 and CUX2 in Adult Mouse Liver.Systematic chromatin state comparison of epigenomes associated with diverse properties including sex and tissue type.CTCF-dependent co-localization of canonical Smad signaling factors at architectural protein binding sites in D. melanogaster.An Alternative Approach to ChIP-Seq Normalization Enables Detection of Genome-Wide Changes in Histone H3 Lysine 27 Trimethylation upon EZH2 Inhibition.

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MAnorm: a robust model for quantitative comparison of ChIP-Seq data sets

http://www.wikidata.org/entity/Q21184004

description

2012 nî lūn-bûn

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MAnorm: a robust model for quantitative comparison of ChIP-Seq data sets

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MAnorm: a robust model for quantitative comparison of ChIP-Seq data sets

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MAnorm: a robust model for quantitative comparison of ChIP-Seq data sets

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MAnorm: a robust model for quantitative comparison of ChIP-Seq data sets

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MAnorm: a robust model for quantitative comparison of ChIP-Seq data sets

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MAnorm: a robust model for quantitative comparison of ChIP-Seq data sets

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MAnorm: a robust model for quantitative comparison of ChIP-Seq data sets

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MAnorm: a robust model for quantitative comparison of ChIP-Seq data sets

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MAnorm: a robust model for quantitative comparison of ChIP-Seq data sets

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Guo-Cheng Yuan

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Stuart H Orkin

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Yijing Zhang

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Model-based analysis of ChIP-Seq (MACS)

Five-vertebrate ChIP-seq reveals the evolutionary dynamics of transcription factor binding

Core transcriptional regulatory circuitry in human embryonic stem cells

PHF8 mediates histone H4 lysine 20 demethylation events involved in cell cycle progression

Significance analysis of microarrays applied to the ionizing radiation response

Genome-wide analysis reveals novel molecular features of mouse recombination hotspots

Histone H3K27ac separates active from poised enhancers and predicts developmental state

c-Myc regulates transcriptional pause release

Genomic mapping of RNA polymerase II reveals sites of co-transcriptional regulation in human cells.

A comparison of normalization methods for high density oligonucleotide array data based on variance and bias

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