Sensitive indicators of injury reveal hippocampal damage in C57BL/6J mice treated with kainic acid in the absence of tonic-clonic seizures
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Striatal dysregulation of Cdk5 alters locomotor responses to cocaine, motor learning, and dendritic morphologyCorticosterone primes the neuroinflammatory response to DFP in mice: potential animal model of Gulf War IllnessEarly activation of STAT3 regulates reactive astrogliosis induced by diverse forms of neurotoxicityAge exaggerates proinflammatory cytokine signaling and truncates signal transducers and activators of transcription 3 signaling following ischemic stroke in the ratVacuolar leukoencephalopathy with widespread astrogliosis in mice lacking transcription factor Nrf2Immediate Epileptogenesis after Kainate-Induced Status Epilepticus in C57BL/6J Mice: Evidence from Long Term Continuous Video-EEG TelemetryThe newly synthesized pool of dopamine determines the severity of methamphetamine-induced neurotoxicityMidkine, heparin-binding growth factor, blocks kainic acid-induced seizure and neuronal cell death in mouse hippocampusOrgan-specific protective role of NKT cells in virus-induced inflammatory demyelination and myocarditis depends on mouse strain.Predominant functional expression of Kv1.3 by activated microglia of the hippocampus after Status epilepticus.Transcriptome analysis of the hippocampal CA1 pyramidal cell region after kainic acid-induced status epilepticus in juvenile rats.Role for complement in the development of seizures following acute viral infection.Unexpected survival of neurons of origin of the pyramidal tract after spinal cord injuryImportance of genetic background for risk of relapse shown in altered prefrontal cortex gene expression during abstinence following chronic alcohol intoxication.Innate but not adaptive immune responses contribute to behavioral seizures following viral infection.A New Transgenic Mouse Model for Studying the Neurotoxicity of Spermine Oxidase Dosage in the Response to Excitotoxic Injury.NMDA-induced seizure intensity is enhanced in COX-2 deficient mice.Interleukin-6, produced by resident cells of the central nervous system and infiltrating cells, contributes to the development of seizures following viral infection.Kainic Acid-induced neurotoxicity: targeting glial responses and glia-derived cytokines.Lack of correlation of central nervous system inflammation and neuropathology with the development of seizures following acute virus infection.Infiltrating macrophages are key to the development of seizures following virus infection.Altered GABAergic neurotransmission is associated with increased kainate-induced seizure in prostaglandin-endoperoxide synthase-2 deficient miceNBQX, a highly selective competitive antagonist of AMPA and KA ionotropic glutamate receptors, increases seizures and mortality following picornavirus infectionThe effects of diet on the severity of central nervous system disease: One part of lab-to-lab variabilityInvolvement of the neuronal phosphotyrosine signal adaptor N-Shc in kainic acid-induced epileptiform activity.Dopamine disposition in the presynaptic process regulates the severity of methamphetamine-induced neurotoxicity.Brevetoxin-induced neural insult in the retrosplenial cortex of mouse brainRecommended neuroanatomical sampling practices for comprehensive brain evaluation in nonclinical safety studies.What is microglia neurotoxicity (Not)?T-bet, but not Gata3, overexpression is detrimental in a neurotropic viral infection.The role of peripheral interleukin-6 in the development of acute seizures following virus encephalitis.Fatherhood alters behavioural and neural responsiveness in a spatial task.Neuroprotective Effects of the Absence of JNK1 or JNK3 Isoforms on Kainic Acid-Induced Temporal Lobe Epilepsy-Like Symptoms.Quantitative Assessment of MRI T2 Response to Kainic Acid Neurotoxicity in Rats in vivo.GFAP expression in the rat brain following sub-chronic exposure to a 900 MHz electromagnetic field signal.Mice Lacking Functional Fas Death Receptors Are Protected from Kainic Acid-Induced Apoptosis in the Hippocampus.The immune response to picornavirus infection and the effect of immune manipulation on acute seizures.
P2860
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P2860
Sensitive indicators of injury reveal hippocampal damage in C57BL/6J mice treated with kainic acid in the absence of tonic-clonic seizures
description
2004 nî lūn-bûn
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2004 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2004 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
name
Sensitive indicators of injury ...... sence of tonic-clonic seizures
@ast
Sensitive indicators of injury ...... sence of tonic-clonic seizures
@en
Sensitive indicators of injury ...... sence of tonic-clonic seizures
@en-gb
Sensitive indicators of injury ...... sence of tonic–clonic seizures
@nl
type
label
Sensitive indicators of injury ...... sence of tonic-clonic seizures
@ast
Sensitive indicators of injury ...... sence of tonic-clonic seizures
@en
Sensitive indicators of injury ...... sence of tonic-clonic seizures
@en-gb
Sensitive indicators of injury ...... sence of tonic–clonic seizures
@nl
prefLabel
Sensitive indicators of injury ...... sence of tonic-clonic seizures
@ast
Sensitive indicators of injury ...... sence of tonic-clonic seizures
@en
Sensitive indicators of injury ...... sence of tonic-clonic seizures
@en-gb
Sensitive indicators of injury ...... sence of tonic–clonic seizures
@nl
P921
P3181
P1433
P1476
Sensitive indicators of injury ...... sence of tonic-clonic seizures
@en
P2093
Diane Bemis Miller
Stanley Anthony Benkovic
P2880
P3181
P356
10.1016/J.BRAINRES.2004.07.021
P407
P577
2004-10-22T00:00:00Z