sameAs
Mal (MyD88-adapter-like) is required for Toll-like receptor-4 signal transductionHuman tribbles, a protein family controlling mitogen-activated protein kinase cascadesTRIL, a functional component of the TLR4 signaling complex, highly expressed in brainPromyelocytic leukemia protein interacts with the apoptosis-associated speck-like protein to limit inflammasome activationWhat the Myddosome structure tells us about the initiation of innate immunityThe myristoylation of TRIF-related adaptor molecule is essential for Toll-like receptor 4 signal transductionIL-10 inhibits miR-155 induction by toll-like receptorsQuestions and controversies in innate immune research: what is the physiological role of NLRP3?Immunometabolism governs dendritic cell and macrophage functionMicroRNAs and the resolution phase of inflammation in macrophagesHigh-resolution structure of murine interleukin 1 homologue IL-1F5 reveals unique loop conformations for receptor binding specificityRas participates in the activation of p38 MAPK by interleukin-1 by associating with IRAK, IRAK2, TRAF6, and TAK-1Toll-like receptors: from the discovery of NFkappaB to new insights into transcriptional regulations in innate immunityModulatory mechanisms controlling the NLRP3 inflammasome in inflammation: recent developmentsCorrection: The Cytokine Release Inhibitory Drug CRID3 Targets ASC Oligomerisation in the NLRP3 and AIM2 InflammasomesSchlafen-1 causes a cell cycle arrest by inhibiting induction of cyclin D1Correction: The Cytokine Release Inhibitory Drug CRID3 Targets ASC Oligomerisation in the NLRP3 and AIM2 InflammasomesThe murine IRAK2 gene encodes four alternatively spliced isoforms, two of which are inhibitoryA Mal functional variant is associated with protection against invasive pneumococcal disease, bacteremia, malaria and tuberculosisAnnotating genes with potential roles in the immune system: six new members of the IL-1 familyThe family of five: TIR-domain-containing adaptors in Toll-like receptor signallingA dimer of the Toll-like receptor 4 cytoplasmic domain provides a specific scaffold for the recruitment of signalling adaptor proteinsDetecting microRNA activity from gene expression dataTILRR, a novel IL-1RI co-receptor, potentiates MyD88 recruitment to control Ras-dependent amplification of NF-kappaBOxidative stress and nuclear factor-kappaB activation: a reassessment of the evidence in the light of recent discoveries.The MyD88+ phenotype is an adverse prognostic factor in epithelial ovarian cancer.TRIL is involved in cytokine production in the brain following Escherichia coli infection.Endosomal NOX2 oxidase exacerbates virus pathogenicity and is a target for antiviral therapy.A role for TLR4 in Clostridium difficile infection and the recognition of surface layer proteins.Mycobacterial PIMs inhibit host inflammatory responses through CD14-dependent and CD14-independent mechanismsMetabolic reprograming in macrophage polarization.IL-1 family nomenclature.Characterization of signaling pathways activated by the interleukin 1 (IL-1) receptor homologue T1/ST2. A role for Jun N-terminal kinase in IL-4 induction.Retrospective. Jürg Tschopp (1951-2011).A trio of microRNAs that control Toll-like receptor signalling.Succinate: a metabolic signal in inflammation.The interleukin-1 receptor/Toll-like receptor superfamily: signal transduction during inflammation and host defense.mTOR- and HIF-1α-mediated aerobic glycolysis as metabolic basis for trained immunity.Adding fuel to fire: microRNAs as a new class of mediators of inflammation.Distinct mechanisms for induction and tolerance regulate the immediate early genes encoding interleukin 1β and tumor necrosis factor α.
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