Loss of intestinal core 1-derived O-glycans causes spontaneous colitis in mice
about
A modular organization of the human intestinal mucosal microbiota and its association with inflammatory bowel diseaseA Bitter Sweet Symphony: Immune Responses to Altered O-glycan Epitopes in CancerTherapeutic Potential to Modify the Mucus Barrier in Inflammatory Bowel DiseaseMechanisms of Microbe-Host Interaction in Crohn's Disease: Dysbiosis vs. Pathobiont SelectionGenetically engineered mouse models for studying inflammatory bowel diseaseUlcerative colitis as a polymicrobial infection characterized by sustained broken mucus barrierGoblet cell compound exocytosis in the defense against bacterial invasion in the colon exposed to ischemia-reperfusionMucin-type O-glycosylation during developmentA refined palate: bacterial consumption of host glycans in the gutSpontaneous colitis in Muc2-deficient mice reflects clinical and cellular features of active ulcerative colitisRole of Cytokine-Induced Glycosylation Changes in Regulating Cell Interactions and Cell Signaling in Inflammatory Diseases and CancerHelminthic therapy: improving mucosal barrier functionModulation of ocular surface glycocalyx barrier function by a galectin-3 N-terminal deletion mutant and membrane-anchored synthetic glycopolymersGenetics and pathogenesis of inflammatory bowel diseaseO-glycosylation regulates polarized secretion by modulating Tango1 stability.C1GALT1 overexpression promotes the invasive behavior of colon cancer cells through modifying O-glycosylation of FGFR2.Uncovering Pathogenic Mechanisms of Inflammatory Bowel Disease Using Mouse Models of Crohn's Disease-Like Ileitis: What is the Right Model?α3/4 Fucosyltransferase 3-dependent synthesis of Sialyl Lewis A on CD44 variant containing exon 6 mediates polymorphonuclear leukocyte detachment from intestinal epithelium during transepithelial migration.Claudin-1 overexpression in intestinal epithelial cells enhances susceptibility to adenamatous polyposis coli-mediated colon tumorigenesis.Activation of epidermal growth factor receptor mediates mucin production stimulated by p40, a Lactobacillus rhamnosus GG-derived protein.Discordance between changes in the gut microbiota and pathogenicity in a mouse model of spontaneous colitis.Immune and genetic gardening of the intestinal microbiome.Membrane protein profiling of human colon reveals distinct regional differencesTargeted disruption of core 1 β1,3-galactosyltransferase (C1galt1) induces apical endocytic trafficking in human corneal keratinocytes.Microbiome and immunological interactions.The gastrointestinal mucus system in health and diseaseFast renewal of the distal colonic mucus layers by the surface goblet cells as measured by in vivo labeling of mucin glycoproteins.Mucus and the goblet cell.Potential of fecal microbiota for early-stage detection of colorectal cancer.Targeted colonic claudin-2 expression renders resistance to epithelial injury, induces immune suppression, and protects from colitis.Suppression of core 1 Gal-transferase is associated with reduction of TF and reciprocal increase of Tn, sialyl-Tn and Core 3 glycans in human colon cancer cellsThe mucus and mucins of the goblet cells and enterocytes provide the first defense line of the gastrointestinal tract and interact with the immune system.Studies of mucus in mouse stomach, small intestine, and colon. III. Gastrointestinal Muc5ac and Muc2 mucin O-glycan patterns reveal a regiospecific distribution.The composition of the gut microbiota throughout life, with an emphasis on early life.Mice overexpressing β-1,4-Galactosyltransferase I are resistant to TNF-induced inflammation and DSS-induced colitisThe composition of the gut microbiota shapes the colon mucus barrier.Influences of the colonic microbiome on the mucous gel layer in ulcerative colitisReprograming of gut microbiome energy metabolism by the FUT2 Crohn's disease risk polymorphism.Alterations in the intestinal microbiome (dysbiosis) as a predictor of relapse after infliximab withdrawal in Crohn's disease.Microbiota-mediated inflammation and antimicrobial defense in the intestine.
P2860
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P2860
Loss of intestinal core 1-derived O-glycans causes spontaneous colitis in mice
description
2011 nî lūn-bûn
@nan
2011 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Loss of intestinal core 1-derived O-glycans causes spontaneous colitis in mice
@ast
Loss of intestinal core 1-derived O-glycans causes spontaneous colitis in mice
@en
Loss of intestinal core 1-derived O-glycans causes spontaneous colitis in mice
@en-gb
Loss of intestinal core 1-derived O-glycans causes spontaneous colitis in mice
@nl
type
label
Loss of intestinal core 1-derived O-glycans causes spontaneous colitis in mice
@ast
Loss of intestinal core 1-derived O-glycans causes spontaneous colitis in mice
@en
Loss of intestinal core 1-derived O-glycans causes spontaneous colitis in mice
@en-gb
Loss of intestinal core 1-derived O-glycans causes spontaneous colitis in mice
@nl
prefLabel
Loss of intestinal core 1-derived O-glycans causes spontaneous colitis in mice
@ast
Loss of intestinal core 1-derived O-glycans causes spontaneous colitis in mice
@en
Loss of intestinal core 1-derived O-glycans causes spontaneous colitis in mice
@en-gb
Loss of intestinal core 1-derived O-glycans causes spontaneous colitis in mice
@nl
P2093
P2860
P3181
P356
P1476
Loss of intestinal core 1-derived O-glycans causes spontaneous colitis in mice
@en
P2093
Emily Bradford
Gunnar C Hansson
J Michael McDaniel
Jerrold R Turner
Jianxin Fu
Jonathan Braun
Kristina A Thomsson
P2860
P304
P3181
P356
10.1172/JCI45538
P407
P577
2011-04-01T00:00:00Z