Influence of XPB helicase on recruitment and redistribution of nucleotide excision repair proteins at sites of UV-induced DNA damage
about
Nucleotide excision repair proteins rapidly accumulate but fail to persist in human XP-E (DDB2 mutant) cellsSequential recruitment of the repair factors during NER: the role of XPG in initiating the resynthesis step.Persistence of repair proteins at unrepaired DNA damage distinguishes diseases with ERCC2 (XPD) mutations: cancer-prone xeroderma pigmentosum vs. non-cancer-prone trichothiodystrophy.A unified model for the molecular basis of Xeroderma pigmentosum-Cockayne Syndrome.XPC branch-point sequence mutations disrupt U2 snRNP binding, resulting in abnormal pre-mRNA splicing in xeroderma pigmentosum patients.XPB induces C1D expression to counteract UV-induced apoptosis.Excision repair is required for genotoxin-induced mutagenesis in mammalian cells.UV-induced histone H2AX phosphorylation and DNA damage related proteins accumulate and persist in nucleotide excision repair-deficient XP-B cellsThe NR4A2 nuclear receptor is recruited to novel nuclear foci in response to UV irradiation and participates in nucleotide excision repairDiagnosis of Xeroderma Pigmentosum and Related DNA Repair-Deficient Cutaneous DiseasesLack of CAK complex accumulation at DNA damage sites in XP-B and XP-B/CS fibroblasts reveals differential regulation of CAK anchoring to core TFIIH by XPB and XPD helicases during nucleotide excision repair.Dissection of the molecular defects caused by pathogenic mutations in the DNA repair factor XPC.XPC initiation codon mutation in xeroderma pigmentosum patients with and without neurological symptomsTelomere attrition and genomic instability in xeroderma pigmentosum type-b deficient fibroblasts under oxidative stressRepair of UV photolesions in xeroderma pigmentosum group C cells induced by translational readthrough of premature termination codons.MC1R and NR4A receptors in cellular stress and DNA repair: implications for UVR protection.Single-stranded DNA binding activity of XPBI, but not XPBII, from Sulfolobus tokodaii causes double-stranded DNA melting.Protein phosphatase 4 regulatory subunit 2 (PPP4R2) is recurrently deleted in acute myeloid leukemia and required for efficient DNA double strand break repair.
P2860
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P2860
Influence of XPB helicase on recruitment and redistribution of nucleotide excision repair proteins at sites of UV-induced DNA damage
description
2007 nî lūn-bûn
@nan
2007 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2007 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
name
Influence of XPB helicase on r ...... sites of UV-induced DNA damage
@ast
Influence of XPB helicase on r ...... sites of UV-induced DNA damage
@en
Influence of XPB helicase on r ...... sites of UV-induced DNA damage
@en-gb
Influence of XPB helicase on r ...... sites of UV-induced DNA damage
@nl
type
label
Influence of XPB helicase on r ...... sites of UV-induced DNA damage
@ast
Influence of XPB helicase on r ...... sites of UV-induced DNA damage
@en
Influence of XPB helicase on r ...... sites of UV-induced DNA damage
@en-gb
Influence of XPB helicase on r ...... sites of UV-induced DNA damage
@nl
prefLabel
Influence of XPB helicase on r ...... sites of UV-induced DNA damage
@ast
Influence of XPB helicase on r ...... sites of UV-induced DNA damage
@en
Influence of XPB helicase on r ...... sites of UV-induced DNA damage
@en-gb
Influence of XPB helicase on r ...... sites of UV-induced DNA damage
@nl
P2093
P2860
P921
P1433
P1476
Influence of XPB helicase on r ...... sites of UV-induced DNA damage
@en
P2093
Jennifer Boyle
Kyoko Imoto
Kyu-Seon Oh
P2860
P304
P356
10.1016/J.DNAREP.2007.03.025
P407
P577
2007-05-16T00:00:00Z