Identification and characterization of a novel Dvl-binding protein that suppresses Wnt signalling pathway
about
A novel missense mutation in CCDC88C activates the JNK pathway and causes a dominant form of spinocerebellar ataxiaA Conserved Role for Girdin in Basal Body Positioning and CiliogenesisCCDC88B is a novel regulator of maturation and effector functions of T cells during pathological inflammationDisturbed Wnt Signalling due to a Mutation in CCDC88C Causes an Autosomal Recessive Non-Syndromic Hydrocephalus with Medial Diverticulum.Proteomics of old world camelid (Camelus dromedarius): Better understanding the interplay between homeostasis and desert environment.A Comprehensive Overview of Skeletal Phenotypes Associated with Alterations in Wnt/β-catenin Signaling in Humans and Mice.Daple is a novel non-receptor GEF required for trimeric G protein activation in Wnt signaling.Role for Daple in non-canonical Wnt signaling during gastric cancer invasion and metastasis.A systems-based approach to investigate dose- and time-dependent methylmercury-induced gene expression response in C57BL/6 mouse embryos undergoing neurulationPlanar Organization of Multiciliated Ependymal (E1) Cells in the Brain Ventricular Epithelium.GIV/Girdin is a rheostat that fine-tunes growth factor signals during tumor progression.HTLV-1 bZIP factor dysregulates the Wnt pathways to support proliferation and migration of adult T-cell leukemia cells.KIAA1509 is a novel PDGFRB fusion partner in imatinib-responsive myeloproliferative disease associated with a t(5;14)(q33;q32).Negative regulation of TCF-dependent signaling by DVL-interacting proteinsA Daple-Akt feed-forward loop enhances noncanonical Wnt signals by compartmentalizing β-catenin.Daple coordinates organ-wide and cell-intrinsic polarity to pattern inner-ear hair bundles.Specific inhibition of GPCR-independent G protein signaling by a rationally engineered protein.DVL binds CCDC88CConvergence of Wnt, growth factor, and heterotrimeric G protein signals on the guanine nucleotide exchange factor Daple.Bi-allelic mutations of CCDC88C are a rare cause of severe congenital hydrocephalus.
P2860
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P2860
Identification and characterization of a novel Dvl-binding protein that suppresses Wnt signalling pathway
description
2003 nî lūn-bûn
@nan
2003 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2003 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2003年の論文
@ja
2003年学术文章
@wuu
2003年学术文章
@zh-cn
2003年学术文章
@zh-hans
2003年学术文章
@zh-my
2003年学术文章
@zh-sg
2003年學術文章
@yue
name
Identification and characteriz ...... presses Wnt signalling pathway
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Identification and characteriz ...... presses Wnt signalling pathway
@en
Identification and characteriz ...... presses Wnt signalling pathway
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Identification and characteriz ...... presses Wnt signalling pathway
@nl
type
label
Identification and characteriz ...... presses Wnt signalling pathway
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Identification and characteriz ...... presses Wnt signalling pathway
@en
Identification and characteriz ...... presses Wnt signalling pathway
@en-gb
Identification and characteriz ...... presses Wnt signalling pathway
@nl
prefLabel
Identification and characteriz ...... presses Wnt signalling pathway
@ast
Identification and characteriz ...... presses Wnt signalling pathway
@en
Identification and characteriz ...... presses Wnt signalling pathway
@en-gb
Identification and characteriz ...... presses Wnt signalling pathway
@nl
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Identification and characteriz ...... presses Wnt signalling pathway
@en
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Akihiko Oshita
Akira Kikuchi
Hiroki Kobayashi
Makoto Asashima
Shosei Kishida
Tatsuo Michiue
Toshimasa Asahara
P2860
P304
P356
10.1111/J.1365-2443.2003.00692.X
P407
P577
2003-12-01T00:00:00Z