An essential function for beta-arrestin 2 in the inhibitory signaling of natural killer cells - Wikidata - awgldk - TriplyDB

An essential function for beta-arrestin 2 in the inhibitory signaling of natural killer cells

An essential function for beta-arrestin 2 in the inhibitory signaling of natural killer cells

http://www.wikidata.org/entity/Q24310297

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Inhibition of TLR signaling by a bacterial protein containing immunoreceptor tyrosine-based inhibitory motifs Multifaceted role of β-arrestins in inflammation and disease Newtonian cell interactions shape natural killer cell education Rhomboid domain-containing protein 3 is a negative regulator of TLR3-triggered natural killer cell activation A Comprehensive View of the β-Arrestinome.Regulation of amygdalar PKA by beta-arrestin-2/phosphodiesterase-4 complex is critical for fear conditioning Imaging immune surveillance of individual natural killer cells confined in microwell arrays.T-cell immunoglobulin and ITIM domain (TIGIT) receptor/poliovirus receptor (PVR) ligand engagement suppresses interferon-γ production of natural killer cells via β-arrestin 2-mediated negative signaling Effects of MHC class I alleles on licensing of Ly49A+ NK cells.Comparative muscle transcriptome associated with carcass traits of Nellore cattle.Beyond desensitization: physiological relevance of arrestin-dependent signaling.Substance P inhibits natural killer cell cytotoxicity through the neurokinin-1 receptor.Identification of AMP-activated protein kinase targets by a consensus sequence search of the proteome.Essential requirement for β-arrestin2 in mouse intestinal tumors with elevated Wnt signaling Recruitment of Grb2 and SHIP1 by the ITT-like motif of TIGIT suppresses granule polarization and cytotoxicity of NK cells.Therapeutic potential of β-arrestin- and G protein-biased agonists.A peptide antagonist disrupts NK cell inhibitory synapse formation β-arrestin-selective G protein-coupled receptor agonists engender unique biological efficacy in vivo The arrestin fold: variations on a theme.Endocytosis as a mechanism of regulating natural killer cell function: unique endocytic and trafficking pathway for CD94/NKG2A.Arrestins in host-pathogen interactions.β-Arrestin-2 modulates radiation-induced intestinal crypt progenitor/stem cell injury.Signal transduction during activation and inhibition of natural killer cells.Natural killer cells: integrating diversity with function.Significant functional heterogeneity among KIR2DL1 alleles and a pivotal role of arginine 245.Building tolerance by dismantling synapses: inhibitory receptor signaling in natural killer cells.β-Arrestins in the immune system β-Arrestins 1 and 2 are critical regulators of inflammation.Identification of Phosphorylation Codes for Arrestin Recruitment by G Protein-Coupled Receptors.An Unbiased Linkage Approach Reveals That the p53 Pathway Is Coupled to NK Cell Maturation.Modulation of host signaling in the inflammatory response by enteropathogenic Escherichia coli virulence proteins.The Diverse Roles of Arrestin Scaffolds in G Protein-Coupled Receptor Signaling.β-Arrestin 1-dependent regulation of Rap2 is required for fMLP-stimulated chemotaxis in neutrophil-like HL-60 cells.β-arrestin 2 attenuates cardiac dysfunction in polymicrobial sepsis through gp130 and p38.MicroRNA-155 attenuates late sepsis-induced cardiac dysfunction through JNK and β-arrestin 2.Loss of β-arrestin 2 exacerbates experimental autoimmune encephalomyelitis with reduced number of Foxp3+ CD4+ regulatory T cells.Immunoreceptor tyrosine-based inhibitory motif-dependent functions of an MHC class I-specific NK cell receptor.

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An essential function for beta-arrestin 2 in the inhibitory signaling of natural killer cells

http://www.wikidata.org/entity/Q24310297

description

2008 nî lūn-bûn

@nan

2008 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2008 թվականի օգոստոսին հրատարակված գիտական հոդված

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年论文

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An essential function for beta ...... naling of natural killer cells

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An essential function for beta ...... naling of natural killer cells

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Nature Immunology

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Bao-Xue Ge

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Dangsheng Li

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Gang Pei

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Hai-Peng Liu

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Jack L Strominger

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Jing-Wu Zang

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Jun-Hao Hu

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Lei Sun

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Lin Zou

http://www.w3.org/2001/XMLSchema#string

Ling Kong

http://www.w3.org/2001/XMLSchema#string

P2860

Pivotal role of phosphoinositide-3 kinase in regulation of cytotoxicity in natural killer cells

Association of beta-arrestin and TRAF6 negatively regulates Toll-like receptor-interleukin 1 receptor signaling

Phosphotyrosines in the killer cell inhibitory receptor motif of NKB1 are required for negative signaling and for association with protein tyrosine phosphatase 1C

Cloning of immunoglobulin-superfamily members associated with HLA-C and HLA-B recognition by human natural killer cells

Recruitment of tyrosine phosphatase HCP by the killer cell inhibitor receptor

Beta-arrestin 2 mediates endocytosis of type III TGF-beta receptor and down-regulation of its signaling

The human natural killer cell immune synapse

The selective downregulation of class I major histocompatibility complex proteins by HIV-1 protects HIV-infected cells from NK cells

Development of lupus-like autoimmune diseases by disruption of the PD-1 gene encoding an ITIM motif-carrying immunoreceptor

KIR2DL4 (CD158d), an NK cell-activating receptor with inhibitory potential

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898-907

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4875044

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10.1038/NI.1635

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8

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9

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2008-08-01T00:00:00Z

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5247220

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1051406071

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Arrestin beta 2

Killer cell immunoglobulin like receptor, two Ig domains and long cytoplasmic tail 1