Down-regulation of a serine protease, myeloblastin, causes growth arrest and differentiation of promyelocytic leukemia cells
about
Human dipeptidyl-peptidase I. Gene characterization, localization, and expressionWegener autoantigen and myeloblastin are encoded by a single mRNAThree human elastase-like genes coordinately expressed in the myelomonocyte lineage are organized as a single genetic locus on 19pterCloning of cDNA for proteinase 3: a serine protease, antibiotic, and autoantigen from human neutrophilsProteinase-3 as the major autoantigen of c-ANCA is strongly expressed in lung tissue of patients with Wegener's granulomatosis.Autoimmunity and glomerulonephritisThe antigenic significance and methods of detection of the anti-neutrophil cytoplasmic autoantibodies (ANCA)Substrate and inhibitor studies on proteinase 3Internalization of proteinase 3 is concomitant with endothelial cell apoptosis and internalization of myeloperoxidase with generation of intracellular oxidantsDysregulation of autoantigen genes in ANCA-associated vasculitis involves alternative transcripts and new protein synthesisSecretory granule proteases in rat mast cells. Cloning of 10 different serine proteases and a carboxypeptidase A from various rat mast cell populationsProteinase 3 and neutrophil elastase enhance inflammation in mice by inactivating antiinflammatory progranulin.Cleavage of p21/WAF1/CIP1 by proteinase 3 modulates differentiation of a monocytic cell line. Molecular analysis of the cleavage site.Isolation, characterization, and transcription of the gene encoding mouse mast cell protease 7Kinetics of the inhibition of proteinase 3 by elafin.Structures of human proteinase 3 and neutrophil elastase--so similar yet so different.PR1-specific T cells are associated with unmaintained cytogenetic remission of chronic myelogenous leukemia after interferon withdrawal.Inhibition of lymphocyte mediated cytotoxicity by perforin antisense oligonucleotides.Neutrophil elastase, proteinase 3, and cathepsin G as therapeutic targets in human diseases.Adoptive transfer of unselected or leukemia-reactive T-cells in the treatment of relapse following allogeneic hematopoietic cell transplantation.Expression profiling of CD34+ hematopoietic stem/ progenitor cells reveals distinct subtypes of therapy-related acute myeloid leukemia.Intracellular serpins in haemopoietic and peripheral blood cells.Testing for antineutrophil cytoplasmic antibodies.Neutrophil surface presentation of the anti-neutrophil cytoplasmic antibody-antigen proteinase 3 depends on N-terminal processing.The basis of T-cell-mediated immunity to chronic myelogenous leukemia.Therapeutic applications of oligonucleotides.Characterization of immunologic properties of a second HLA-A2 epitope from a granule protease in CML patients and HLA-A2 transgenic miceB cell epitope specificity in ANCA-associated vasculitis: does it matter?Fundamental and clinical aspects of anti neutrophil cytoplasmic antibodies (ANCA).Role of saliva proteinase 3 in dental cariesProteinase 3 on apoptotic cells disrupts immune silencing in autoimmune vasculitisVitamin D3 upregulated protein 1 (VDUP1) is a regulator for redox signaling and stress-mediated diseases.Colony-stimulating factor 1 expression is down-regulated during the adipocyte differentiation of H-1/A marrow stromal cells and induced by cachectin/tumor necrosis factorInduction of a cytoplasmic activator of DNA synthesis in lymphocytes is mediated through a membrane-associated protein kinase.Translational mini-review series on vaccines: Peptide vaccines for myeloid leukaemias.Myeloblastin is a granulocyte colony-stimulating factor-responsive gene conferring factor-independent growth to hematopoietic cells.Characterizing and optimizing immune responses to leukaemia antigens after allogeneic stem cell transplantation.Immunotherapy for myeloid leukemias: current status and future directions.The lost intrinsic fragmentation of MAT1 protein during granulopoiesis promotes the growth and metastasis of leukemic myeloblasts.PR1 vaccination in myeloid malignancies.
P2860
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P2860
Down-regulation of a serine protease, myeloblastin, causes growth arrest and differentiation of promyelocytic leukemia cells
description
1989 nî lūn-bûn
@nan
1989 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
1989 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
1989年の論文
@ja
1989年論文
@yue
1989年論文
@zh-hant
1989年論文
@zh-hk
1989年論文
@zh-mo
1989年論文
@zh-tw
1989年论文
@wuu
name
Down-regulation of a serine pr ...... f promyelocytic leukemia cells
@ast
Down-regulation of a serine pr ...... f promyelocytic leukemia cells
@en
Down-regulation of a serine pr ...... f promyelocytic leukemia cells
@en-gb
Down-regulation of a serine pr ...... f promyelocytic leukemia cells
@nl
type
label
Down-regulation of a serine pr ...... f promyelocytic leukemia cells
@ast
Down-regulation of a serine pr ...... f promyelocytic leukemia cells
@en
Down-regulation of a serine pr ...... f promyelocytic leukemia cells
@en-gb
Down-regulation of a serine pr ...... f promyelocytic leukemia cells
@nl
prefLabel
Down-regulation of a serine pr ...... f promyelocytic leukemia cells
@ast
Down-regulation of a serine pr ...... f promyelocytic leukemia cells
@en
Down-regulation of a serine pr ...... f promyelocytic leukemia cells
@en-gb
Down-regulation of a serine pr ...... f promyelocytic leukemia cells
@nl
P2093
P921
P1433
P1476
Down-regulation of a serine pr ...... f promyelocytic leukemia cells
@en
P2093
P304
P356
10.1016/0092-8674(89)90752-6
P407
P577
1989-12-22T00:00:00Z