BOD1L Is Required to Suppress Deleterious Resection of Stressed Replication Forks
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Replication-Associated Recombinational Repair: Lessons from Budding YeastPolymerase δ replicates both strands after homologous recombination-dependent fork restartA Selective Small Molecule DNA2 Inhibitor for Sensitization of Human Cancer Cells to ChemotherapyNEK8 regulates DNA damage-induced RAD51 foci formation and replication fork protectionReplication intermediates that escape Dna2 activity are processed by Holliday junction resolvase Yen1.Abro1 maintains genome stability and limits replication stress by protecting replication fork stability.Protection or resection: BOD1L as a novel replication fork protection factor.FANCI-FANCD2 stabilizes the RAD51-DNA complex by binding RAD51 and protects the 5'-DNA end.WRNIP1 protects stalled forks from degradation and promotes fork restart after replication stress.Elevated PTTG and PBF predicts poor patient outcome and modulates DNA damage response genes in thyroid cancer.Combining electron microscopy with single molecule DNA fiber approaches to study DNA replication dynamics.Smarcal1-Mediated Fork Reversal Triggers Mre11-Dependent Degradation of Nascent DNA in the Absence of Brca2 and Stable Rad51 Nucleofilaments.Mutations in DONSON disrupt replication fork stability and cause microcephalic dwarfismThe endonuclease EEPD1 mediates synthetic lethality in RAD52-depleted BRCA1 mutant breast cancer cells.The end-joining factor Ku acts in the end-resection of double strand break-free arrested replication forks.Megakaryocytes in Myeloproliferative Neoplasms Have Unique Somatic Mutations.Around and beyond 53BP1 Nuclear Bodies.Rad9/53BP1 protects stalled replication forks from degradation in Mec1/ATR-defective cells.Restoration of Replication Fork Stability in BRCA1- and BRCA2-Deficient Cells by Inactivation of SNF2-Family Fork Remodelers.BLM helicase regulates DNA repair by counteracting RAD51 loading at DNA double-strand break sites.53BP1 Mediates ATR-Chk1 Signaling And Protects Replication Forks Under Conditions Of Replication Stress.PARP1 and PARP2 stabilise replication forks at base excision repair intermediates through Fbh1-dependent Rad51 regulation.SIRF: Quantitative in situ analysis of protein interactions at DNA replication forks.Mechanisms of Oncogene-Induced Replication Stress: Jigsaw Falling into Place.Strong antitumor synergy between DNA crosslinking and HSP90 inhibition causes massive premitotic DNA fragmentation in ovarian cancer cells.Transcriptional Regulation of Emergency Granulopoiesis in Leukemia.Histone Methylation by SETD1A Protects Nascent DNA through the Nucleosome Chaperone Activity of FANCD2.RPA and RAD51: fork reversal, fork protection, and genome stabilityPreserving replication fork integrity and competence via the homologous recombination pathwayProcessing of DNA Ends in the Maintenance of Genome Stability
P2860
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P2860
BOD1L Is Required to Suppress Deleterious Resection of Stressed Replication Forks
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2015 nî lūn-bûn
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2015 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
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2015 թվականի օգոստոսին հրատարակված գիտական հոդված
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2015年の論文
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2015年論文
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2015年論文
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2015年論文
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2015年論文
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2015年論文
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2015年论文
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BOD1L Is Required to Suppress Deleterious Resection of Stressed Replication Forks
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BOD1L Is Required to Suppress Deleterious Resection of Stressed Replication Forks
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BOD1L Is Required to Suppress Deleterious Resection of Stressed Replication Forks
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BOD1L Is Required to Suppress Deleterious Resection of Stressed Replication Forks
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type
label
BOD1L Is Required to Suppress Deleterious Resection of Stressed Replication Forks
@ast
BOD1L Is Required to Suppress Deleterious Resection of Stressed Replication Forks
@en
BOD1L Is Required to Suppress Deleterious Resection of Stressed Replication Forks
@en-gb
BOD1L Is Required to Suppress Deleterious Resection of Stressed Replication Forks
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BOD1L Is Required to Suppress Deleterious Resection of Stressed Replication Forks
@ast
BOD1L Is Required to Suppress Deleterious Resection of Stressed Replication Forks
@en
BOD1L Is Required to Suppress Deleterious Resection of Stressed Replication Forks
@en-gb
BOD1L Is Required to Suppress Deleterious Resection of Stressed Replication Forks
@nl
P2093
P50
P3181
P1433
P1476
BOD1L Is Required to Suppress Deleterious Resection of Stressed Replication Forks
@en
P2093
Anastasia Zlatanou
Andrew N Blackford
Edward S Miller
Ellis L Ryan
Jadwiga Nieminuszczy
John J Reynolds
Martin R Higgs
Nicholas J Davies
Simon J Boulton
Tatjana Stankovic
P304
P3181
P356
10.1016/J.MOLCEL.2015.06.007
P407
P577
2015-08-06T00:00:00Z