The probability of in vivo reactivation of herpes simplex virus type 1 increases with the number of latently infected neurons in the ganglia.
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B-virus (Cercopithecine herpesvirus 1) infection in humans and macaques: potential for zoonotic diseaseA comparison of herpes simplex virus type 1 and varicella-zoster virus latency and reactivationOcular herpes simplex virus: how are latency, reactivation, recurrent disease and therapy interrelated?Herpes simplex virus and the lexicon of latency and reactivation: a call for defining terms and building an integrated collective frameworkLaser-capture microdissection: refining estimates of the quantity and distribution of latent herpes simplex virus 1 and varicella-zoster virus DNA in human trigeminal Ganglia at the single-cell level.De novo synthesis of VP16 coordinates the exit from HSV latency in vivo.Immunization with a dominant-negative recombinant Herpes Simplex Virus (HSV) type 1 protects against HSV-2 genital disease in guinea pigs.Repeated social stress enhances the innate immune response to a primary HSV-1 infection in the cornea and trigeminal ganglia of Balb/c miceReplication of herpes simplex virus type 1 within trigeminal ganglia is required for high frequency but not high viral genome copy number latency.Optimized viral dose and transient immunosuppression enable herpes simplex virus ICP0-null mutants To establish wild-type levels of latency in vivo.Lack of interleukin-6 (IL-6) enhances susceptibility to infection but does not alter latency or reactivation of herpes simplex virus type 1 in IL-6 knockout miceQuantitation of latent varicella-zoster virus and herpes simplex virus genomes in human trigeminal ganglia.Enhancer and long-term expression functions of herpes simplex virus type 1 latency-associated promoter are both located in the same regionLatency, chromatin remodeling, and reactivation of human cytomegalovirus in the dendritic cells of healthy carriers.Recent progress in herpes simplex virus immunobiology and vaccine researchEfficacies of gel formulations containing foscarnet, alone or combined with sodium lauryl sulfate, against establishment and reactivation of latent herpes simplex virus type 1HSV-1-based vectors for gene therapy of neurological diseases and brain tumors: part I. HSV-1 structure, replication and pathogenesis.Inflation and long-term maintenance of CD8 T cells responding to a latent herpesvirus depend upon establishment of latency and presence of viral antigens.Herpes simplex virus DNA synthesis is not a decisive regulatory event in the initiation of lytic viral protein expression in neurons in vivo during primary infection or reactivation from latency.In vivo reactivation of latent herpes simplex virus 1 in mice can occur in the brain before occurring in the trigeminal ganglionHSV-1 genome subnuclear positioning and associations with host-cell PML-NBs and centromeres regulate LAT locus transcription during latency in neurons.Failure of thymidine kinase-negative herpes simplex virus to reactivate from latency following efficient establishment.Varicella zoster virus latency.Establishment and maintenance of gammaherpesvirus latency are independent of infective dose and route of infectionEstablishment of HSV1 latency in immunodeficient mice facilitates efficient in vivo reactivation.CMX001 potentiates the efficacy of acyclovir in herpes simplex virus infectionsC21orf91 genotypes correlate with herpes simplex labialis (cold sore) frequency: description of a cold sore susceptibility geneOpposing biological functions of tryptophan catabolizing enzymes during intracellular infection.Role of ICP0 in the strategy of conquest of the host cell by herpes simplex virus 1Cross-presentation and genome-wide screening reveal candidate T cells antigens for a herpes simplex virus type 1 vaccine.Rates of reactivation of latent herpes simplex virus from mouse trigeminal ganglia ex vivo correlate directly with viral load and inversely with number of infiltrating CD8+ T cells.The HSV-1 Latency-Associated Transcript Functions to Repress Latent Phase Lytic Gene Expression and Suppress Virus Reactivation from Latently Infected NeuronsCD8 T cell control of HSV reactivation from latency is abrogated by viral inhibition of MHC class IImmunization with a replication-defective herpes simplex virus 2 mutant reduces herpes simplex virus 1 infection and prevents ocular disease.A CD8+ T cell immune evasion protein specific to Epstein-Barr virus and its close relatives in Old World primates.The number of herpes simplex virus-infected neurons and the number of viral genome copies per neuron correlate with the latent viral load in gangliaHerpes simplex virus 1 tropism for human sensory ganglion neurons in the severe combined immunodeficiency mouse model of neuropathogenesisDNA damage promotes herpes simplex virus-1 protein expression in a neuroblastoma cell line.Vaxfectin-adjuvanted plasmid DNA vaccine improves protection and immunogenicity in a murine model of genital herpes infectionImpairment in reactivation of a latency associated transcript (LAT)-deficient HSV-2 is not solely dependent on the latent viral load or the number of CD8(+) T cells infiltrating the ganglia.
P2860
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P2860
The probability of in vivo reactivation of herpes simplex virus type 1 increases with the number of latently infected neurons in the ganglia.
description
1998 nî lūn-bûn
@nan
1998 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
1998 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
1998年の論文
@ja
1998年論文
@yue
1998年論文
@zh-hant
1998年論文
@zh-hk
1998年論文
@zh-mo
1998年論文
@zh-tw
1998年论文
@wuu
name
The probability of in vivo rea ...... nfected neurons in the ganglia
@nl
The probability of in vivo rea ...... fected neurons in the ganglia.
@ast
The probability of in vivo rea ...... fected neurons in the ganglia.
@en
The probability of in vivo rea ...... fected neurons in the ganglia.
@en-gb
type
label
The probability of in vivo rea ...... nfected neurons in the ganglia
@nl
The probability of in vivo rea ...... fected neurons in the ganglia.
@ast
The probability of in vivo rea ...... fected neurons in the ganglia.
@en
The probability of in vivo rea ...... fected neurons in the ganglia.
@en-gb
prefLabel
The probability of in vivo rea ...... nfected neurons in the ganglia
@nl
The probability of in vivo rea ...... fected neurons in the ganglia.
@ast
The probability of in vivo rea ...... fected neurons in the ganglia.
@en
The probability of in vivo rea ...... fected neurons in the ganglia.
@en-gb
P2860
P3181
P1433
P1476
The probability of in vivo rea ...... fected neurons in the ganglia.
@en
P2093
N M Sawtell
P2860
P304
P3181
P407
P577
1998-08-01T00:00:00Z