Signaling disrupts mSin3A binding to the Mad1-like Sin3-interacting domain of TIEG2, an Sp1-like repressor
about
A functional family-wide screening of SP/KLF proteins identifies a subset of suppressors of KRAS-mediated cell growthKruppel-like transcription factor 13 regulates T lymphocyte survival in vivoSp1- and Krüppel-like transcription factorsMnt transcriptional repressor is functionally regulated during cell cycle progressionSplenomegaly and modified erythropoiesis in KLF13-/- miceThe zinc finger transcription factor transforming growth factor beta-inducible early gene-1 confers myeloid-specific activation of the leukocyte integrin CD11d promoter.Wnt-mediated down-regulation of Sp1 target genes by a transcriptional repressor Sp5.Single and combinatorial chromatin coupling events underlies the function of transcript factor Krüppel-like factor 11 in the regulation of gene networks.Distinct role of Kruppel-like factor 11 in the regulation of prostaglandin E2 biosynthesis.Transcriptional activity and nuclear localization of Cabut, the Drosophila ortholog of vertebrate TGF-β-inducible early-response gene (TIEG) proteins.Differential coupling of KLF10 to Sin3-HDAC and PCAF regulates the inducibility of the FOXP3 geneMolecular mechanisms of the suppression of axon regeneration by KLF transcription factors.KLF11-mediated repression antagonizes Sp1/sterol-responsive element-binding protein-induced transcriptional activation of caveolin-1 in response to cholesterol signaling.Functional role of KLF10 in multiple disease processesKrüppel-like factor KLF9 regulates PPARγ transactivation at the middle stage of adipogenesis.An mSin3A interaction domain links the transcriptional activity of KLF11 with its role in growth regulation.The family feud: turning off Sp1 by Sp1-like KLF proteins.Protein kinase Calpha-induced derepression of the human luteinizing hormone receptor gene transcription through ERK-mediated release of HDAC1/Sin3A repressor complex from Sp1 sites.NFAT-induced histone acetylation relay switch promotes c-Myc-dependent growth in pancreatic cancer cells.CIITA mediates interferon-gamma repression of collagen transcription through phosphorylation-dependent interactions with co-repressor molecules.Functional analysis of the Mad1-mSin3A repressor-corepressor interaction reveals determinants of specificity, affinity, and transcriptional response.CUTL1 is phosphorylated by protein kinase A, modulating its effects on cell proliferation and motility.Bone morphogenetic protein 2 opposes Shh-mediated proliferation in cerebellar granule cells through a TIEG-1-based regulation of Nmyc.The potential of targeting Sin3B and its associated complexes for cancer therapy.
P2860
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P2860
Signaling disrupts mSin3A binding to the Mad1-like Sin3-interacting domain of TIEG2, an Sp1-like repressor
description
2002 nî lūn-bûn
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2002 թուականի Մայիսին հրատարակուած գիտական յօդուած
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2002 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2002年の論文
@ja
2002年論文
@yue
2002年論文
@zh-hant
2002年論文
@zh-hk
2002年論文
@zh-mo
2002年論文
@zh-tw
2002年论文
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name
Signaling disrupts mSin3A bind ...... f TIEG2, an Sp1-like repressor
@ast
Signaling disrupts mSin3A bind ...... f TIEG2, an Sp1-like repressor
@en
Signaling disrupts mSin3A bind ...... f TIEG2, an Sp1-like repressor
@nl
type
label
Signaling disrupts mSin3A bind ...... f TIEG2, an Sp1-like repressor
@ast
Signaling disrupts mSin3A bind ...... f TIEG2, an Sp1-like repressor
@en
Signaling disrupts mSin3A bind ...... f TIEG2, an Sp1-like repressor
@nl
prefLabel
Signaling disrupts mSin3A bind ...... f TIEG2, an Sp1-like repressor
@ast
Signaling disrupts mSin3A bind ...... f TIEG2, an Sp1-like repressor
@en
Signaling disrupts mSin3A bind ...... f TIEG2, an Sp1-like repressor
@nl
P2093
P2860
P356
P1433
P1476
Signaling disrupts mSin3A bind ...... f TIEG2, an Sp1-like repressor
@en
P2093
Jin-San Zhang
Joanna Kaczynski
Raul Urrutia
Volker Ellenrieder
P2860
P304
P356
10.1093/EMBOJ/21.10.2451
P407
P577
2002-05-01T00:00:00Z