The low molecular mass GTP-binding protein Rho is affected by toxin A from Clostridium difficile
about
Clostridium difficile toxins: mechanism of action and role in disease.Rho protein regulates tight junctions and perijunctional actin organization in polarized epitheliaClostridium difficile Toxins A and B: Insights into Pathogenic Properties and Extraintestinal EffectsMicrobial sphingomyelinase induces RhoA-mediated reorganization of the apical brush border membrane and is protective against invasion.In vivo covalent cross-linking of cellular actin by the Vibrio cholerae RTX toxinContribution of adenosine A(2B) receptors in Clostridium difficile intoxication and infection.Enteric bacterial toxins: mechanisms of action and linkage to intestinal secretion.Effects of toxin A from Clostridium difficile on mast cell activation and survival.Clostridium difficile toxin B differentially affects GPCR-stimulated Ca2+ responses in macrophages: independent roles for Rho and PLA2.Production of alpha 1,3-galactosyltransferase-deficient pigs.Coronaviruses induce entry-independent, continuous macropinocytosisClostridium difficile toxin A decreases acetylation of tubulin, leading to microtubule depolymerization through activation of histone deacetylase 6, and this mediates acute inflammation.Glucosyltransferases of viridans group streptococci modulate interleukin-6 and adhesion molecule expression in endothelial cells and augment monocytic cell adherence.Utility of Clostridium difficile toxin B for inducing anti-tumor immunity.Translocation domain mutations affecting cellular toxicity identify the Clostridium difficile toxin B pore.Critical roles of Clostridium difficile toxin B enzymatic activities in pathogenesis.The flagellin FliC of Clostridium difficile is responsible for pleiotropic gene regulation during in vivo infection.Effects of Clostridium difficile toxin A and toxin B on phospholipase D activation in human promyelocytic leukemic HL60 cells.Cholesterol-dependent actin remodeling via RhoA and Rac1 activation by the Streptococcus pneumoniae toxin pneumolysinObservations on the Role of TcdE Isoforms in Clostridium difficile Toxin SecretionColonization Resistance of the Gut Microbiota against Clostridium difficile.Rabbit sucrase-isomaltase contains a functional intestinal receptor for Clostridium difficile toxin A.Novel therapeutic strategies for Clostridium difficile infections.Pathogenic effects of glucosyltransferase from Clostridium difficile toxins.Old and new models for studying host-microbe interactions in health and disease: C. difficile as an example.Tumor necrosis factor-alpha induces stress fiber formation through ceramide production: role of sphingosine kinase.Clostridium difficile toxins A and B: Receptors, pores, and translocation into cells.Lysine and polyamines are substrates for transglutamination of Rho by the Bordetella dermonecrotic toxinClostridium difficile infection of the gutGlucosyltransferase Activity of Clostridium difficile Toxin B Triggers Autophagy-mediated Cell Growth Arrest.A Novel Fic (Filamentation Induced by cAMP) Protein from Clostridium difficile Reveals an Inhibitory Motif-independent Adenylylation/AMPylation Mechanism.Mechanism of lovastatin-induced apoptosis in intestinal epithelial cells.Clostridium difficile Toxins TcdA and TcdB Cause Colonic Tissue Damage by Distinct Mechanisms.Inhibition of Fc epsilon-RI-mediated activation of rat basophilic leukemia cells by Clostridium difficile toxin B (monoglucosyltransferase)UDP-glucose deficiency in a mutant cell line protects against glucosyltransferase toxins from Clostridium difficile and Clostridium sordellii.Interleukin-22 and CD160 play additive roles in the host mucosal response to Clostridium difficile infection in mice.Inositol hexakisphosphate-dependent processing of Clostridium sordellii lethal toxin and Clostridium novyi alpha-toxin.Gln 63 of Rho is deamidated by Escherichia coli cytotoxic necrotizing factor-1.A Crk-II/TC10 signaling pathway is required for osmotic shock-stimulated glucose transport.A Nutrient-Regulated Cyclic Diguanylate Phosphodiesterase Controls Clostridium difficile Biofilm and Toxin Production during Stationary Phase.
P2860
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P2860
The low molecular mass GTP-binding protein Rho is affected by toxin A from Clostridium difficile
description
1995 nî lūn-bûn
@nan
1995 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
1995 թվականի մարտին հրատարակված գիտական հոդված
@hy
1995年の論文
@ja
1995年論文
@yue
1995年論文
@zh-hant
1995年論文
@zh-hk
1995年論文
@zh-mo
1995年論文
@zh-tw
1995年论文
@wuu
name
The low molecular mass GTP-bin ...... n A from Clostridium difficile
@ast
The low molecular mass GTP-bin ...... n A from Clostridium difficile
@en
The low molecular mass GTP-bin ...... n A from Clostridium difficile
@nl
type
label
The low molecular mass GTP-bin ...... n A from Clostridium difficile
@ast
The low molecular mass GTP-bin ...... n A from Clostridium difficile
@en
The low molecular mass GTP-bin ...... n A from Clostridium difficile
@nl
prefLabel
The low molecular mass GTP-bin ...... n A from Clostridium difficile
@ast
The low molecular mass GTP-bin ...... n A from Clostridium difficile
@en
The low molecular mass GTP-bin ...... n A from Clostridium difficile
@nl
P2093
P2860
P3181
P356
P1476
The low molecular mass GTP-bin ...... n A from Clostridium difficile
@en
P2093
C von Eichel-Streiber
K Aktories
P2860
P304
P3181
P356
10.1172/JCI117747
P407
P577
1995-03-01T00:00:00Z