The requirement for Phr1 in CNS axon tract formation reveals the corticostriatal boundary as a choice point for cortical axons
about
Regulation of Rap2A by the ubiquitin ligase Nedd4-1 controls neurite developmentE3 ligase Nedd4 promotes axon branching by downregulating PTENAbnormal development of the neuromuscular junction in Nedd4-deficient miceThe PHR proteins: intracellular signaling hubs in neuronal development and axon degenerationThe DLK signalling pathway--a double-edged sword in neural development and regenerationThe Nesprin family member ANC-1 regulates synapse formation and axon termination by functioning in a pathway with RPM-1 and β-CateninThe Highwire ubiquitin ligase promotes axonal degeneration by tuning levels of Nmnat proteinStructures of PHR Domains from Mus musculus Phr1 (Mycbp2) Explain the Loss-of-Function Mutation (Gly1092→Glu) of the C. elegans Ortholog RPM-1RPM-1 uses both ubiquitin ligase and phosphatase-based mechanisms to regulate DLK-1 during neuronal developmentFbxo45 forms a novel ubiquitin ligase complex and is required for neuronal developmentZPK/DLK and MKK4 form the critical gateway to axotomy-induced motoneuron death in neonatesDevelopmental Function of the PHR Protein RPM-1 Is Required for Learning in Caenorhabditis elegans.The adenomatous polyposis coli protein is an essential regulator of radial glial polarity and construction of the cerebral cortex.The DLK-1 kinase promotes mRNA stability and local translation in C. elegans synapses and axon regenerationGenetic evidence that Celsr3 and Celsr2, together with Fzd3, regulate forebrain wiring in a Vangl-independent mannerA ubiquitin E2 variant protein acts in axon termination and synaptogenesis in Caenorhabditis elegansFbxo45 inhibits calcium-sensitive proteolysis of N-cadherin and promotes neuronal differentiation.A forward genetic screen in mice identifies mutants with abnormal cortical patterning.Identification of a peptide inhibitor of the RPM-1 · FSN-1 ubiquitin ligase complexBimodal control of dendritic and axonal growth by the dual leucine zipper kinase pathway.Ubiquitination in postsynaptic function and plasticityDrosophila Rae1 controls the abundance of the ubiquitin ligase Highwire in post-mitotic neurons.In vitro interaction between coxsackievirus B3 VP1 protein and human pleckstrin homology domain retinal protein (PHR1).Nonspecific labeling limits the utility of Cre-Lox bred CST-YFP mice for studies of corticospinal tract regenerationThe E3 ubiquitin ligase protein associated with Myc (Pam) regulates mammalian/mechanistic target of rapamycin complex 1 (mTORC1) signaling in vivo through N- and C-terminal domains.Highwire regulates guidance of sister axons in the Drosophila mushroom body.MYCBP2 Is a Guanosine Exchange Factor for Ran Protein and Determines Its Localization in Neurons of Dorsal Root Ganglia.Molecular control of axon branching.Identification of potential mediators of retinotopic mapping: a comparative proteomic analysis of optic nerve from WT and Phr1 retinal knockout miceBreaking it down: the ubiquitin proteasome system in neuronal morphogenesis.Functional genomic screening identifies dual leucine zipper kinase as a key mediator of retinal ganglion cell death.Lack of association of rare functional variants in TSC1/TSC2 genes with autism spectrum disorderPam (Protein associated with Myc) functions as an E3 ubiquitin ligase and regulates TSC/mTOR signalingSpatial organization of ubiquitin ligase pathways orchestrates neuronal connectivity.Attenuation of insulin signalling contributes to FSN-1-mediated regulation of synapse development.Independent pathways downstream of the Wnd/DLK MAPKKK regulate synaptic structure, axonal transport, and injury signalingRoles of ubiquitination at the synapse.Pathfinding of corticothalamic axons relies on a rendezvous with thalamic projectionsBuilding a synapse: lessons on synaptic specificity and presynaptic assembly from the nematode C. elegansAutophagy linked FYVE (Alfy/WDFY3) is required for establishing neuronal connectivity in the mammalian brain
P2860
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P2860
The requirement for Phr1 in CNS axon tract formation reveals the corticostriatal boundary as a choice point for cortical axons
description
2007 nî lūn-bûn
@nan
2007 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2007 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
name
The requirement for Phr1 in CN ...... hoice point for cortical axons
@ast
The requirement for Phr1 in CN ...... hoice point for cortical axons
@en
The requirement for Phr1 in CN ...... hoice point for cortical axons
@nl
type
label
The requirement for Phr1 in CN ...... hoice point for cortical axons
@ast
The requirement for Phr1 in CN ...... hoice point for cortical axons
@en
The requirement for Phr1 in CN ...... hoice point for cortical axons
@nl
prefLabel
The requirement for Phr1 in CN ...... hoice point for cortical axons
@ast
The requirement for Phr1 in CN ...... hoice point for cortical axons
@en
The requirement for Phr1 in CN ...... hoice point for cortical axons
@nl
P2093
P2860
P3181
P356
P1433
P1476
The requirement for Phr1 in CN ...... hoice point for cortical axons
@en
P2093
A Joseph Bloom
Bradley R Miller
Joshua R Sanes
P2860
P304
P3181
P356
10.1101/GAD.1592107
P407
P577
2007-09-27T00:00:00Z