Absolute dependence on kappa B responsive elements for initiation and Tat-mediated amplification of HIV transcription in blood CD4 T lymphocytes
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Hostile takeovers: viral appropriation of the NF-kB pathwayNitric oxide inhibits HIV tat-induced NF-kB activationAmplification of the inflammatory cellular redox state by human immunodeficiency virus type 1-immunosuppressive tat and gp160 proteinsMycobacterium avium complex augments macrophage HIV-1 production and increases CCR5 expressionModulation of human immunodeficiency virus 1 replication by interferon regulatory factorsTransdominant mutants of I kappa B alpha block Tat-tumor necrosis factor synergistic activation of human immunodeficiency virus type 1 gene expression and virus multiplicationActivation of transcription factor NF-kappaB by the Tat protein of human immunodeficiency virus type 1Extracellular human immunodeficiency virus type 1 Tat protein is associated with an increase in both NF-kappa B binding and protein kinase C activity in primary human astrocytesA Tat-induced auto-up-regulatory loop for superactivation of the human immunodeficiency virus type 1 promoterInhibition of HIV-1 replication in primary human monocytes by the IkappaB-alphaS32/36A repressor of NF-kappaBCrystal Structure of NFAT Bound to the HIV-1 LTR Tandem κB Enhancer ElementStructural Basis of HIV-1 Activation by NF-κB—A Higher-Order Complex of p50:RelA Bound to the HIV-1 LTRInhibition of phosphodiesterase type IV suppresses human immunodeficiency virus type 1 replication and cytokine production in primary T cells: involvement of NF-kappaB and NFATRegulation of human immunodeficiency virus type 1 replication in human T lymphocytes by nitric oxideThe roles of HIV-1 proteins and antiretroviral drug therapy in HIV-1-associated endothelial dysfunctionThe cytokine network of acute HIV infection: a promising target for vaccines and therapy to reduce viral set-point?Basal shuttle of NF-kappaB/I kappaB alpha in resting T lymphocytes regulates HIV-1 LTR dependent expression.Combinatorial signals from CD28 differentially regulate human immunodeficiency virus transcription in T cellsThe CCR5-antagonist Maraviroc reverses HIV-1 latency in vitro alone or in combination with the PKC-agonist Bryostatin-1Plasma membrane-targeted Raf kinase activates NF-kappaB and human immunodeficiency virus type 1 replication in T lymphocytesNaturally occurring human immunodeficiency virus type 1 long terminal repeats have a frequently observed duplication that binds RBF-2 and represses transcription.Mechanisms of HIV Transcriptional Regulation by Drugs of Abuse.Genetic variation and HIV-associated neurologic disease.NF-kappaB cis-acting motifs of the human immunodeficiency virus (HIV) long terminal repeat regulate HIV transcription in human macrophages.Modifications in host cell cytoskeleton structure and function mediated by intracellular HIV-1 Tat protein are greatly dependent on the second coding exonReciprocal modulatory interaction between human immunodeficiency virus type 1 Tat and transcription factor NFAT1.Innate immune recognition and activation during HIV infection.Transcriptional and posttranscriptional regulation of HIV-1 gene expressionSeries introduction: the transcription factor NF-kappaB and human diseaseMicroarray analysis of HIV resistant female sex workers reveal a gene expression signature pattern reminiscent of a lowered immune activation state.An analog of the natural steroidal alkaloid cortistatin A potently suppresses Tat-dependent HIV transcription.The tale of the long tail: the cytoplasmic domain of HIV-1 gp41.RelB-p50 NF-kappa B complexes are selectively induced by cytomegalovirus immediate-early protein 1: differential regulation of Bcl-x(L) promoter activity by NF-kappa B family membersTopoisomerase poisons activate the transcription factor NF-kappaB in ACH-2 and CEM cells.Protein kinase Ctheta is a specific target for inhibition of the HIV type 1 replication in CD4+ T lymphocytes.RETRACTED: MAVS, cGAS, and endogenous retroviruses in T-independent B cell responses.Expression and reactivation of HIV in a chemokine induced model of HIV latency in primary resting CD4+ T cells.The cytoplasmic domain of the HIV-1 glycoprotein gp41 induces NF-κB activation through TGF-β-activated kinase 1.Human immunodeficiency virus type 1 long terminal repeat variants from 42 patients representing all stages of infection display a wide range of sequence polymorphism and transcription activity.Cooperation between herpes simplex virus type 1-encoded ICP0 and Tat to support transcription of human immunodeficiency virus type 1 long terminal repeat in vivo can occur in the absence of the TAR binding site.
P2860
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P2860
Absolute dependence on kappa B responsive elements for initiation and Tat-mediated amplification of HIV transcription in blood CD4 T lymphocytes
description
1995 nî lūn-bûn
@nan
1995 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
1995 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
1995年の論文
@ja
1995年論文
@yue
1995年論文
@zh-hant
1995年論文
@zh-hk
1995年論文
@zh-mo
1995年論文
@zh-tw
1995年论文
@wuu
name
Absolute dependence on kappa B ...... ion in blood CD4 T lymphocytes
@ast
Absolute dependence on kappa B ...... ion in blood CD4 T lymphocytes
@en
Absolute dependence on kappa B ...... ion in blood CD4 T lymphocytes
@nl
type
label
Absolute dependence on kappa B ...... ion in blood CD4 T lymphocytes
@ast
Absolute dependence on kappa B ...... ion in blood CD4 T lymphocytes
@en
Absolute dependence on kappa B ...... ion in blood CD4 T lymphocytes
@nl
prefLabel
Absolute dependence on kappa B ...... ion in blood CD4 T lymphocytes
@ast
Absolute dependence on kappa B ...... ion in blood CD4 T lymphocytes
@en
Absolute dependence on kappa B ...... ion in blood CD4 T lymphocytes
@nl
P2093
P2860
P3181
P1433
P1476
Absolute dependence on kappa B ...... ion in blood CD4 T lymphocytes
@en
P2093
A R Noriega
F Bachelerie
J M Jacqué
L Folgueira
M A Pedraza
R B Gaynor
T Laín de Lera
P2860
P304
P3181
P407
P577
1995-04-03T00:00:00Z