Commensal bacteria regulate Toll-like receptor 3-dependent inflammation after skin injury - Wikidata - awgldk - TriplyDB

Commensal bacteria regulate Toll-like receptor 3-dependent inflammation after skin injury

Commensal bacteria regulate Toll-like receptor 3-dependent inflammation after skin injury

http://www.wikidata.org/entity/Q24600221

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Compartmentalized control of skin immunity by resident commensals Skin microbiome: genomics-based insights into the diversity and role of skin microbes Skin commensals regulate skin immunity The skin microbiome Mechanisms underlying skin disorders induced by EGFR inhibitors The Innate Immune System in Acute and Chronic Wounds Bygiene: The New Paradigm of Bidirectional Hygiene Localization-triggered bacterial pathogenesis Regulation of T Cell Immunity in Atopic Dermatitis by Microbes: The Yin and Yang of Cutaneous Inflammation Microbiology and ecology are vitally important to premedical curricula The skin microbiome: a focus on pathogens and their association with skin disease The skin microbiome: potential for novel diagnostic and therapeutic approaches to cutaneous disease Toll like receptor 3 plays a critical role in the progression and severity of acetaminophen-induced hepatotoxicity Interactions between host factors and the skin microbiome Human Microbiome: When a Friend Becomes an Enemy Papulopustular rosacea, skin immunity and Demodex: pityriasis folliculorum as a missing link Microbial ecology of the skin in the era of metagenomics and molecular microbiology The effects of airway microbiome on corticosteroid responsiveness in asthma Human beta-defensin 3 is up-regulated in cutaneous leprosy type 1 reactions The antimicrobial protein REG3A regulates keratinocyte proliferation and differentiation after skin injury Poly I:C induces collective migration of HaCaT keratinocytes via IL-8.Longitudinal shift in diabetic wound microbiota correlates with prolonged skin defense response.Beneficial and Harmful Interactions of Antibiotics with Microbial Pathogens and the Host Innate Immune System Staphylococcus epidermidis strategies to avoid killing by human neutrophils Role of the microbiota in immunity and inflammation.The effects of MyD88 deficiency on disease phenotype in dysferlin-deficient A/J mice: role of endogenous TLR ligands.Unexplored diversity and strain-level structure of the skin microbiome associated with psoriasis.Skin microbiota: microbial community structure and its potential association with health and disease.Understanding the significance of Staphylococcus epidermidis bacteremia in babies and children Allergic contact dermatitis in psoriasis patients: typical, delayed, and non-interacting.Microbiota in healthy skin and in atopic eczema.c-di-AMP is a new second messenger in Staphylococcus aureus with a role in controlling cell size and envelope stress Activation of TLR2 by a small molecule produced by Staphylococcus epidermidis increases antimicrobial defense against bacterial skin infections Triggering of the dsRNA sensors TLR3, MDA5, and RIG-I induces CD55 expression in synovial fibroblasts Ultraviolet radiation damages self noncoding RNA and is detected by TLR3.TLR2-dependent activation of β-catenin pathway in dendritic cells induces regulatory responses and attenuates autoimmune inflammation.Complement modulates the cutaneous microbiome and inflammatory milieu.Toll-like receptors promote mutually beneficial commensal-host interactions.Functions of the skin microbiota in health and disease.Plasmacytoid dendritic cells sense skin injury and promote wound healing through type I interferons.

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Commensal bacteria regulate Toll-like receptor 3-dependent inflammation after skin injury

http://www.wikidata.org/entity/Q24600221

description

2009 nî lūn-bûn

@nan

2009 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի դեկտեմբերին հրատարակված գիտական հոդված

@hy

2009年の論文

@ja

2009年論文

@yue

2009年論文

@zh-hant

2009年論文

@zh-hk

2009年論文

@zh-mo

2009年論文

@zh-tw

2009年论文

@wuu

name

Commensal bacteria regulate Toll-like receptor 3-dependent inflammation after skin injury

@ast

Commensal bacteria regulate Toll-like receptor 3-dependent inflammation after skin injury

@en

Commensal bacteria regulate Toll-like receptor 3-dependent inflammation after skin injury

@nl

type

label

Commensal bacteria regulate Toll-like receptor 3-dependent inflammation after skin injury

@ast

Commensal bacteria regulate Toll-like receptor 3-dependent inflammation after skin injury

@en

Commensal bacteria regulate Toll-like receptor 3-dependent inflammation after skin injury

@nl

prefLabel

Commensal bacteria regulate Toll-like receptor 3-dependent inflammation after skin injury

@ast

Commensal bacteria regulate Toll-like receptor 3-dependent inflammation after skin injury

@en

Commensal bacteria regulate Toll-like receptor 3-dependent inflammation after skin injury

@nl

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Nature Medicine

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Commensal bacteria regulate Toll-like receptor 3-dependent inflammation after skin injury

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Allen F Ryan

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Anke Leichtle

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Anna L Cogen

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Chun-Ming Huang

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Katherine A Radek

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Lora V Hooper

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Richard R Schmidt

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Sonja von Aulock

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Teruaki Nakatsuji

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Yan Yang

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P2860

A Genomic View of the Human-Bacteroides thetaiotaomicron Symbiosis

Recognition of double-stranded RNA and activation of NF-kappaB by Toll-like receptor 3

Tolerance, danger, and the extended family

Recognition of commensal microflora by toll-like receptors is required for intestinal homeostasis

Chromatin-IgG complexes activate B cells by dual engagement of IgM and Toll-like receptors

How dying cells alert the immune system to danger

Induction of release of tumor necrosis factor from human monocytes by staphylococci and staphylococcal peptidoglycans.

Innate antimicrobial peptide protects the skin from invasive bacterial infection.

Toll-like receptors in systemic autoimmune disease.

Synthesis of glycerol phosphate lipoteichoic acid in Staphylococcus aureus

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10.1038/NM.2062

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12

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15

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40453731

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1048811014

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19966777

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Toll-like receptor

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2880863

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