The dynamics of Ku70/80 and DNA-PKcs at DSBs induced by ionizing radiation is dependent on the complexity of damage - Wikidata - awgldk - TriplyDB

The dynamics of Ku70/80 and DNA-PKcs at DSBs induced by ionizing radiation is dependent on the complexity of damage

The dynamics of Ku70/80 and DNA-PKcs at DSBs induced by ionizing radiation is dependent on the complexity of damage

http://www.wikidata.org/entity/Q24605592

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Evaluating biomarkers to model cancer risk post cosmic ray exposure Microirradiation techniques in radiobiological research The role of RecQ helicases in non-homologous end-joining DNA-PK: a dynamic enzyme in a versatile DSB repair pathway Genotypic characteristics of resistant tumors to pre-operative ionizing radiation in rectal cancer Mechanisms and Consequences of Double-Strand DNA Break Formation in Chromatin Non-homologous end joining: emerging themes and unanswered questions.Mice and the A-Bomb: Irradiation Systems for Realistic Exposure Scenarios DNA Ligase IV regulates XRCC4 nuclear localization.Interaction between HIV-1 Tat and DNA-PKcs modulates HIV transcription and class switch recombination.DNA repair kinetics in SCID mice Sertoli cells and DNA-PKcs-deficient mouse embryonic fibroblasts.Spatiotemporal dynamics of DNA repair proteins following laser microbeam induced DNA damage - when is a DSB not a DSB?Scanning fluorescence correlation spectroscopy techniques to quantify the kinetics of DNA double strand break repair proteins after γ-irradiation and bleomycin treatment.Modeling damage complexity-dependent non-homologous end-joining repair pathway.Disruption of PARP1 function inhibits base excision repair of a sub-set of DNA lesions Organization and dynamics of the nonhomologous end-joining machinery during DNA double-strand break repair Interplay between Ku and Replication Protein A in the Restriction of Exo1-mediated DNA Break End Resection LRF maintains genome integrity by regulating the non-homologous end joining pathway of DNA repair Clustered DNA damage induces pan-nuclear H2AX phosphorylation mediated by ATM and DNA-PK DNA double strand break repair via non-homologous end-joining.A role for XLF in DNA repair and recombination in human somatic cells.Detection and repair of ionizing radiation-induced DNA double strand breaks: new developments in nonhomologous end joining.Repair of double-strand breaks by end joining.Iodine-125-labeled DNA-Triplex-forming oligonucleotides reveal increased cyto- and genotoxic effectiveness compared to Phosphorus-32.Functional genetic variants of XRCC4 and ERCC1 predict survival of gastric cancer patients treated with chemotherapy by regulating the gene expression.The influence of AKT isoforms on radiation sensitivity and DNA repair in colon cancer cell lines.The major DNA repair pathway after both proton and carbon-ion radiation is NHEJ, but the HR pathway is more relevant in carbon ions.Intense THz pulses cause H2AX phosphorylation and activate DNA damage response in human skin tissue.Modeling radiation-induced cell death: role of different levels of DNA damage clustering.Sulforaphane enhances irradiation effects in terms of perturbed cell cycle progression and increased DNA damage in pancreatic cancer cells.Calcium influx-mediated translocation of m-calpain induces Ku80 cleavage and enhances the Ku80-related DNA repair pathway.Studies of lncRNAs in DNA double strand break repair: what is new?Meta-analysis of DNA double-strand break response kinetics.How cells ensure correct repair of DNA double-strand breaks.Regulation of human polλ by ATM-mediated phosphorylation during non-homologous end joining.

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The dynamics of Ku70/80 and DNA-PKcs at DSBs induced by ionizing radiation is dependent on the complexity of damage

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2012 nî lūn-bûn

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2012 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

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2012年の論文

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The dynamics of Ku70/80 and DN ...... nt on the complexity of damage

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The dynamics of Ku70/80 and DN ...... nt on the complexity of damage

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The dynamics of Ku70/80 and DN ...... nt on the complexity of damage

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Jane V Harper

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Jennifer A Anderson

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Mark A Hill

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Pamela Reynolds

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Peter O'Neill

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Stanley W Botchway

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P2860

The mechanism of double-strand DNA break repair by the nonhomologous DNA end-joining pathway

XLF interacts with the XRCC4-DNA ligase IV complex to promote DNA nonhomologous end-joining

Ku recruits XLF to DNA double-strand breaks

Involvement of human polynucleotide kinase in double-strand break repair by non-homologous end joining

Processing of DNA for nonhomologous end-joining by cell-free extract

Dynamic assembly of end-joining complexes requires interaction between Ku70/80 and XRCC4

Evidence for a lack of DNA double-strand break repair in human cells exposed to very low x-ray doses

Biochemical evidence for Ku-independent backup pathways of NHEJ

Geometry of a complex formed by double strand break repair proteins at a single DNA end: recruitment of DNA-PKcs induces inward translocation of Ku protein.

A pathway of double-strand break rejoining dependent upon ATM, Artemis, and proteins locating to gamma-H2AX foci.

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4894629

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10.1093/NAR/GKS879

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21

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40

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Anthony W Parker

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2012-11-01T00:00:00Z

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231211969

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23012265

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3510491

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