IL-1F5, -F6, -F8, and -F9: a novel IL-1 family signaling system that is active in psoriasis and promotes keratinocyte antimicrobial peptide expression - Wikidata - awgldk - TriplyDB

IL-1F5, -F6, -F8, and -F9: a novel IL-1 family signaling system that is active in psoriasis and promotes keratinocyte antimicrobial peptide expression

IL-1F5, -F6, -F8, and -F9: a novel IL-1 family signaling system that is active in psoriasis and promotes keratinocyte antimicrobial peptide expression

http://www.wikidata.org/entity/Q24620194

about

Cytokines in psoriasis Systematic identification and characterization of novel human skin-associated genes encoding membrane and secreted proteins Psoriasiform dermatitis is driven by IL-36-mediated DC-keratinocyte crosstalk Genome-wide association study identifies eight new susceptibility loci for atopic dermatitis in the Japanese population Mutations in IL36RN/IL1F5 are associated with the severe episodic inflammatory skin disease known as generalized pustular psoriasis.IL-36 promotes myeloid cell infiltration, activation, and inflammatory activity in skin.IL-36 receptor deletion attenuates lung injury and decreases mortality in murine influenza pneumonia.IL-6 as a druggable target in psoriasis: focus on pustular variants.Decreased Langerhans cell responses to IL-36γ: altered innate immunity in patients with recurrent respiratory papillomatosis.IL-36α exerts pro-inflammatory effects in the lungs of mice Cathepsin S is the major activator of the psoriasis-associated proinflammatory cytokine IL-36γ.PSORS2 is due to mutations in CARD14.Decreased expression of interleukin-36α predicts poor prognosis in colorectal cancer patients Mechanisms of foot-and-mouth disease virus tropism inferred from differential tissue gene expression.Foxo1 inhibits diabetic mucosal wound healing but enhances healing of normoglycemic wounds.Proteomics of skin proteins in psoriasis: from discovery and verification in a mouse model to confirmation in humans.IL-17 induces an expanded range of downstream genes in reconstituted human epidermis model Transcriptional landscape of psoriasis identifies the involvement of IL36 and IL36RN.Immune Dysregulation in Patients Persistently Infected with Human Papillomaviruses 6 and 11.Histological Stratification of Thick and Thin Plaque Psoriasis Explores Molecular Phenotypes with Clinical Implications.IL-38 binds to the IL-36 receptor and has biological effects on immune cells similar to IL-36 receptor antagonist The double-stranded RNA analogue polyinosinic-polycytidylic acid induces keratinocyte pyroptosis and release of IL-36γ.Increased concentrations of soluble B7-H3 and interleukin 36 in bronchoalveolar lavage fluid of Children with Mycoplasma pneumoniae pneumonia.IL-36γ Transforms the Tumor Microenvironment and Promotes Type 1 Lymphocyte-Mediated Antitumor Immune Responses.Chronic skin-specific inflammation promotes vascular inflammation and thrombosis.Alteration of the EphA2/Ephrin-A signaling axis in psoriatic epidermis.Keratinocyte overexpression of IL-17C promotes psoriasiform skin inflammation.Promotion of autoimmune diabetes by cereal diet in the presence or absence of microbes associated with gut immune activation, regulatory imbalance, and altered cathelicidin antimicrobial Peptide.Opposing Functions of Classic and Novel IL-1 Family Members in Gut Health and Disease.IL-36γ Augments Host Defense and Immune Responses in Human Female Reproductive Tract Epithelial Cells.The IL-23/T17 pathogenic axis in psoriasis is amplified by keratinocyte responses.Prokineticin 2 Plays a Pivotal Role in Psoriasis.Hair follicle disruption facilitates pathogenesis to UVB-induced cutaneous inflammation and basal cell carcinoma development in Ptch(+/-) mice.New Insights in the Immunobiology of IL-1 Family Members.TLRs to cytokines: mechanistic insights from the imiquimod mouse model of psoriasis.Genome-wide association studies of atopic dermatitis.IL-1 and IL-36 are dominant cytokines in generalized pustular psoriasis.Increased Expression of Interleukin-36, a Member of the Interleukin-1 Cytokine Family, in Inflammatory Bowel Disease.Distinct expression of interleukin (IL)-36α, β and γ, their antagonist IL-36Ra and IL-38 in psoriasis, rheumatoid arthritis and Crohn's disease.Therapeutic targeting of Toll-like receptors: a review of Toll-like receptors and their signaling pathways in psoriasis.

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IL-1F5, -F6, -F8, and -F9: a novel IL-1 family signaling system that is active in psoriasis and promotes keratinocyte antimicrobial peptide expression

http://www.wikidata.org/entity/Q24620194

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2011 nî lūn-bûn

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2011 թուականի Փետրուարին հրատարակուած գիտական յօդուած

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2011 թվականի փետրվարին հրատարակված գիտական հոդված

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2011年の論文

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IL-1F5, -F6, -F8, and -F9: a n ...... timicrobial peptide expression

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IL-1F5, -F6, -F8, and -F9: a n ...... timicrobial peptide expression

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JIMMUNOL.1003162

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Journal of Immunology

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IL-1F5, -F6, -F8, and -F9: a n ...... timicrobial peptide expression

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Andrew Johnston

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Andrew M Guzman

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Candace M Loyd

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Frank Wang

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James T Elder

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Johann E Gudjonsson

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John J Voorhees

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Lisa E Maier

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MaryBeth Riblett

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Sewon Kang

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P2860

The new IL-1 family member IL-1F8 stimulates production of inflammatory mediators by synovial fibroblasts and articular chondrocytes

Four new members expand the interleukin-1 superfamily

Identification and initial characterization of four novel members of the interleukin-1 family

IL-33, an interleukin-1-like cytokine that signals via the IL-1 receptor-related protein ST2 and induces T helper type 2-associated cytokines

NALP3 forms an IL-1beta-processing inflammasome with increased activity in Muckle-Wells autoinflammatory disorder

Interaction of the small interstitial proteoglycans biglycan, decorin and fibromodulin with transforming growth factor beta

Opposing activities of two novel members of the IL-1 ligand family regulate skin inflammation

Summaries of Affymetrix GeneChip probe level data

IL1HY1: A novel interleukin-1 receptor antagonist gene

Transforming growth factor-beta and p-21: multiple molecular targets of decorin-mediated suppression of neoplastic growth

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4

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186

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