Molecular basis of cardiac performance. Plasticity of the myocardium generated through protein isoform switches
about
Myosin heavy chain gene expression in human heart failureRole of cardiac stem cells in cardiac pathophysiology: a paradigm shift in human myocardial biologyCardiac hypertrophy and impaired relaxation in transgenic mice overexpressing triadin 1A molecular mechanism improving the contractile state in human myocardial hypertrophyMaturing human pluripotent stem cell-derived cardiomyocytes in human engineered cardiac tissuesDistinct roles of mitogen-activated protein kinase pathways in GATA-4 transcription factor-mediated regulation of B-type natriuretic peptide geneDevelopmental expression and cardiac transcriptional regulation of Myh7b, a third myosin heavy chain in the vertebrate heartSexually dimorphic myofilament function and cardiac troponin I phosphospecies distribution in hypertrophic cardiomyopathy miceProgression from hypertrophic to dilated cardiomyopathy in mice that express a mutant myosin transgene.The genetics of hypertrophic cardiomyopathy.In vitro rescue study of a malignant familial hypertrophic cardiomyopathy phenotype by pseudo-phosphorylation of myosin regulatory light chain.Myc controls transcriptional regulation of cardiac metabolism and mitochondrial biogenesis in response to pathological stress in mice.Iron overload exacerbates age-associated cardiac hypertrophy in a mouse model of hemochromatosisHighly efficient gene transfer into adult ventricular myocytes by recombinant adenovirus.Myosin binding-induced cooperative activation of the thin filament in cardiac myocytes and skeletal muscle fibersTropomyosin modulates pH dependence of isometric tension.Decreased Gs alpha mRNA levels accompany the fall in Gs and adenylyl cyclase activities in compensated left ventricular hypertrophy. In heart failure, only the impairment in adenylyl cyclase activation progresses.The vascular smooth muscle alpha-actin gene is reactivated during cardiac hypertrophy provoked by load.Accumulation of fetal fibronectin mRNAs during the development of rat cardiac hypertrophy induced by pressure overload.Essential role of sympathetic endothelin A receptors for adverse cardiac remodelingSkeletal actin mRNA increases in the human heart during ontogenic development and is the major isoform of control and failing adult hearts.The role of MEKK1 in hypertrophic cardiomyopathy.Cardiac remodeling and physical training post myocardial infarctionSynergistic and antagonistic interplay between myostatin gene expression and physical activity levels on gene expression patterns in triceps Brachii muscles of C57/BL6 mice.Posttranscriptional modification of myosin heavy-chain gene expression in the hypertrophied rat myocardium.Evaluation of Cardioprotective Effect of 3,5,3'-Tri-iodo-L-thyronine in Isoproterenol-Induced CardiotoxicityHemodynamic regulation of myosin heavy chain gene expression. Studies in the transplanted rat heart.Fibronectin expression in the normal and hypertrophic rat heartHeat stress in rat adriamycin cardiomyopathy: heat shock protein 25 and Myosin accumulationSerum troponins as biomarkers of drug-induced cardiac toxicity.The Early-Onset Myocardial Infarction Associated PHACTR1 Gene Regulates Skeletal and Cardiac Alpha-Actin Gene Expression.Myocardial performance and its acute response to angiotensin II infusion in fetal sheep adapted to chronic anemia.Restoration of mechanical and energetic function in failing aortic-banded rat hearts by gene transfer of calcium cycling proteins.Developmental regulation of myosin gene expression in mouse cardiac muscleLost in transgenesis: a user's guide for genetically manipulating the mouse in cardiac research.Electrical and mechanical stimulation of cardiac cells and tissue constructs.Cardiac myosin isoforms exhibit differential rates of MgADP release and MgATP binding detected by myocardial viscoelasticity.Myofibrillar remodeling in cardiac hypertrophy, heart failure and cardiomyopathies.The CAAT-binding transcription factor 1/nuclear factor 1 binding site is important in beta-myosin heavy chain antisense promoter regulation in rats.Cardiovascular remodeling in response to long-term exposure to fine particulate matter air pollution
P2860
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P2860
Molecular basis of cardiac performance. Plasticity of the myocardium generated through protein isoform switches
description
1989 nî lūn-bûn
@nan
1989 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
1989 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
1989年の論文
@ja
1989年論文
@yue
1989年論文
@zh-hant
1989年論文
@zh-hk
1989年論文
@zh-mo
1989年論文
@zh-tw
1989年论文
@wuu
name
Molecular basis of cardiac per ...... rough protein isoform switches
@ast
Molecular basis of cardiac per ...... rough protein isoform switches
@en
Molecular basis of cardiac per ...... rough protein isoform switches
@nl
type
label
Molecular basis of cardiac per ...... rough protein isoform switches
@ast
Molecular basis of cardiac per ...... rough protein isoform switches
@en
Molecular basis of cardiac per ...... rough protein isoform switches
@nl
prefLabel
Molecular basis of cardiac per ...... rough protein isoform switches
@ast
Molecular basis of cardiac per ...... rough protein isoform switches
@en
Molecular basis of cardiac per ...... rough protein isoform switches
@nl
P2860
P356
P1476
Molecular basis of cardiac per ...... rough protein isoform switches
@en
P2093
B Nadal-Ginard
P2860
P304
P356
10.1172/JCI114351
P407
P577
1989-12-01T00:00:00Z