Transgenic hCFTR expression fails to correct β-ENaC mouse lung disease - Wikidata - awgldk - TriplyDB

Transgenic hCFTR expression fails to correct β-ENaC mouse lung disease

Transgenic hCFTR expression fails to correct β-ENaC mouse lung disease

http://www.wikidata.org/entity/Q24623934

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CFTR regulates early pathogenesis of chronic obstructive lung disease in βENaC-overexpressing mice CFTR and lung homeostasis.Status of fluid and electrolyte absorption in cystic fibrosis.Loss of Cftr function exacerbates the phenotype of Na(+) hyperabsorption in murine airways.Cigarette smoke and CFTR: implications in the pathogenesis of COPD Diffusion-sensitive optical coherence tomography for real-time monitoring of mucus thinning treatments.Role of CFTR in epithelial physiology.Does epithelial sodium channel hyperactivity contribute to cystic fibrosis lung disease?Regulation of ion transport by oxidants.The CFTR and ENaC debate: how important is ENaC in CF lung disease?Influenza A matrix protein M2 downregulates CFTR: inhibition of chloride transport by a proton channel of the viral envelope.

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P2860

Transgenic hCFTR expression fails to correct β-ENaC mouse lung disease

http://www.wikidata.org/entity/Q24623934

description

2012 nî lūn-bûn

@nan

2012 թուականի Յունուարին հրատարակուած գիտական յօդուած

@hyw

2012 թվականի հունվարին հրատարակված գիտական հոդված

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

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name

Transgenic hCFTR expression fails to correct β-ENaC mouse lung disease

@ast

Transgenic hCFTR expression fails to correct β-ENaC mouse lung disease

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Transgenic hCFTR expression fails to correct β-ENaC mouse lung disease

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type

label

Transgenic hCFTR expression fails to correct β-ENaC mouse lung disease

@ast

Transgenic hCFTR expression fails to correct β-ENaC mouse lung disease

@en

Transgenic hCFTR expression fails to correct β-ENaC mouse lung disease

@nl

prefLabel

Transgenic hCFTR expression fails to correct β-ENaC mouse lung disease

@ast

Transgenic hCFTR expression fails to correct β-ENaC mouse lung disease

@en

Transgenic hCFTR expression fails to correct β-ENaC mouse lung disease

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AJPLUNG.00083.2011

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American Journal of Physiology - Lung Cellular and Molecular Physiology

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Transgenic hCFTR expression fails to correct β-ENaC mouse lung disease

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P2093

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B R Grubb

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P2860

Evidence for periciliary liquid layer depletion, not abnormal ion composition, in the pathogenesis of cystic fibrosis airways disease

SLC26A9 is expressed in gastric surface epithelial cells, mediates Cl-/HCO3- exchange, and is inhibited by NH4+

SLC26A9 is a constitutively active, CFTR-regulated anion conductance in human bronchial epithelia

Airway surface liquid volume regulation determines different airway phenotypes in liddle compared with betaENaC-overexpressing mice

Identification of the cystic fibrosis gene: cloning and characterization of complementary DNA

Increased airway epithelial Na+ absorption produces cystic fibrosis-like lung disease in mice

Cell-specific expression of epithelial sodium channel alpha, beta, and gamma subunits in aldosterone-responsive epithelia from the rat: localization by in situ hybridization and immunocytochemistry

Expression and localization of epithelial aquaporins in the adult human lung.

Cell-specific expression of a Clara cell secretory protein-human growth hormone gene in the bronchiolar epithelium of transgenic mice.

Cystic fibrosis pigs develop lung disease and exhibit defective bacterial eradication at birth.

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