ACTH protects against glucocorticoid-induced osteonecrosis of bone - Wikidata - awgldk - TriplyDB

ACTH protects against glucocorticoid-induced osteonecrosis of bone

ACTH protects against glucocorticoid-induced osteonecrosis of bone

http://www.wikidata.org/entity/Q24631407

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Multiple sclerosis, relapses, and the mechanism of action of adrenocorticotropic hormone Genome-wide association study of N370S homozygous Gaucher disease reveals the candidacy of CLN8 gene as a genetic modifier contributing to extreme phenotypic variation The pathogenesis of nontraumatic osteonecrosis.Summary of the various treatments for osteonecrosis of the femoral head by mechanism: A review.MMP-3 and MMP-8 single-nucleotide polymorphisms are related to alcohol-induced osteonecrosis of the femoral head in Chinese males.eIF2α signaling regulates ischemic osteonecrosis through endoplasmic reticulum stress Calcium and bone disease.Glucocorticoid-induced osteonecrosis.Oxytocin and bone The atherogenic Scarb1 null mouse model shows a high bone mass phenotype Knee loading protects against osteonecrosis of the femoral head by enhancing vessel remodeling and bone healing.Role of proopiomelanocortin-derived peptides and their receptors in the osteoarticular system: from basic to translational research.Bone marrow oxytocin mediates the anabolic action of estrogen on the skeleton.Genetic association of the ApoB and ApoA1 gene polymorphisms with the risk for alcohol-induced osteonecrosis of femoral head Functional melanocortin-2 receptors are expressed by mouse aorta-derived mesenchymal progenitor cells.Functions of vasopressin and oxytocin in bone mass regulation.Treatment of acute relapses in multiple sclerosis.Mechanisms of action of adrenocorticotropic hormone and other melanocortins relevant to the clinical management of patients with multiple sclerosis.ACTH promotes chondrogenic nodule formation and induces transient elevations in intracellular calcium in rat bone marrow cell cultures via MC2-R signaling Effects of sodium ferulate on preventing steroid-induced femoral head osteonecrosis in rabbits Genetic confirmation for a central role for TNFα in the direct action of thyroid stimulating hormone on the skeleton Advanced diabetic nephropathy with nephrotic range proteinuria: a pilot study of the long-term efficacy of subcutaneous ACTH gel on proteinuria, progression of CKD, and urinary levels of VEGF and MCP-1.Proteomic analysis of gingival tissue and alveolar bone during alveolar bone healing Regulation of bone remodeling by vasopressin explains the bone loss in hyponatremia.Neuropeptide Y1 Receptor Regulates Glucocorticoid-Induced Inhibition of Osteoblast Differentiation in Murine MC3T3-E1 Cells via ERK Signaling.Leveraging melanocortin pathways to treat glomerular diseases.Administration of erythropoietin exerts protective effects against glucocorticoid-induced osteonecrosis of the femoral head in rats.Old drugs with new skills: fenoprofen as an allosteric enhancer at melanocortin receptor 3 A single-nucleotide polymorphism in MMP9 is associated with decreased risk of steroid-induced osteonecrosis of the femoral head.Management of osteonecrosis in children and young adults with acute lymphoblastic leukaemia.Skeletal receptors for steroid-family regulating glycoprotein hormones: A multilevel, integrated physiological control system.There are many potential medical therapies for atraumatic osteonecrosis.Bone, brain & beyond.The Role of Alpha-MSH as a Modulator of Ocular Immunobiology Exemplifies Mechanistic Differences between Melanocortins and Steroids.Efficacy and safety of adrenocorticotropic hormone treatment in glomerular diseases: a systematic review and meta-analysis.Osteoblast Differentiation and Bone Matrix Formation In Vivo and In Vitro.Is MC1 dispensable for regulation of cutaneous inflammatory and immune responses?Management of femoral head osteonecrosis: Current concepts.Adrenocorticotropic hormone and 1,25-dihydroxyvitamin D3 enhance human osteogenesis in vitro by synergistically accelerating the expression of bone-specific genes.Do Neuroendocrine Peptides and Their Receptors Qualify as Novel Therapeutic Targets in Osteoarthritis?

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ACTH protects against glucocorticoid-induced osteonecrosis of bone

http://www.wikidata.org/entity/Q24631407

description

2010 nî lūn-bûn

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2010 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2010 թվականի մայիսին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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ACTH protects against glucocorticoid-induced osteonecrosis of bone

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ACTH protects against glucocorticoid-induced osteonecrosis of bone

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ACTH protects against glucocorticoid-induced osteonecrosis of bone

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type

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ACTH protects against glucocorticoid-induced osteonecrosis of bone

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ACTH protects against glucocorticoid-induced osteonecrosis of bone

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ACTH protects against glucocorticoid-induced osteonecrosis of bone

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prefLabel

ACTH protects against glucocorticoid-induced osteonecrosis of bone

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ACTH protects against glucocorticoid-induced osteonecrosis of bone

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ACTH protects against glucocorticoid-induced osteonecrosis of bone

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Proceedings of the National Academy of Sciences of the United States of America

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ACTH protects against glucocorticoid-induced osteonecrosis of bone

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Alice Levine

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Beatrice B Yaroslavskiy

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Guozhe Yang

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Harry C Blair

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Irina L Tourkova

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Jameel Iqbal

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Jianhua Li

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Li Liu

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Li Sun

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Ling-Ling Zhu

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P2860

Proteolytic processing regulates receptor specificity and activity of VEGF-C.

Skeletal remodeling in health and disease

FSH directly regulates bone mass

Regional trabecular bone matrix degeneration and osteocyte death in femora of glucocorticoid- treated rabbits.

Does statin usage reduce the risk of corticosteroid-related osteonecrosis in renal transplant population?

Multiple melanocortin receptors are expressed in bone cells.

Relationship between systemic corticosteroids and osteonecrosis.

Treatment of pediatric epilepsy: European expert opinion, 2007.

The developmental basis of skeletal cell differentiation and the molecular basis of major skeletal defects.

Pathogenesis and natural history of osteonecrosis.

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