From nose to brain: development of gonadotrophin-releasing hormone-1 neurones - Wikidata - awgldk - TriplyDB

From nose to brain: development of gonadotrophin-releasing hormone-1 neurones

From nose to brain: development of gonadotrophin-releasing hormone-1 neurones

http://www.wikidata.org/entity/Q24633621

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Calcium release-dependent actin flow in the leading process mediates axophilic migration Sexual attractiveness of male chemicals and vocalizations in mice.On the value of seasonal mammals for identifying mechanisms underlying the control of food intake and body weight Neural crest and olfactory system: new prospective Transcriptional regulation of cranial sensory placode development Suppression of β1-integrin in gonadotropin-releasing hormone cells disrupts migration and axonal extension resulting in severe reproductive alterations Neuroendocrine control of the onset of puberty Mutual influences between the main olfactory and vomeronasal systems in development and evolution IGSF10 mutations dysregulate gonadotropin-releasing hormone neuronal migration resulting in delayed puberty.Neural crest and ectodermal cells intermix in the nasal placode to give rise to GnRH-1 neurons, sensory neurons, and olfactory ensheathing cells Dysregulation of Semaphorin7A/β1-integrin signaling leads to defective GnRH-1 cell migration, abnormal gonadal development and altered fertility Haploinsufficiency of Dmxl2, encoding a synaptic protein, causes infertility associated with a loss of GnRH neurons in mouse SEMA3A, a gene involved in axonal pathfinding, is mutated in patients with Kallmann syndrome GnRH, anosmia and hypogonadotropic hypogonadism--where are we?Changes in methylation patterns of kiss1 and kiss1r gene promoters across puberty Progressive effects of N-myc deficiency on proliferation, neurogenesis, and morphogenesis in the olfactory epithelium.Physiology of the gonadotrophin-releasing hormone (GnRH) neurone: studies from embryonic GnRH neurones Dysfunctional SEMA3E signaling underlies gonadotropin-releasing hormone neuron deficiency in Kallmann syndrome.Impaired sense of smell and altered olfactory system in RAG-1(-∕-) immunodeficient mice.Culturing embryonic nasal explants for developmental and physiological study Ectodermal Wnt controls nasal pit morphogenesis through modulation of the BMP/FGF/JNK signaling axis.Loss-of-function mutations in SOX10 cause Kallmann syndrome with deafness Epigenetic control of gonadotropin releasing hormone neurons.CCDC141 Mutation Identified in Anosmic Hypogonadotropic Hypogonadism (Kallmann Syndrome) Alters GnRH Neuronal Migration AXL and MET crosstalk to promote gonadotropin releasing hormone (GnRH) neuronal cell migration and survival.The Novel Actions of the Metabolite GnRH-(1-5) are Mediated by a G Protein-Coupled Receptor.Ontogenesis of gonadotropin-releasing hormone neurons: a model for hypothalamic neuroendocrine cell development.Evidence for a Notch1-mediated transition during olfactory ensheathing cell development.Semaphorin signaling in the development and function of the gonadotropin hormone-releasing hormone system.The pre-vertebrate origins of neurogenic placodes.CXC chemokine receptor 7 (CXCR7) affects the migration of GnRH neurons by regulating CXCL12 availability.The indirect role of fibroblast growth factor-8 in defining neurogenic niches of the olfactory/GnRH systems.P75 nerve growth factor receptors modulate development of GnRH neurons and olfactory ensheating cells.Differential expression of nasal embryonic LHRH factor (NELF) variants in immortalized GnRH neuronal cell lines.Olfactory system and demyelination.The spatiotemporal segregation of GAD forms defines distinct GABA signaling functions in the developing mouse olfactory system and provides novel insights into the origin and migration of GnRH neurons.Commissural axonal corridors instruct neuronal migration in the mouse spinal cord.The Vomeronasal Organ: A Neglected Organ Olfactory ensheathing glia are required for embryonic olfactory axon targeting and the migration of gonadotropin-releasing hormone neurons.Transient appearance of the epithelial invagination in the olfactory pit of chick embryos.

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From nose to brain: development of gonadotrophin-releasing hormone-1 neurones

http://www.wikidata.org/entity/Q24633621

description

2010 nî lūn-bûn

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2010 թուականի Յուլիսին հրատարակուած գիտական յօդուած

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2010 թվականի հուլիսին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

From nose to brain: development of gonadotrophin-releasing hormone-1 neurones

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From nose to brain: development of gonadotrophin-releasing hormone-1 neurones

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From nose to brain: development of gonadotrophin-releasing hormone-1 neurones

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From nose to brain: development of gonadotrophin-releasing hormone-1 neurones

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From nose to brain: development of gonadotrophin-releasing hormone-1 neurones

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From nose to brain: development of gonadotrophin-releasing hormone-1 neurones

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From nose to brain: development of gonadotrophin-releasing hormone-1 neurones

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From nose to brain: development of gonadotrophin-releasing hormone-1 neurones

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From nose to brain: development of gonadotrophin-releasing hormone-1 neurones

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J.1365-2826.2010.02034.X

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Journal of Neuroendocrinology

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From nose to brain: development of gonadotrophin-releasing hormone-1 neurones

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P2860

Kallmann syndrome: mutations in the genes encoding prokineticin-2 and prokineticin receptor-2

NELF is a nuclear protein involved in hypothalamic GnRH neuronal migration

Cholecystokinin modulates migration of gonadotropin-releasing hormone-1 neurons

Novel gene expressed in nasal region influences outgrowth of olfactory axons and migration of luteinizing hormone-releasing hormone (LHRH) neurons

Signaling and epigenetic regulation of pituitary development

Digenic mutations account for variable phenotypes in idiopathic hypogonadotropic hypogonadism

The candidate gene for the X-linked Kallmann syndrome encodes a protein related to adhesion molecules

Mouse Gli1 mutants are viable but have defects in SHH signaling in combination with a Gli2 mutation

Expression of three mouse homologs of the Drosophila segment polarity gene cubitus interruptus, Gli, Gli-2, and Gli-3, in ectoderm- and mesoderm-derived tissues suggests multiple roles during postimplantation development

Characterization of the human nasal embryonic LHRH factor gene, NELF, and a mutation screening among 65 patients with idiopathic hypogonadotropic hypogonadism (IHH)

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