Neonatal bisphenol-a exposure alters rat reproductive development and ovarian morphology without impairing activation of gonadotropin-releasing hormone neurons - Wikidata - awgldk - TriplyDB

Neonatal bisphenol-a exposure alters rat reproductive development and ovarian morphology without impairing activation of gonadotropin-releasing hormone neurons

Neonatal bisphenol-a exposure alters rat reproductive development and ovarian morphology without impairing activation of gonadotropin-releasing hormone neurons

http://www.wikidata.org/entity/Q24643460

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Long-term effects of environmental endocrine disruptors on reproductive physiology and behavior Polycystic ovary syndrome: do endocrine-disrupting chemicals play a role?Non-monotonic dose-response relationships and endocrine disruptors: a qualitative method of assessment EDC-2: The Endocrine Society's Second Scientific Statement on Endocrine-Disrupting Chemicals In utero and peripubertal exposure to phthalates and BPA in relation to female sexual maturation Epigenetic effects of endocrine-disrupting chemicals on female reproduction: an ovarian perspective Bisphenol a: a model endocrine disrupting chemical with a new potential mechanism of action The pros and cons of phytoestrogens Evidence for bisphenol A-induced female infertility: a review (2007-2016)Soy but not bisphenol A (BPA) induces hallmarks of polycystic ovary syndrome (PCOS) and related metabolic co-morbidities in rats Effects of Korean red ginseng (Panax Ginseng Meyer) on bisphenol A exposure and gynecologic complaints: single blind, randomized clinical trial of efficacy and safety Bisphenol a and reproductive health: update of experimental and human evidence, 2007-2013 The role of Bisphenol A in shaping the brain, epigenome and behavior.Elucidating the links between endocrine disruptors and neurodevelopment.Neonatal exposure to bisphenol a and reproductive and endocrine alterations resembling the polycystic ovarian syndrome in adult rats.Developmental programming: impact of fetal exposure to endocrine-disrupting chemicals on gonadotropin-releasing hormone and estrogen receptor mRNA in sheep hypothalamus.The effects of postnatal exposure to low-dose bisphenol-A on activity-dependent plasticity in the mouse sensory cortex.The impact of neonatal bisphenol-A exposure on sexually dimorphic hypothalamic nuclei in the female rat.Endocrine disruption of brain sexual differentiation by developmental PCB exposure Endocrine disrupting chemicals promote the growth of ovarian cancer cells via the ER-CXCL12-CXCR4 signaling axis.Long-term effects of early-life exposure to environmental oestrogens on ovarian function: role of epigenetics Potential protective effect of Tualang honey on BPA-induced ovarian toxicity in prepubertal rat.Systemic compensatory response to neonatal estradiol exposure does not prevent depletion of the oocyte pool in the rat.Neonatal agonism of ERβ impairs male reproductive behavior and attractiveness.Bisphenol A increases mammary cancer risk in two distinct mouse models of breast cancer Preimplantation exposure to bisphenol A (BPA) affects embryo transport, preimplantation embryo development, and uterine receptivity in mice.Early life exposure to endocrine-disrupting chemicals causes lifelong molecular reprogramming of the hypothalamus and premature reproductive aging.Endocrine disrupters: a review of some sources, effects, and mechanisms of actions on behaviour and neuroendocrine systems.Neonatal Bisphenol A exposure alters sexually dimorphic gene expression in the postnatal rat hypothalamus.Influence of ERβ selective agonism during the neonatal period on the sexual differentiation of the rat hypothalamic-pituitary-gonadal (HPG) axis.Impact of environmental exposures on ovarian function and role of xenobiotic metabolism during ovotoxicity Bisphenol A and hormone-associated cancers: current progress and perspectives Disrupted organization of RFamide pathways in the hypothalamus is associated with advanced puberty in female rats neonatally exposed to bisphenol A.Prenatal exposure to low doses of bisphenol A increases pituitary proliferation and gonadotroph number in female mice offspring at birth.Maternal exposure to bisphenol A and genistein has minimal effect on A(vy)/a offspring coat color but favors birth of agouti over nonagouti mice.Developmental programming: impact of prenatal exposure to bisphenol-A and methoxychlor on steroid feedbacks in sheep.Bisphenol A exposure during adulthood causes augmentation of follicular atresia and luteal regression by decreasing 17β-estradiol synthesis via downregulation of aromatase in rat ovary.BPA Directly Decreases GnRH Neuronal Activity via Noncanonical Pathway Disruption of reproductive aging in female and male rats by gestational exposure to estrogenic endocrine disruptors Stereological study on the effect of vitamin C in preventing the adverse effects of bisphenol A on rat ovary.

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P2860

Neonatal bisphenol-a exposure alters rat reproductive development and ovarian morphology without impairing activation of gonadotropin-releasing hormone neurons

http://www.wikidata.org/entity/Q24643460

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2009 nî lūn-bûn

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2009 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

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2009 թվականի հոտեմբերին հրատարակված գիտական հոդված

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Neonatal bisphenol-a exposure ...... opin-releasing hormone neurons

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Neonatal bisphenol-a exposure ...... opin-releasing hormone neurons

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Neonatal bisphenol-a exposure ...... opin-releasing hormone neurons

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Neonatal bisphenol-a exposure ...... opin-releasing hormone neurons

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Neonatal bisphenol-a exposure ...... opin-releasing hormone neurons

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Neonatal bisphenol-a exposure ...... opin-releasing hormone neurons

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Neonatal bisphenol-a exposure ...... opin-releasing hormone neurons

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Biology of Reproduction

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Neonatal bisphenol-a exposure ...... opin-releasing hormone neurons

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Heather B Adewale

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Heather B Patisaul

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Retha R Newbold

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Wendy N Jefferson

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P2860

Urinary concentrations of bisphenol A and 4-nonylphenol in a human reference population

Kisspeptin directly stimulates gonadotropin-releasing hormone release via G protein-coupled receptor 54.

Chapel Hill bisphenol A expert panel consensus statement: integration of mechanisms, effects in animals and potential to impact human health at current levels of exposure

No effect of route of exposure (oral; subcutaneous injection) on plasma bisphenol A throughout 24h after administration in neonatal female mice

Role for estradiol in female-typical brain and behavioral sexual differentiation

Bisphenol-A and the great divide: a review of controversies in the field of endocrine disruption

Direct evidence revealing structural elements essential for the high binding ability of bisphenol A to human estrogen-related receptor-gamma

Exposure of the U.S. population to bisphenol A and 4-tertiary-octylphenol: 2003-2004

Maternal nutrient supplementation counteracts bisphenol A-induced DNA hypomethylation in early development

Parent bisphenol A accumulation in the human maternal-fetal-placental unit

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