The VEGF IRESes are differentially susceptible to translation inhibition by miR-16 - Wikidata - awgldk - TriplyDB

The VEGF IRESes are differentially susceptible to translation inhibition by miR-16

The VEGF IRESes are differentially susceptible to translation inhibition by miR-16

http://www.wikidata.org/entity/Q24647478

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mRNA and miRNA regulatory networks reflective of multi-walled carbon nanotube-induced lung inflammatory and fibrotic pathologies in mice miRNA and vascular cell movement The miR-15/107 group of microRNA genes: evolutionary biology, cellular functions, and roles in human diseases Kinetic signatures of microRNA modes of action Role of hypoxia and vascular endothelial growth factors in lymphangiogenesis A microRNA code for prostate cancer metastasis.Bridging IRES elements in mRNAs to the eukaryotic translation apparatus hnRNP A1 mediates the activation of the IRES-dependent SREBP-1a mRNA translation in response to endoplasmic reticulum stress Dynamical modeling of microRNA action on the protein translation process.Cellular microRNAs and picornaviral infections The expression levels of microRNA-361-5p and its target VEGFA are inversely correlated in human cutaneous squamous cell carcinoma Cellular IRES-mediated translation: the war of ITAFs in pathophysiological states.Alternative RNA structure-coupled gene regulations in tumorigenesis.Effects of dietary L-arginine or N-carbamylglutamate supplementation during late gestation of sows on the miR-15b/16, miR-221/222, VEGFA and eNOS expression in umbilical vein.Vascular endothelial cell-specific microRNA-15a inhibits angiogenesis in hindlimb ischemia.Stabilization of the G-quadruplex at the VEGF IRES represses cap-independent translation.Key contribution of eIF4H-mediated translational control in tumor promotion.Cellular microRNAs Repress Vesicular Stomatitis Virus but Not Theiler's Virus Replication.VEGF-A mRNA processing, stability and translation: a paradigm for intricate regulation of gene expression at the post-transcriptional level.Diet, microRNAs and prostate cancer.MicroRNAs mediate metabolic stresses and angiogenesis.E2F and microRNA regulation of angiogenesis.MicroRNAs and tumor vasculature normalization: impact on anti-tumor immune response.Aberrant regulation of miR-15b in human malignant tumors and its effects on the hallmarks of cancer.Role of hypoxia and vascular endothelial growth factors in lymphangiogenesis.Mammalian Polycistronic mRNAs and Disease.Bone marrow stromal cells protect myeloma cells from bortezomib induced apoptosis by suppressing microRNA-15a expression.microRNAs related to angiogenesis are dysregulated in endometrioid endometrial cancer.Repression of VEGFA by CA-rich element-binding microRNAs is modulated by hnRNP L.Hypoxia alters MicroRNA expression in rat cortical pericytes.IRES-Dependent Translational Control during Virus-Induced Endoplasmic Reticulum Stress and Apoptosis The microRNA-15a-PAI-2 axis in cholangiocarcinoma-associated fibroblasts promotes migration of cancer cells.Defining the role of the tumor vasculature in antitumor immunity and immunotherapy.PERK mediates the IRES-dependent translational activation of mRNAs encoding angiogenic growth factors after ischemic stress.Hypoxia-microRNA-16 downregulation induces VEGF expression in anaplastic lymphoma kinase (ALK)-positive anaplastic large-cell lymphomas.Old and Young Actors Playing Novel Roles in the Drama of Multiple Myeloma Bone Marrow Microenvironment Dependent Drug Resistance.

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P2860

The VEGF IRESes are differentially susceptible to translation inhibition by miR-16

http://www.wikidata.org/entity/Q24647478

description

2009 nî lūn-bûn

@nan

2009 թուականի Փետրուարին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի փետրվարին հրատարակված գիտական հոդված

@hy

2009年の論文

@ja

2009年論文

@yue

2009年論文

@zh-hant

2009年論文

@zh-hk

2009年論文

@zh-mo

2009年論文

@zh-tw

2009年论文

@wuu

name

The VEGF IRESes are differentially susceptible to translation inhibition by miR-16

@ast

The VEGF IRESes are differentially susceptible to translation inhibition by miR-16

@en

The VEGF IRESes are differentially susceptible to translation inhibition by miR-16

@nl

type

label

The VEGF IRESes are differentially susceptible to translation inhibition by miR-16

@ast

The VEGF IRESes are differentially susceptible to translation inhibition by miR-16

@en

The VEGF IRESes are differentially susceptible to translation inhibition by miR-16

@nl

prefLabel

The VEGF IRESes are differentially susceptible to translation inhibition by miR-16

@ast

The VEGF IRESes are differentially susceptible to translation inhibition by miR-16

@en

The VEGF IRESes are differentially susceptible to translation inhibition by miR-16

@nl

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P1476

The VEGF IRESes are differentially susceptible to translation inhibition by miR-16

@en

P2093

Hervé Prats

http://www.w3.org/2001/XMLSchema#string

Jason S Iacovoni

http://www.w3.org/2001/XMLSchema#string

Zeïneb S Karaa

http://www.w3.org/2001/XMLSchema#string

P2860

An mRNA m7G cap binding-like motif within human Ago2 represses translation

Two independent internal ribosome entry sites are involved in translation initiation of vascular endothelial growth factor mRNA

MicroRNAs control translation initiation by inhibiting eukaryotic initiation factor 4E/cap and poly(A) tail function

Mechanisms of post-transcriptional regulation by microRNAs: are the answers in sight?

Inhibition of translational initiation by Let-7 MicroRNA in human cells

Heterozygous embryonic lethality induced by targeted inactivation of the VEGF gene

Abnormal blood vessel development and lethality in embryos lacking a single VEGF allele

Target mRNAs are repressed as efficiently by microRNA-binding sites in the 5' UTR as in the 3' UTR

Translation of vascular endothelial growth factor mRNA by internal ribosome entry: implications for translation under hypoxia.

Short RNAs repress translation after initiation in mammalian cells.

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249-254

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scholarly article

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10.1261/RNA.1301109

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2

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15

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Christian Touriol

Amandine Bastide

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2009-02-01T00:00:00Z

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23789001

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19144909

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2648711

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