Neuroprotective effects of inositol 1,4,5-trisphosphate receptor C-terminal fragment in a Huntington's disease mouse model
about
Integration of modeling with experimental and clinical findings synthesizes and refines the central role of inositol 1,4,5-trisphosphate receptor 1 in spinocerebellar ataxiaMouse models of polyglutamine diseases in therapeutic approaches: review and data table. Part II.Genetics and neuropathology of Huntington's diseaseAAV-dominant negative tumor necrosis factor (DN-TNF) gene transfer to the striatum does not rescue medium spiny neurons in the YAC128 mouse model of Huntington's diseaseProtein transamidation by transglutaminase 2 in cells: a disputed Ca2+-dependent action of a multifunctional proteinThe role of ryanodine receptor type 3 in a mouse model of Alzheimer diseaseTetrabenazine is neuroprotective in Huntington's disease mice.Inositol trisphosphate receptor Ca2+ release channels in neurological diseases.Role of presenilins in neuronal calcium homeostasisInhibition of lipid signaling enzyme diacylglycerol kinase epsilon attenuates mutant huntingtin toxicity.Computational analysis of calcium signaling and membrane electrophysiology in cerebellar Purkinje neurons associated with ataxia.Altered Ca(2+) signaling in skeletal muscle fibers of the R6/2 mouse, a model of Huntington's diseaseAβ42-binding peptoids as amyloid aggregation inhibitors and detection ligands.Role of inositol 1,4,5-trisphosphate receptors in pathogenesis of Huntington's disease and spinocerebellar ataxiasComputational neurobiology is a useful tool in translational neurology: the example of ataxiaAn automated and quantitative method to evaluate progression of striatal pathology in Huntington's disease transgenic mice.Neuronal store-operated calcium entry pathway as a novel therapeutic target for Huntington's disease treatment.Huntington's Disease: From Mutant Huntingtin Protein to Neurotrophic Factor Therapy.Expanded polyglutamine-binding peptoid as a novel therapeutic agent for treatment of Huntington's disease.Huntington's Disease and Striatal Signaling.The role for alterations in neuronal activity in the pathogenesis of polyglutamine repeat disorders.Dantrolene is neuroprotective in Huntington's disease transgenic mouse model.The sigma-1 receptor mediates the beneficial effects of pridopidine in a mouse model of Huntington diseaseEnhanced Store-Operated Calcium Entry Leads to Striatal Synaptic Loss in a Huntington's Disease Mouse Model.The type III inositol 1,4,5-trisphosphate receptor is associated with aggressiveness of colorectal carcinomaInositol 1,4,5-tripshosphate receptor, calcium signaling, and polyglutamine expansion disorders.Deranged calcium signaling and neurodegeneration in spinocerebellar ataxia type 2.Control of intracellular calcium signaling as a neuroprotective strategyScreening of therapeutic strategies for Huntington's disease in YAC128 transgenic mice.Inositol 1,4,5-trisphosphate receptor-mediated calcium release in Purkinje cells: from molecular mechanism to behavior.Pharmacological protein targets in polyglutamine diseases: mutant polypeptides and their interactors.Modulating Ca2+ release by the IP3R/Ca2+ channel as a potential therapeutic treatment for neurological diseases.Intracellular calcium channels: inositol-1,4,5-trisphosphate receptors.Corticostriatal circuit dysfunction in Huntington's disease: intersection of glutamate, dopamine and calcium.The entangled ER-mitochondrial axis as a potential therapeutic strategy in neurodegeneration: A tangled duo unchained.Determining the Roles of Inositol Trisphosphate Receptors in Neurodegeneration: Interdisciplinary Perspectives on a Complex Topic.IP3 receptor mutations and brain diseases in human and rodents.Calcium signaling and molecular mechanisms underlying neurodegenerative diseases.High-content RNAi screening identifies the Type 1 inositol triphosphate receptor as a modifier of TDP-43 localization and neurotoxicity.Huntington's Disease: Calcium Dyshomeostasis and Pathology Models
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P2860
Neuroprotective effects of inositol 1,4,5-trisphosphate receptor C-terminal fragment in a Huntington's disease mouse model
description
2009 nî lūn-bûn
@nan
2009 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2009年の論文
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2009年学术文章
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2009年学术文章
@zh-cn
2009年学术文章
@zh-hans
2009年学术文章
@zh-my
2009年学术文章
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2009年學術文章
@yue
name
Neuroprotective effects of ino ...... ntington's disease mouse model
@ast
Neuroprotective effects of ino ...... ntington's disease mouse model
@en
Neuroprotective effects of ino ...... ntington's disease mouse model
@nl
type
label
Neuroprotective effects of ino ...... ntington's disease mouse model
@ast
Neuroprotective effects of ino ...... ntington's disease mouse model
@en
Neuroprotective effects of ino ...... ntington's disease mouse model
@nl
prefLabel
Neuroprotective effects of ino ...... ntington's disease mouse model
@ast
Neuroprotective effects of ino ...... ntington's disease mouse model
@en
Neuroprotective effects of ino ...... ntington's disease mouse model
@nl
P2093
P2860
P1476
Neuroprotective effects of ino ...... ntington's disease mouse model
@en
P2093
Caixia Guo
Hongyu Wang
Ilya Bezprozvanny
Tie-Shan Tang
P2860
P304
P356
10.1523/JNEUROSCI.4411-08.2009
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P577
2009-02-04T00:00:00Z