Human oligodendrocytes express Cx31.3: function and interactions with Cx32 mutants - Wikidata - awgldk - TriplyDB

Human oligodendrocytes express Cx31.3: function and interactions with Cx32 mutants

Human oligodendrocytes express Cx31.3: function and interactions with Cx32 mutants

http://www.wikidata.org/entity/Q24655300

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Gap junctions in inherited human disorders of the central nervous system.Simvastatin Sodium Salt and Fluvastatin Interact with Human Gap Junction Gamma-3 Protein Connexin32 mutations cause loss of function in Schwann cells and oligodendrocytes leading to PNS and CNS myelination defects The role of gap junction channels during physiologic and pathologic conditions of the human central nervous system How do mutations in GJB1 cause X-linked Charcot-Marie-Tooth disease?Molecular mechanisms of inherited demyelinating neuropathies.X-linked Charcot-Marie-Tooth disease.GJB1-associated X-linked Charcot-Marie-Tooth disease, a disorder affecting the central and peripheral nervous systems.The oligodendroglial precursor cell line Oli-neu represents a cell culture system to examine functional expression of the mouse gap junction gene connexin29 (Cx29).Gene therapy targeting oligodendrocytes provides therapeutic benefit in a leukodystrophy model.Gap junction pathology in multiple sclerosis lesions and normal-appearing white matter.Human connexin30.2/31.3 (GJC3) does not form functional gap junction channels but causes enhanced ATP release in HeLa cells.Recurrent central nervous system white matter changes in charcot-Marie-tooth type X disease.What's the Function of Connexin 32 in the Peripheral Nervous System?

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P2860

Human oligodendrocytes express Cx31.3: function and interactions with Cx32 mutants

http://www.wikidata.org/entity/Q24655300

description

2008 nî lūn-bûn

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2008 թուականի Մայիսին հրատարակուած գիտական յօդուած

@hyw

2008 թվականի մայիսին հրատարակված գիտական հոդված

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年论文

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name

Human oligodendrocytes express Cx31.3: function and interactions with Cx32 mutants

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Human oligodendrocytes express Cx31.3: function and interactions with Cx32 mutants

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Human oligodendrocytes express Cx31.3: function and interactions with Cx32 mutants

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type

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Human oligodendrocytes express Cx31.3: function and interactions with Cx32 mutants

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Human oligodendrocytes express Cx31.3: function and interactions with Cx32 mutants

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Human oligodendrocytes express Cx31.3: function and interactions with Cx32 mutants

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Human oligodendrocytes express Cx31.3: function and interactions with Cx32 mutants

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Human oligodendrocytes express Cx31.3: function and interactions with Cx32 mutants

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Human oligodendrocytes express Cx31.3: function and interactions with Cx32 mutants

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P356

J.NBD.2008.01.009

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Neurobiology of Disease

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Human oligodendrocytes express Cx31.3: function and interactions with Cx32 mutants

@en

P2093

Alan D Enriquez

http://www.w3.org/2001/XMLSchema#string

Alejandro Peinado

http://www.w3.org/2001/XMLSchema#string

Charles Abrams

http://www.w3.org/2001/XMLSchema#string

Irene Sargiannidou

http://www.w3.org/2001/XMLSchema#string

Kleopas A Kleopa

http://www.w3.org/2001/XMLSchema#string

Meejin Ahn

http://www.w3.org/2001/XMLSchema#string

Richard Reynolds

http://www.w3.org/2001/XMLSchema#string

Steven S Scherer

http://www.w3.org/2001/XMLSchema#string

P2860

Connexin mutations in X-linked Charcot-Marie-Tooth disease

Cell-specific expression of connexins and evidence of restricted gap junctional coupling between glial cells and between neurons

Connexin-47 and connexin-32 in gap junctions of oligodendrocyte somata, myelin sheaths, paranodal loops and Schmidt-Lanterman incisures: implications for ionic homeostasis and potassium siphoning.

Loss-of-function GJA12/Connexin47 mutations cause Pelizaeus-Merzbacher-like disease

The mouse gap junction gene connexin29 is highly expressed in sciatic nerve and regulated during brain development

Gating and regulation of connexin 43 (Cx43) hemichannels.

Coupling of astrocyte connexins Cx26, Cx30, Cx43 to oligodendrocyte Cx29, Cx32, Cx47: Implications from normal and connexin32 knockout mice.

Connexin47, connexin29 and connexin32 co-expression in oligodendrocytes and Cx47 association with zonula occludens-1 (ZO-1) in mouse brain.

Defective N-acetylaspartate catabolism reduces brain acetate levels and myelin lipid synthesis in Canavan's disease.

Structural and functional diversity of connexin genes in the mouse and human genome.

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221-33

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scholarly article

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10.1016/J.NBD.2008.01.009

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2

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oligodendrocyte

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