Human APOBEC3G can restrict retroviral infection in avian cells and acts independently of both UNG and SMUG1 - Wikidata - awgldk - TriplyDB

Human APOBEC3G can restrict retroviral infection in avian cells and acts independently of both UNG and SMUG1

Human APOBEC3G can restrict retroviral infection in avian cells and acts independently of both UNG and SMUG1

http://www.wikidata.org/entity/Q24655541

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Differential anti-APOBEC3G activity of HIV-1 Vif proteins derived from different subtypes HIV-1 Vif versus the APOBEC3 cytidine deaminases: an intracellular duel between pathogen and host restriction factors HIV-1 Vif, APOBEC, and intrinsic immunity Multiple APOBEC3 restriction factors for HIV-1 and one Vif to rule them all Multiple ways of targeting APOBEC3-virion infectivity factor interactions for anti-HIV-1 drug development.AID and Apobec3G haphazard deamination and mutational diversity APOBEC3G inhibits elongation of HIV-1 reverse transcripts.Are viruses inhibited by APOBEC3 molecules from their host species?Recruitment of the nuclear form of uracil DNA glycosylase into virus particles participates in the full infectivity of HIV-1.HIV-1 Vpr loads uracil DNA glycosylase-2 onto DCAF1, a substrate recognition subunit of a cullin 4A-ring E3 ubiquitin ligase for proteasome-dependent degradation.APOBEC3G-induced hypermutation of human immunodeficiency virus type-1 is typically a discrete "all or nothing" phenomenon.Local sequence targeting in the AID/APOBEC family differentially impacts retroviral restriction and antibody diversification.Uracil DNA glycosylase counteracts APOBEC3G-induced hypermutation of hepatitis B viral genomes: excision repair of covalently closed circular DNA.Target cell-mediated editing of HIV-1 cDNA by APOBEC3 proteins in human macrophages.HIV Restriction Factors and Mechanisms of Evasion.Uracil DNA glycosylase interacts with the p32 subunit of the replication protein A complex to modulate HIV-1 reverse transcription for optimal virus dissemination.APOBEC3 versus Retroviruses, Immunity versus Invasion: Clash of the Titans.Antiviral Mechanism and Biochemical Basis of the Human APOBEC3 Family.A novel role for APOBEC3: susceptibility to sexual transmission of murine acquired immunodeficiency virus (mAIDS) is aggravated in APOBEC3 deficient mice.Running loose or getting lost: how HIV-1 counters and capitalizes on APOBEC3-induced mutagenesis through its Vif protein Suppression of HIV-1 infection by APOBEC3 proteins in primary human CD4(+) T cells is associated with inhibition of processive reverse transcription as well as excessive cytidine deamination.Uracil DNA glycosylase initiates degradation of HIV-1 cDNA containing misincorporated dUTP and prevents viral integration.DNA Editing of LTR Retrotransposons Reveals the Impact of APOBECs on Vertebrate Genomes.APOBEC proteins and intrinsic resistance to HIV-1 infection The AID/APOBEC family of nucleic acid mutators.Molecular mechanism for generation of antibody memory.Tumultuous relationship between the human immunodeficiency virus type 1 viral infectivity factor (Vif) and the human APOBEC-3G and APOBEC-3F restriction factors.APOBEC3G-Mediated G-to-A Hypermutation of the HIV-1 Genome: The Missing Link in Antiviral Molecular Mechanisms HIV-1 Vpr-a still "enigmatic multitasker".Type I Interferon at the Interface of Antiviral Immunity and Immune Regulation: The Curious Case of HIV-1.Concerted action of activation-induced cytidine deaminase and uracil-DNA glycosylase reduces covalently closed circular DNA of duck hepatitis B virus.Evolution of HIV-1 isolates that use a novel Vif-independent mechanism to resist restriction by human APOBEC3G.The role of cytidine deaminases on innate immune responses against human viral infections.Deep sequencing of HIV-1 reverse transcripts reveals the multifaceted antiviral functions of APOBEC3G.

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P2860

Human APOBEC3G can restrict retroviral infection in avian cells and acts independently of both UNG and SMUG1

http://www.wikidata.org/entity/Q24655541

description

2008 nî lūn-bûn

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2008 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年论文

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Human APOBEC3G can restrict re ...... endently of both UNG and SMUG1

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Human APOBEC3G can restrict re ...... endently of both UNG and SMUG1

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Human APOBEC3G can restrict re ...... endently of both UNG and SMUG1

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P1433

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P1433

Journal of Virology

P1476

Human APOBEC3G can restrict re ...... endently of both UNG and SMUG1

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P2093

Marc-André Langlois

http://www.w3.org/2001/XMLSchema#string

Michael S Neuberger

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P2860

hUNG2 is the major repair enzyme for removal of uracil from U:A matches, U:G mismatches, and U in single-stranded DNA, with hSMUG1 as a broad specificity backup

The cytidine deaminase CEM15 induces hypermutation in newly synthesized HIV-1 DNA

APOBEC3F can inhibit the accumulation of HIV-1 reverse transcription products in the absence of hypermutation. Comparisons with APOBEC3G

Mutational comparison of the single-domained APOBEC3C and double-domained APOBEC3F/G anti-retroviral cytidine deaminases provides insight into their DNA target site specificities

Human immunodeficiency virus type 1 Vpr induces the degradation of the UNG and SMUG uracil-DNA glycosylases

SMUG1 is able to excise uracil from immunoglobulin genes: insight into mutation versus repair

Inhibition of tRNA₃(Lys)-primed reverse transcription by human APOBEC3G during human immunodeficiency virus type 1 replication

7SL RNA mediates virion packaging of the antiviral cytidine deaminase APOBEC3G

Human immunodeficiency virus type 1 cDNAs produced in the presence of APOBEC3G exhibit defects in plus-strand DNA transfer and integration

APOBEC3G & HTLV-1: inhibition without deamination

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4660-4

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scholarly article

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2371828

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10.1128/JVI.02469-07

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9

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82

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18272574

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2293047

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