Absence of CD36 protects against atherosclerosis in ApoE knock-out mice with no additional protection provided by absence of scavenger receptor A I/II - Wikidata - awgldk - TriplyDB

Absence of CD36 protects against atherosclerosis in ApoE knock-out mice with no additional protection provided by absence of scavenger receptor A I/II

Absence of CD36 protects against atherosclerosis in ApoE knock-out mice with no additional protection provided by absence of scavenger receptor A I/II

http://www.wikidata.org/entity/Q24658096

about

Monocytes in atherosclerosis: subsets and functions Scavenger Receptors: Emerging Roles in Cancer Biology and Immunology The Role of Different Monocyte Subsets in the Pathogenesis of Atherosclerosis and Acute Coronary Syndromes The innate immune response to products of phospholipid peroxidation CD36 coordinates NLRP3 inflammasome activation by facilitating intracellular nucleation of soluble ligands into particulate ligands in sterile inflammation Regulation of Transcription Factors by Reactive Oxygen Species and Nitric Oxide in Vascular Physiology and Pathology.Anti-inflammatory immune skewing is atheroprotective: Apoe−/−FcγRIIb−/− mice develop fibrous carotid plaques.Oxidized LDL/CD36 interaction induces loss of cell polarity and inhibits macrophage locomotion.CD36, a scavenger receptor involved in immunity, metabolism, angiogenesis, and behavior.Type 2 scavenger receptor CD36 in platelet activation: the role of hyperlipemia and oxidative stress.CD36, a scavenger receptor implicated in atherosclerosis.CD36 deletion improves recovery from spinal cord injury.Oxidized LDL: diversity, patterns of recognition, and pathophysiology.Class B scavenger receptor types I and II and CD36 mediate bacterial recognition and proinflammatory signaling induced by Escherichia coli, lipopolysaccharide, and cytosolic chaperonin 60 Internalization of modified lipids by CD36 and SR-A leads to hepatic inflammation and lysosomal cholesterol storage in Kupffer cells.Macrophages in atherosclerosis: a dynamic balance.Inflammatory therapeutic targets in coronary atherosclerosis-from molecular biology to clinical application.Mipu1 overexpression protects macrophages from oxLDL-induced foam cell formation and cell apoptosis Vav family Rho guanine nucleotide exchange factors regulate CD36-mediated macrophage foam cell formation.Hematopoietic cell-restricted deletion of CD36 reduces high-fat diet-induced macrophage infiltration and improves insulin signaling in adipose tissue CD36-dependent 7-ketocholesterol accumulation in macrophages mediates progression of atherosclerosis in response to chronic air pollution exposure.The role of macrophage class a scavenger receptors in a laser-induced murine choroidal neovascularization model.Role of scavenger receptor A and CD36 in diet-induced nonalcoholic steatohepatitis in hyperlipidemic mice.Adiponectin receptor 2 deficiency results in reduced atherosclerosis in the brachiocephalic artery in apolipoprotein E deficient mice Enhanced CD36 scavenger receptor expression in THP-1 human monocytes in the presence of lupus plasma: linking autoimmunity and atherosclerosis D4F alleviates macrophage-derived foam cell apoptosis by inhibiting CD36 expression and ER stress-CHOP pathway CD36/SR-B2-TLR2 Dependent Pathways Enhance Porphyromonas gingivalis Mediated Atherosclerosis in the Ldlr KO Mouse Model.Myeloid deletion of nuclear factor erythroid 2-related factor 2 increases atherosclerosis and liver injury.Salvianolic acid B inhibits macrophage uptake of modified low density lipoprotein (mLDL) in a scavenger receptor CD36-dependent manner.Endothelial cell activation promotes foam cell formation by monocytes following transendothelial migration in an in vitro model.The gut microbiota and its relationship to diet and obesity: new insights Cleavage and reduced CD36 ectodomain density on heart and spleen macrophages in the spontaneously hypertensive rat.Class A1 scavenger receptors in cardiovascular diseases.CD36 and Na/K-ATPase-α1 form a proinflammatory signaling loop in kidney.Antagonism of scavenger receptor CD36 by 5A peptide prevents chronic kidney disease progression in mice independent of blood pressure regulation.Degradation of aggregated LDL occurs in complex extracellular sub-compartments of the lysosomal synapse.Dominant negative PPARγ promotes atherosclerosis, vascular dysfunction, and hypertension through distinct effects in endothelium and vascular muscle ApoE deficiency exacerbates the development and sustainment of a semi-chronic K/BxN serum transfer-induced arthritis model CD36 modulates migration of mouse and human macrophages in response to oxidized LDL and may contribute to macrophage trapping in the arterial intima.Loss of SR-A and CD36 activity reduces atherosclerotic lesion complexity without abrogating foam cell formation in hyperlipidemic mice.

P2860

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P2860

Absence of CD36 protects against atherosclerosis in ApoE knock-out mice with no additional protection provided by absence of scavenger receptor A I/II

http://www.wikidata.org/entity/Q24658096

description

2008 nî lūn-bûn

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2008 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2008 թվականի ապրիլին հրատարակված գիտական հոդված

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年论文

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name

Absence of CD36 protects again ...... e of scavenger receptor A I/II

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Absence of CD36 protects again ...... e of scavenger receptor A I/II

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Absence of CD36 protects again ...... e of scavenger receptor A I/II

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type

label

Absence of CD36 protects again ...... e of scavenger receptor A I/II

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Absence of CD36 protects again ...... e of scavenger receptor A I/II

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Absence of CD36 protects again ...... e of scavenger receptor A I/II

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Absence of CD36 protects again ...... e of scavenger receptor A I/II

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Absence of CD36 protects again ...... e of scavenger receptor A I/II

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Absence of CD36 protects again ...... e of scavenger receptor A I/II

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Cardiovascular Research

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Absence of CD36 protects again ...... e of scavenger receptor A I/II

@en

P2093

David J Kennedy

http://www.w3.org/2001/XMLSchema#string

Difernando Vanegas

http://www.w3.org/2001/XMLSchema#string

Ella Guy

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George Nimako

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Maria Febbraio

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Richard E Morton

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Sai Kuchibhotla

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P2860

Macrophage scavenger receptor CD36 is the major receptor for LDL modified by monocyte-generated reactive nitrogen species

Scavenger receptors class A-I/II and CD36 are the principal receptors responsible for the uptake of modified low density lipoprotein leading to lipid loading in macrophages

PPARgamma promotes monocyte/macrophage differentiation and uptake of oxidized LDL

Endoplasmic reticulum stress: cell life and death decisions.

Targeted disruption of the class B scavenger receptor CD36 protects against atherosclerotic lesion development in mice

A PPAR gamma-LXR-ABCA1 pathway in macrophages is involved in cholesterol efflux and atherogenesis

CD36 is a sensor of diacylglycerides

Interferon-gamma and atherosclerosis: pro- or anti-atherogenic?

Macrophage scavenger receptor class A: A multifunctional receptor in atherosclerosis.

Loss of receptor-mediated lipid uptake via scavenger receptor A or CD36 pathways does not ameliorate atherosclerosis in hyperlipidemic mice

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185-96

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scholarly article

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895845

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10.1093/CVR/CVM093

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1

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78

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2008-04-01T00:00:00Z

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5783773

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18065445

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P921

atherosclerosis

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2810680

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