The extracellular matrix, p53 and estrogen compete to regulate cell-surface Fas/Apo-1 suicide receptor expression in proliferating embryonic cerebral cortical precursors, and reciprocally, Fas-ligand modifies estrogen control of cell-cycle proteins

The extracellular matrix, p53 and estrogen compete to regulate cell-surface Fas/Apo-1 suicide receptor expression in proliferating embryonic cerebral cortical precursors, and reciprocally, Fas-ligand modifies estrogen control of cell-cycle proteins

Item
http://www.wikidata.org/entity/Q24791837
Ethanol induces cell-cycle activity and reduces stem cell diversity to alter both regenerative capacity and differentiation potential of cerebral cortical neuroepithelial precursors.Transplantation of human neural precursor cells in Matrigel scaffolding improves outcome from focal cerebral ischemia after delayed postischemic treatment in rats.Ethanol regulates angiogenic cytokines during neural development: evidence from an in vitro model of mitogen-withdrawal-induced cerebral cortical neuroepithelial differentiation.Modeling the impact of alcohol on cortical development in a dish: strategies from mapping neural stem cell fate.Embryonic cerebral cortical progenitors are resistant to apoptosis, but increase expression of suicide receptor DISC-complex genes and suppress autophagy following ethanol exposureCompeting interactions between micro-RNAs determine neural progenitor survival and proliferation after ethanol exposure: evidence from an ex vivo model of the fetal cerebral cortical neuroepithelium.p53 and Cell Cycle Proteins Participate in Spinal Motor Neuron Cell Death in ALSApoptosis related to steroid sulfatase at the cone-shaped projection in nephron morphogenesis.Tumor necrosis factor p55 and p75 receptors are involved in chemical-induced apoptosis of dentate granule neurons.Nell-1 induces acrania-like cranioskeletal deformities during mouse embryonic development.Steroid and Xenobiotic Receptor Signalling in Apoptosis and Autophagy of the Nervous System.Control of neuronal branching by the death receptor CD95 (Fas/Apo-1).
P2860

The extracellular matrix, p53 and estrogen compete to regulate cell-surface Fas/Apo-1 suicide receptor expression in proliferating embryonic cerebral cortical precursors, and reciprocally, Fas-ligand modifies estrogen control of cell-cycle proteins

Item
http://www.wikidata.org/entity/Q24791837
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2004 nî lūn-bûn
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2004 թուականի Մարտին հրատարակուած գիտական յօդուած
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2004 թվականի մարտին հրատարակված գիտական հոդված
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2004年の論文
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2004年論文
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2004年論文
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2004年論文
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2004年論文
@zh-mo
2004年論文
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2004年论文
@wuu
name
The extracellular matrix, p53 ...... control of cell-cycle proteins
@ast
The extracellular matrix, p53 ...... control of cell-cycle proteins
@en
The extracellular matrix, p53 ...... control of cell-cycle proteins
@nl
type
label
The extracellular matrix, p53 ...... control of cell-cycle proteins
@ast
The extracellular matrix, p53 ...... control of cell-cycle proteins
@en
The extracellular matrix, p53 ...... control of cell-cycle proteins
@nl
prefLabel
The extracellular matrix, p53 ...... control of cell-cycle proteins
@ast
The extracellular matrix, p53 ...... control of cell-cycle proteins
@en
The extracellular matrix, p53 ...... control of cell-cycle proteins
@nl
P1433
P1476
P2093
P2860
P2888
P304
P31
P356
P407
P433
P478
P50
P577
P5875
P6179
P698
P921
P932
P356
P1433
P1476
The extracellular matrix, p53 ...... control of cell-cycle proteins
@en
P2093
P2860
P2888
P304
P31
P356
P407
P433
P478
P50
P577
P5875
P6179
P698
P921
P932