Catechol-O-methyltransferase (COMT) Val108/158 Met polymorphism does not modulate executive function in children with ADHD - Wikidata - awgldk - TriplyDB

Catechol-O-methyltransferase (COMT) Val108/158 Met polymorphism does not modulate executive function in children with ADHD

Catechol-O-methyltransferase (COMT) Val108/158 Met polymorphism does not modulate executive function in children with ADHD

http://www.wikidata.org/entity/Q24802440

about

Candidate genes and neuropsychological phenotypes in children with ADHD: review of association studies Dopamine transporter 3'UTR VNTR genotype is a marker of performance on executive function tasks in children with ADHD.The endophenotype concept in psychiatric genetics.Neurobehavioral evidence for changes in dopamine system activity during adolescence.COMT Val108/158Met polymorphism and the modulation of task-oriented behavior in children with ADHD.Influence of the COMT val(108/158)met polymorphism on continuous performance task indices.Comprehensive phenotype/genotype analyses of the norepinephrine transporter gene (SLC6A2) in ADHD: relation to maternal smoking during pregnancy.The COMT Val158 allele is associated with impaired delayed-match-to-sample performance in ADHD.Are endophenotypes based on measures of executive functions useful for molecular genetic studies of ADHD?The molecular genetics of cognition: dopamine, COMT and BDNF.A comparison of molecular alterations in environmental and genetic rat models of ADHD: a pilot study Neuropsychological endophenotypes in attention-deficit/hyperactivity disorder: a review of genetic association studies.Differences in planning performance, a neurocognitive endophenotype, are associated with a functional variant in PER3 gene.Association between obesity-related gene FTO and ADHD.DRD3, but not COMT or DRD2, genotype affects executive functions in healthy and first-episode psychosis adolescents.EEG Source Imaging Indices of Cognitive Control Show Associations with Dopamine System Genes.Genetic evidence for the association of the hypothalamic-pituitary-adrenal (HPA) axis with ADHD and methylphenidate treatment response.The divergent impact of COMT Val158Met on executive function in children with and without attention-deficit/hyperactivity disorder.Does adult ADHD interact with COMT val (158) met genotype to influence working memory performance?Effects of the catechol-O-methyltransferase Val158Met polymorphism on executive function: a meta-analysis of the Wisconsin Card Sort Test in schizophrenia and healthy controls

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Catechol-O-methyltransferase (COMT) Val108/158 Met polymorphism does not modulate executive function in children with ADHD

http://www.wikidata.org/entity/Q24802440

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2004 nî lūn-bûn

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2004 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

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2004年の論文

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2004年論文

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2004年論文

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2004年論文

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2004年論文

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2004年論文

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BMC Medical Genetics

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Catechol-O-methyltransferase ( ...... function in children with ADHD

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Adam Torkaman-Zehi

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Chantal Baron

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Evan Taerk

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Leila Ben Amor

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Marina Ter Stepanian

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Natalie Grizenko

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Philippe Lageix

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Ridha Joober

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Rosherie Deguzman

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Valentin Mbekou

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P2860

Hyperlocomotion and indifference to cocaine and amphetamine in mice lacking the dopamine transporter

Catechol-O-methyltransferase-deficient mice exhibit sexually dimorphic changes in catecholamine levels and behavior.

No evidence of association between Catechol-O-Methyltransferase (COMT) Val158Met genotype and performance on neuropsychological tasks in children with ADHD: a case-control study

Dopamine uptake through the norepinephrine transporter in brain regions with low levels of the dopamine transporter: evidence from knock-out mouse lines

Specific impairments of planning

Behavioral inhibition, sustained attention, and executive functions: constructing a unifying theory of ADHD

Haplotype relative risk study of catechol-O-methyltransferase (COMT) and attention deficit hyperactivity disorder (ADHD): association of the high-enzyme activity Val allele with ADHD impulsive-hyperactive phenotype

Effect of COMT Val108/158 Met genotype on frontal lobe function and risk for schizophrenia

Characterization of extracellular dopamine clearance in the medial prefrontal cortex: role of monoamine uptake and monoamine oxidase inhibition.

Haplotype study of three polymorphisms at the dopamine transporter locus confirm linkage to attention-deficit/hyperactivity disorder.

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10.1186/1471-2350-5-30

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1045224701

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