about
Transforming growth factor-beta1, transforming growth factor-beta2, and transforming growth factor-beta3 enhance ovarian cancer metastatic potential by inducing a Smad3-dependent epithelial-to-mesenchymal transitionSTAMP alters the growth of transformed and ovarian cancer cells.CCR9 interactions support ovarian cancer cell survival and resistance to cisplatin-induced apoptosis in a PI3K-dependent and FAK-independent fashion.CCL25-CCR9 interaction modulates ovarian cancer cell migration, metalloproteinase expression, and invasionChicken pleiotrophin: regulation of tissue specific expression by estrogen in the oviduct and distinct expression pattern in the ovarian carcinomasAcellular fraction of ovarian cancer ascites induce apoptosis by activating JNK and inducing BRCA1, Fas and FasL expression in ovarian cancer cells.Measuring the intra-individual variability of the plasma proteome in the chicken model of spontaneous ovarian adenocarcinoma.Differential expression of alpha 2 macroglobulin in response to dietylstilbestrol and in ovarian carcinomas in chickensIsolation and characterization of stromal progenitor cells from ascites of patients with epithelial ovarian adenocarcinoma.Characterization of ovarian cancer ascites on cell invasion, proliferation, spheroid formation, and gene expression in an in vitro model of epithelial ovarian cancer.Molecular mechanisms and biological plausibility underlying the malignant transformation of endometriosis: a critical analysis.Gene methylation of human ovarian carcinoma stromal progenitor cells promotes tumorigenesisThe role of activin A and Akt/GSK signaling in ovarian tumor biology.Expression and histopathological correlation of CCR9 and CCL25 in ovarian cancer.Future directions in the management of epithelial ovarian cancer.Dual detection of cancer biomarker CA125 using absorbance and electrochemical methods.The C-terminus of IGFBP-5 suppresses tumor growth by inhibiting angiogenesisEffect of ovarian cancer ascites on cell migration and gene expression in an epithelial ovarian cancer in vitro model.Spheroid growth in ovarian cancer alters transcriptome responses for stress pathways and epigenetic responses.The Proangiogenic Capabilities of Malignant Ascites Generated by Aggressive Ovarian Tumors.Mediation analysis of alcohol consumption, DNA methylation, and epithelial ovarian cancer.Biochemical composition of malignant ascites determines high aggressiveness of undifferentiated ovarian tumors.Ovarian cancer-derived ascitic fluids induce a senescence-dependent pro-cancerogenic phenotype in normal peritoneal mesothelial cells.
P2860
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P2860
description
2003 nî lūn-bûn
@nan
2003 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2003 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
name
Early events in ovarian oncogenesis
@ast
Early events in ovarian oncogenesis
@en
Early events in ovarian oncogenesis
@nl
type
label
Early events in ovarian oncogenesis
@ast
Early events in ovarian oncogenesis
@en
Early events in ovarian oncogenesis
@nl
prefLabel
Early events in ovarian oncogenesis
@ast
Early events in ovarian oncogenesis
@en
Early events in ovarian oncogenesis
@nl
P2860
P921
P356
P1476
Early events in ovarian oncogenesis
@en
P2093
Dusica Cvetkovic
P2860
P2888
P356
10.1186/1477-7827-1-68
P407
P577
2003-10-07T00:00:00Z
P5875
P6179
1032553834