Regulatory variation at glypican-3 underlies a major growth QTL in mice - Wikidata - awgldk - TriplyDB

Regulatory variation at glypican-3 underlies a major growth QTL in mice

Regulatory variation at glypican-3 underlies a major growth QTL in mice

http://www.wikidata.org/entity/Q24806137

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Ancient and recent positive selection transformed opioid cis-regulation in humans Overgrowth of a mouse model of Simpson-Golabi-Behmel syndrome is partly mediated by Indian hedgehog Genetics of Rapid and Extreme Size Evolution in Island Mice Allele-specific KRT1 expression is a complex trait.A wild derived quantitative trait locus on mouse chromosome 2 prevents obesity.Quantitative trait loci for energy balance traits in an advanced intercross line derived from mice divergently selected for heat loss.The glypican 3-hosted murine mir717 gene: sequence conservation, seed region polymorphisms and putative targets.Analysis of allelic differential expression in human white blood cells.PAPPA2, an enzyme that cleaves an insulin-like growth-factor-binding protein, is a candidate gene for a quantitative trait locus affecting body size in mice Dissection of a genetic locus influencing renal function in the rat and its concordance with kidney disease loci on human chromosome 1q21 Fine mapping reveals sex bias in quantitative trait loci affecting growth, skeletal size and obesity-related traits on mouse chromosomes 2 and 11.Mapping genetic loci that interact with myostatin to affect growth traits.RNA world - the dark matter of evolutionary genomics.Genome clashes in hybrids: insights from gene expression.The genetic structure of domestic rabbits Glypican-1 controls brain size through regulation of fibroblast growth factor signaling in early neurogenesis.LRP1 mediates Hedgehog-induced endocytosis of the GPC3-Hedgehog complex.The genetic architecture of growth rate in juvenile Takifugu species.Identification of positional candidate genes for body weight and adiposity in subcongenic mice.Intersubspecific subcongenic mouse strain analysis reveals closely linked QTLs with opposite effects on body weight.Genetics of metabolic syndrome: potential clues from wild-derived inbred mouse strains.Genetical genomics of growth in a chicken model.Marker-assisted selection based on a multi-trait economic index in chicken: Experimental results and simulation.

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Regulatory variation at glypican-3 underlies a major growth QTL in mice

http://www.wikidata.org/entity/Q24806137

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2005 nî lūn-bûn

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2005 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2005 թվականի մայիսին հրատարակված գիտական հոդված

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2005年の論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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2005年论文

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name

Regulatory variation at glypican-3 underlies a major growth QTL in mice

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Regulatory variation at glypican-3 underlies a major growth QTL in mice

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Regulatory variation at glypican-3 underlies a major growth QTL in mice

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Regulatory variation at glypican-3 underlies a major growth QTL in mice

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Regulatory variation at glypican-3 underlies a major growth QTL in mice

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Regulatory variation at glypican-3 underlies a major growth QTL in mice

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prefLabel

Regulatory variation at glypican-3 underlies a major growth QTL in mice

@ast

Regulatory variation at glypican-3 underlies a major growth QTL in mice

@en

Regulatory variation at glypican-3 underlies a major growth QTL in mice

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JOURNAL.PBIO.0030135

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Regulatory variation at glypican-3 underlies a major growth QTL in mice

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Claire Davison

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Fiona Oliver

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Peter D Keightley

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Steve D M Brown

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Susan Rhind

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Vinesh Verma

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Xiaojun Liu

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P2860

Mutations in GPC3, a glypican gene, cause the Simpson-Golabi-Behmel overgrowth syndrome

K-glypican: a novel GPI-anchored heparan sulfate proteoglycan that is highly expressed in developing brain and kidney

Combining mapping and arraying: An approach to candidate gene identification.

A gene atlas of the mouse and human protein-encoding transcriptomes

The functional landscape of mouse gene expression

Glypican-3-deficient mice exhibit developmental overgrowth and some of the abnormalities typical of Simpson-Golabi-Behmel syndrome

Mutational analysis of the GPC3/GPC4 glypican gene cluster on Xq26 in patients with Simpson-Golabi-Behmel syndrome: identification of loss-of-function mutations in the GPC3 gene

Simpson Golabi Behmel syndrome: progress toward understanding the molecular basis for overgrowth, malformation, and cancer predisposition

Regulation of bone mass in mice by the lipoxygenase gene Alox15

Positional identification of Ncf1 as a gene that regulates arthritis severity in rats

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Julian K Christians

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