about
SAHA ameliorates the SMA phenotype in two mouse models for spinal muscular atrophyEffects of lithium and valproic acid on gene expression and phenotypic markers in an NT2 neurosphere model of neural developmentMicroRNA profiling as tool for in vitro developmental neurotoxicity testing: the case of sodium valproateMetabolomics in the assessment of chemical-induced reproductive and developmental outcomes using non-invasive biological fluids: application to the study of butylbenzyl phthalateGenetic and maternal effects on valproic acid teratogenesis in C57BL/6J and DBA/2J mice.Interaction networks of lithium and valproate molecular targets reveal a striking enrichment of apoptosis functional clusters and neurotrophin signalingEpidemiology, prenatal management, and prevention of neural tube defects.Alternative methods for developmental toxicity testing.Inhibition of histone deacetylase as a new mechanism of teratogenesis.Teratogenicity of antiepileptic drugs: role of drug metabolism and pharmacogenomics.Risk factors for neural tube defects in Riyadh City, Saudi Arabia: Case-control studyKey developments in endocrine disrupter research and human health.The clinical impact of pharmacogenetics on the treatment of epilepsy.Epilepsy pharmacogenetics.A human pluripotent stem cell platform for assessing developmental neural toxicity screeningA role for cytoskeletal protein acetylation in modulating myometrial activity.Transgelin-2: A potential oncogenic factor.Ethanol exposure alters protein expression in a mouse model of fetal alcohol spectrum disordersEpigenetic changes and disturbed neural development in a human embryonic stem cell-based model relating to the fetal valproate syndrome.Ascorbic acid reverses valproic acid-induced inhibition of hoxa2 and maintains glutathione homeostasis in mouse embryos in culture.Microarray analysis of murine limb bud ectoderm and mesoderm after exposure to cadmium or acetazolamide.Identification of early-responsive genes correlated to valproic acid-induced neural tube defects in mice.Resveratrol and vitamin E rescue valproic acid-induced teratogenicity: the mechanism of action.Expression of homeotic genes Hoxa3, Hoxb3, Hoxd3 and Hoxc4 is decreased in the lungs but not in the hearts of adriamycin-exposed mice.Molecular approaches to developmental malformations using analogous forms of valproic acid.Reduction in valproic acid-induced neural tube defects by maternal immune stimulation: role of apoptosis.
P2860
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P2860
description
2004 nî lūn-bûn
@nan
2004 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2004 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
name
Valproic acid teratogenicity: a toxicogenomics approach
@ast
Valproic acid teratogenicity: a toxicogenomics approach
@en
Valproic acid teratogenicity: a toxicogenomics approach
@nl
type
label
Valproic acid teratogenicity: a toxicogenomics approach
@ast
Valproic acid teratogenicity: a toxicogenomics approach
@en
Valproic acid teratogenicity: a toxicogenomics approach
@nl
prefLabel
Valproic acid teratogenicity: a toxicogenomics approach
@ast
Valproic acid teratogenicity: a toxicogenomics approach
@en
Valproic acid teratogenicity: a toxicogenomics approach
@nl
P2093
P2860
P356
P1476
Valproic acid teratogenicity: a toxicogenomics approach
@en
P2093
Anna-Maja Nyström
Anne-Lee Gustafson
Birger Scholz
Kim Kultima
Lennart Dencker
Michael Stigson
P2860
P304
P356
10.1289/EHP.7034
P407
P577
2004-08-01T00:00:00Z