Cu/Zn superoxide dismutase expression in the postnatal rat brain following an excitotoxic injury.
about
Neuroprotection from NMDA excitotoxic lesion by Cu/Zn superoxide dismutase gene delivery to the postnatal rat brain by a modular protein vector.Protective effects of phosphodiesterase 2 inhibitor on depression- and anxiety-like behaviors: involvement of antioxidant and anti-apoptotic mechanismsCD300f immunoreceptor contributes to peripheral nerve regeneration by the modulation of macrophage inflammatory phenotype.Oxidative stress in immature brain after traumatic brain injury.Gel-based hippocampal proteomic analysis 2 weeks following traumatic brain injury to immature rats using controlled cortical impact.Novel therapeutic targets in depression and anxiety: antioxidants as a candidate treatment.Managing odds in stem cells: insights into the role of mitochondrial antioxidant enzyme MnSOD.Involvement of Neuroinflammation during Brain Development in Social Cognitive Deficits in Autism Spectrum Disorder and Schizophrenia.Quantitative proteomic analysis of induced pluripotent stem cells derived from a human Huntington's disease patient.Tanshinone IIA Inhibits Glutamate-Induced Oxidative Toxicity through Prevention of Mitochondrial Dysfunction and Suppression of MAPK Activation in SH-SY5Y Human Neuroblastoma Cells.Overexpression of the nuclear factor kappaB inhibitor A20 is neurotoxic after an excitotoxic injury to the immature rat brain.Hyperbaric oxygenation alters temporal expression pattern of superoxide dismutase 2 after cortical stab injury in rats.Protective reactivity and alteration of the brain tissue in alcoholics evidenced by SOD1, MMP9 immunohistochemistry, and electron microscopy.Interleukin-10 overexpression does not synergize with the neuroprotective action of RGD-containing vectors after postnatal brain excitotoxicity but modulates the main inflammatory cell responses.Interleukin-10 and interleukin-10 receptor-I are upregulated in glial cells after an excitotoxic injury to the postnatal rat brain.Time course of heme oxygenase-1 and oxidative stress after experimental intracerebral hemorrhage.Characterization of traumatic brain injury in human brains reveals distinct cellular and molecular changes in contusion and pericontusion.Recombinant PEP-1-SOD1 improves functional recovery after neural stem cell transplantation in rats with traumatic brain injury.
P2860
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P2860
Cu/Zn superoxide dismutase expression in the postnatal rat brain following an excitotoxic injury.
description
2005 nî lūn-bûn
@nan
2005 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
name
Cu/Zn superoxide dismutase exp ...... ollowing an excitotoxic injury
@nl
Cu/Zn superoxide dismutase exp ...... llowing an excitotoxic injury.
@ast
Cu/Zn superoxide dismutase exp ...... llowing an excitotoxic injury.
@en
type
label
Cu/Zn superoxide dismutase exp ...... ollowing an excitotoxic injury
@nl
Cu/Zn superoxide dismutase exp ...... llowing an excitotoxic injury.
@ast
Cu/Zn superoxide dismutase exp ...... llowing an excitotoxic injury.
@en
prefLabel
Cu/Zn superoxide dismutase exp ...... ollowing an excitotoxic injury
@nl
Cu/Zn superoxide dismutase exp ...... llowing an excitotoxic injury.
@ast
Cu/Zn superoxide dismutase exp ...... llowing an excitotoxic injury.
@en
P2093
P2860
P356
P1476
Cu/Zn superoxide dismutase exp ...... llowing an excitotoxic injury.
@en
P2093
Bernardo Castellano
Berta Gonzalez
Hugo Peluffo
Laia Acarin
Maryam Faiz
P2860
P2888
P356
10.1186/1742-2094-2-12
P577
2005-06-01T00:00:00Z
P5875
P6179
1020321245