Inhibition of endothelial activation: a new way to treat cerebral malaria? - Wikidata - awgldk - TriplyDB

Inhibition of endothelial activation: a new way to treat cerebral malaria?

Inhibition of endothelial activation: a new way to treat cerebral malaria?

http://www.wikidata.org/entity/Q24814891

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Investigating the Pathogenesis of Severe Malaria: A Multidisciplinary and Cross-Geographical Approach Microparticles as mediators and biomarkers of rheumatic disease Infectious Diseases of the Nervous System and Their Impact in Developing Countries Inhibition of Plasmodium falciparum field isolates-mediated endothelial cell apoptosis by Fasudil: therapeutic implications for severe malaria Genome-wide association study indicates two novel resistance loci for severe malaria A novel role for von Willebrand factor in the pathogenesis of experimental cerebral malaria Comparison of apoptosis in human primary pulmonary endothelial cells and a brain microvascular endothelial cell line co-cultured with Plasmodium falciparum field isolates.The systemic pathology of cerebral malaria in African children A subset of group A-like var genes encodes the malaria parasite ligands for binding to human brain endothelial cells Inflammatory responses associated with the induction of cerebral malaria: lessons from experimental murine models Dysregulation of coagulation in cerebral malaria.Rosetting Plasmodium falciparum-infected erythrocytes bind to human brain microvascular endothelial cells in vitro, demonstrating a dual adhesion phenotype mediated by distinct P. falciparum erythrocyte membrane protein 1 domains.Inhibition of the multidrug resistance P-glycoprotein: time for a change of strategy?Peroxisome proliferator activating receptor (PPAR) in cerebral malaria (CM): a novel target for an additional therapy.Malaria: modification of the red blood cell and consequences in the human host.Do multiple concurrent infections in African children cause irreversible immunological damage?Cerebral malaria: we have come a long way Cellular Models and In Vitro Assays for the Screening of modulators of P-gp, MRP1 and BCRP.Metabolic acidosis induced by Plasmodium falciparum intraerythrocytic stages alters blood-brain barrier integrity.LMP-420, a small-molecule inhibitor of TNF-alpha, reduces replication of HIV-1 and Mycobacterium tuberculosis in human cells.Hyperbaric oxygen prevents early death caused by experimental cerebral malaria.Do we know enough to find an adjunctive therapy for cerebral malaria in African children?

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P2860

Inhibition of endothelial activation: a new way to treat cerebral malaria?

http://www.wikidata.org/entity/Q24814891

description

2005 nî lūn-bûn

@nan

2005 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2005 թվականի օգոստոսին հրատարակված գիտական հոդված

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2005年の論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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2005年论文

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name

Inhibition of endothelial activation: a new way to treat cerebral malaria?

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Inhibition of endothelial activation: a new way to treat cerebral malaria?

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Inhibition of endothelial activation: a new way to treat cerebral malaria?

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Inhibition of endothelial activation: a new way to treat cerebral malaria?

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Inhibition of endothelial activation: a new way to treat cerebral malaria?

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Inhibition of endothelial activation: a new way to treat cerebral malaria?

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Inhibition of endothelial activation: a new way to treat cerebral malaria?

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Inhibition of endothelial activation: a new way to treat cerebral malaria?

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Inhibition of endothelial activation: a new way to treat cerebral malaria?

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Inhibition of endothelial activation: a new way to treat cerebral malaria?

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George J Cianciolo

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Georges E Grau

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Samuel C Wassmer

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Valéry Combes

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P2860

Human malaria parasites in continuous culture

Glucocorticoids selectively inhibit the transcription of the interleukin 1 beta gene and decrease the stability of interleukin 1 beta mRNA.

Biological and biochemical characteristics of cytoadhesion of Plasmodium falciparum-infected erythrocytes to chondroitin-4-sulfate.

Cytokine pathways and joint inflammation in rheumatoid arthritis.

Adalimumab, a fully human anti-tumor necrosis factor alpha monoclonal antibody, for the treatment of rheumatoid arthritis in patients taking concomitant methotrexate: the ARMADA trial.

Cytokines: accelerators and brakes in the pathogenesis of cerebral malaria.

Ultrastructure of malaria-infected erythrocytes.

Cytoadherence and sequestration in falciparum malaria.

Locally up-regulated lymphotoxin alpha, not systemic tumor necrosis factor alpha, is the principle mediator of murine cerebral malaria.

Receptor-specific adhesion and clinical disease in Plasmodium falciparum.

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10.1371/JOURNAL.PMED.0020245

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16104828

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cerebral malaria

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