The estrogen receptor-alpha A908G (K303R) mutation occurs at a low frequency in invasive breast tumors: results from a population-based study - Wikidata - awgldk - TriplyDB

The estrogen receptor-alpha A908G (K303R) mutation occurs at a low frequency in invasive breast tumors: results from a population-based study

The estrogen receptor-alpha A908G (K303R) mutation occurs at a low frequency in invasive breast tumors: results from a population-based study

http://www.wikidata.org/entity/Q25257794

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Tamoxifen Resistance: Emerging Molecular Targets Regulation of Estrogen Receptor α by the SET7 Lysine Methyltransferase Structural Basis for Ca2+-induced Activation and Dimerization of Estrogen Receptor by Calmodulin BRCA1 regulates acetylation and ubiquitination of estrogen receptor-alpha.Expression of the K303R estrogen receptor-alpha breast cancer mutation induces resistance to an aromatase inhibitor via addiction to the PI3K/Akt kinase pathway.Leptin mediates tumor-stromal interactions that promote the invasive growth of breast cancer cells Estrogen receptor (ER) α mutations in breast cancer: hidden in plain sight Phosphorylation of the mutant K303R estrogen receptor alpha at serine 305 affects aromatase inhibitor sensitivity Estrogen receptor mutations and changes in downstream gene expression and signaling Growth factor-induced resistance to tamoxifen is associated with a mutation of estrogen receptor alpha and its phosphorylation at serine 305.Association between the estrogen receptor alpha A908G mutation and outcomes in invasive breast cancer.Accelerated mammary maturation and differentiation, and delayed MMTVneu-induced tumorigenesis of K303R mutant ERalpha transgenic mice.Post-translational modifications of nuclear receptors and human disease Risk factors for breast cancer characterized by the estrogen receptor alpha A908G (K303R) mutation Absence of the K303R estrogen receptor α mutation in breast cancer patients exhibiting different responses to aromatase inhibitor anastrozole neoadjuvant treatment.Association of estrogen receptor-α A908G (K303R) mutation with breast cancer risk.Regulatory function of the P295-T311 motif of the estrogen receptor alpha - does proteasomal degradation of the receptor induce emergence of peptides implicated in estrogenic responses?Sensitive Detection of Mono- and Polyclonal ESR1 Mutations in Primary Tumors, Metastatic Lesions, and Cell-Free DNA of Breast Cancer Patients.ESR1 mutations—a mechanism for acquired endocrine resistance in breast cancer.Epigenetics meets estrogen receptor: regulation of estrogen receptor by direct lysine methylation.Regulation of estrogen receptor signaling in breast carcinogenesis and breast cancer therapy.Mutations and polymorphisms of estrogens receptors genes and diseases susceptibility.Chronic oxidative stress causes estrogen-independent aggressive phenotype, and epigenetic inactivation of estrogen receptor alpha in MCF-7 breast cancer cells.

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The estrogen receptor-alpha A908G (K303R) mutation occurs at a low frequency in invasive breast tumors: results from a population-based study

http://www.wikidata.org/entity/Q25257794

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2005 nî lūn-bûn

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2005 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

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2005 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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Breast Cancer Research

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Beth Newman

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Chad A Livasy

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Chiu-Kit Tse

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Dawn Tolbert

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Robert C Millikan

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Sharon N Edmiston

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Rapid p53 sequence analysis in primary lung cancer using an oligonucleotide probe array

Hypersensitive K303R oestrogen receptor-alpha variant not found in invasive carcinomas

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Manganese citrate improves base-calling accuracy in DNA sequencing reactions using rhodamine-based fluorescent dye-terminators.

Phosphorylation of estrogen receptor alpha blocks its acetylation and regulates estrogen sensitivity.

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10.1186/BCR1315

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