Cystic Fibrosis Lung Infections: Polymicrobial, Complex, and Hard to Treat
about
Pseudomonas aeruginosa Alginate Overproduction Promotes Coexistence with Staphylococcus aureus in a Model of Cystic Fibrosis Respiratory InfectionPseudomonas aeruginosa Alters Staphylococcus aureus Sensitivity to Vancomycin in a Biofilm Model of Cystic Fibrosis Infection.Pyrosequencing Unveils Cystic Fibrosis Lung Microbiome Differences Associated with a Severe Lung Function DeclineA commensal streptococcus hijacks a Pseudomonas aeruginosa exopolysaccharide to promote biofilm formation.A Low-Molecular-Weight Alginate Oligosaccharide Disrupts Pseudomonal Microcolony Formation and Enhances Antibiotic Effectiveness.Bile signalling promotes chronic respiratory infections and antibiotic toleranceLong-Term Evolution of Burkholderia multivorans during a Chronic Cystic Fibrosis Infection Reveals Shifting Forces of SelectionSputum DNA sequencing in cystic fibrosis: non-invasive access to the lung microbiome and to pathogen details.Phenotypic diversity and genotypic flexibility of Burkholderia cenocepacia during long-term chronic infection of cystic fibrosis lungs.Role of vitamin D in cystic fibrosis and non-cystic fibrosis bronchiectasis.Interactions between Pseudomonas aeruginosa and Staphylococcus aureus during co-cultivations and polymicrobial infections.Polymicrobial infections involving clinically relevant Gram-negative bacteria and fungi.Understanding the Entanglement: Neutrophil Extracellular Traps (NETs) in Cystic FibrosisA pan-genomic approach to understand the basis of host adaptation in Achromobacter.Developing selective media for quantification of multispecies biofilms following antibiotic treatment.Anti-Psl Targeting of Pseudomonas aeruginosa Biofilms for Neutrophil-Mediated Disruption.Optimization of a meropenem plus tobramycin combination dosage regimen against hypermutable and non-hypermutable Pseudomonas aeruginosa via mechanism-based modeling and the hollow-fiber infection model.Cigarette Smoke-Induced Acquired Dysfunction of Cystic Fibrosis Transmembrane Conductance Regulator in the Pathogenesis of Chronic Obstructive Pulmonary Disease.Hypertonic Saline Suppresses NADPH Oxidase-Dependent Neutrophil Extracellular Trap Formation and Promotes Apoptosis.Pathogen Identification Direct From Polymicrobial Specimens Using Membrane Glycolipids
P2860
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P2860
Cystic Fibrosis Lung Infections: Polymicrobial, Complex, and Hard to Treat
description
2015 nî lūn-bûn
@nan
2015 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2015 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2015年の論文
@ja
2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
name
Cystic Fibrosis Lung Infections: Polymicrobial, Complex, and Hard to Treat
@ast
Cystic Fibrosis Lung Infections: Polymicrobial, Complex, and Hard to Treat
@en
Cystic Fibrosis Lung Infections: Polymicrobial, Complex, and Hard to Treat
@nl
type
label
Cystic Fibrosis Lung Infections: Polymicrobial, Complex, and Hard to Treat
@ast
Cystic Fibrosis Lung Infections: Polymicrobial, Complex, and Hard to Treat
@en
Cystic Fibrosis Lung Infections: Polymicrobial, Complex, and Hard to Treat
@nl
prefLabel
Cystic Fibrosis Lung Infections: Polymicrobial, Complex, and Hard to Treat
@ast
Cystic Fibrosis Lung Infections: Polymicrobial, Complex, and Hard to Treat
@en
Cystic Fibrosis Lung Infections: Polymicrobial, Complex, and Hard to Treat
@nl
P2860
P3181
P1433
P1476
Cystic Fibrosis Lung Infections: Polymicrobial, Complex, and Hard to Treat
@en
P2093
George A O'Toole
Laura M Filkins
P2860
P304
P3181
P356
10.1371/JOURNAL.PPAT.1005258
P407
P5008
P577
2015-12-01T00:00:00Z