The Nrf2/HO-1 Axis in Cancer Cell Growth and Chemoresistance - Wikidata - awgldk - TriplyDB

The Nrf2/HO-1 Axis in Cancer Cell Growth and Chemoresistance

The Nrf2/HO-1 Axis in Cancer Cell Growth and Chemoresistance

http://www.wikidata.org/entity/Q26772298

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Redox Homeostasis and Cellular Antioxidant Systems: Crucial Players in Cancer Growth and Therapy Clinical significance of T cell metabolic reprogramming in cancer The Influence of the Gut Microbiota on Host Physiology: In Pursuit of Mechanisms Antioxidant Functions of the Aryl Hydrocarbon Receptor Macrophage Migration Inhibitory Factor as an Emerging Drug Target to Regulate Antioxidant Response Element System The role of quercetin and vitamin C in Nrf2-dependent oxidative stress production in breast cancer cells IDH1 R132H mutation regulates glioma chemosensitivity through Nrf2 pathway.Redox signaling mediated by the gut microbiota.Role of Nrf2, HO-1 and GSH in Neuroblastoma Cell Resistance to Bortezomib Upregulation of Heme Oxygenase-1 in Response to Wild Thyme Treatment Protects against Hypertension and Oxidative Stress The aqueous extract of Brucea javanica suppresses cell growth and alleviates tumorigenesis of human lung cancer cells by targeting mutated epidermal growth factor receptor.The Ratio of Hmox1/Nrf2 mRNA Level in the Tumor Tissue Is a Predictor of Distant Metastasis in Colorectal Cancer Growth inhibitory effect of β-eudesmol on cholangiocarcinoma cells and its potential suppressive effect on heme oxygenase-1 production, STAT1/3 activation, and NF-κB downregulation.ABCF2, an Nrf2 target gene, contributes to cisplatin resistance in ovarian cancer cells.Esculetin induces antiproliferative and apoptotic response in pancreatic cancer cells by directly binding to KEAP1.Inhibition of malic enzyme 1 disrupts cellular metabolism and leads to vulnerability in cancer cells in glucose-restricted conditions.p62/SQSTM1-Dr. Jekyll and Mr. Hyde that prevents oxidative stress but promotes liver cancer.HO-1 Induction in Cancer Progression: A Matter of Cell Adaptation.Activation of Nrf2 Signaling Augments Vesicular Stomatitis Virus Oncolysis via Autophagy-Driven Suppression of Antiviral Immunity.Co-Activation of PKC-δ by CRIF1 Modulates Oxidative Stress in Bone Marrow Multipotent Mesenchymal Stromal Cells after Irradiation by Phosphorylating NRF2 Ser40.Abnormal expression of Nrf2 may play an important role in the pathogenesis and development of adenomyosis.Perspectives of the Nrf-2 signaling pathway in cancer progression and therapy.Red Yeast Rice Protects Circulating Bone Marrow-Derived Proangiogenic Cells against High-Glucose-Induced Senescence and Oxidative Stress: The Role of Heme Oxygenase-1.The Role of Genetic Polymorphisms in Nrf2 and P73 in Egyptian Women with Breast Cancer Establishment and characterization of human osteosarcoma cells resistant to pyropheophorbide-α methyl ester-mediated photodynamic therapy.Bioengineered NRF2-siRNA Is Effective to Interfere with NRF2 Pathways and Improve Chemosensitivity of Human Cancer Cells.ROS Modulator Molecules with Therapeutic Potential in Cancers Treatments.Aldose reductase inhibitor, fidarestat regulates mitochondrial biogenesis via Nrf2/HO-1/AMPK pathway in colon cancer cells.Roles of LPA receptor signaling in breast cancer.microRNA-494 Favors HO-1 Expression in Neuroblastoma Cells Exposed to Oxidative Stress in a Bach1-Independent Way.

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The Nrf2/HO-1 Axis in Cancer Cell Growth and Chemoresistance

http://www.wikidata.org/entity/Q26772298

description

2015 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

@hyw

2015 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2016 nî lūn-bûn

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2016年の論文

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2016年論文

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2016年論文

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2016年論文

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2016年論文

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2016年論文

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2016年论文

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name

The Nrf2/HO-1 Axis in Cancer Cell Growth and Chemoresistance

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The Nrf2/HO-1 Axis in Cancer Cell Growth and Chemoresistance

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The Nrf2/HO-1 Axis in Cancer Cell Growth and Chemoresistance

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The Nrf2/HO-1 Axis in Cancer Cell Growth and Chemoresistance

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The Nrf2/HO-1 Axis in Cancer Cell Growth and Chemoresistance

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The Nrf2/HO-1 Axis in Cancer Cell Growth and Chemoresistance

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The Nrf2/HO-1 Axis in Cancer Cell Growth and Chemoresistance

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The Nrf2/HO-1 Axis in Cancer Cell Growth and Chemoresistance

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The Nrf2/HO-1 Axis in Cancer Cell Growth and Chemoresistance

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Oxidative medicine and cellular longevity

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The Nrf2/HO-1 Axis in Cancer Cell Growth and Chemoresistance

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The selective autophagy substrate p62 activates the stress responsive transcription factor Nrf2 through inactivation of Keap1

DJ-1, a cancer- and Parkinson's disease-associated protein, stabilizes the antioxidant transcriptional master regulator Nrf2

DJ-1 has a role in antioxidative stress to prevent cell death

p62/SQSTM1 is a target gene for transcription factor NRF2 and creates a positive feedback loop by inducing antioxidant response element-driven gene transcription

Isolation of NF-E2-related factor 2 (Nrf2), a NF-E2-like basic leucine zipper transcriptional activator that binds to the tandem NF-E2/AP1 repeat of the beta-globin locus control region

Oxidative stress sensor Keap1 functions as an adaptor for Cul3-based E3 ligase to regulate proteasomal degradation of Nrf2

Upsides and downsides of reactive oxygen species for cancer: the roles of reactive oxygen species in tumorigenesis, prevention, and therapy

Nrf2 regulates haematopoietic stem cell function

Keap1 represses nuclear activation of antioxidant responsive elements by Nrf2 through binding to the amino-terminal Neh2 domain

Direct interaction between Nrf2 and p21(Cip1/WAF1) upregulates the Nrf2-mediated antioxidant response

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