Transforming growth factor-beta1 in inflammatory airway disease: a key for understanding inflammation and remodeling - Wikidata - awgldk - TriplyDB

Transforming growth factor-beta1 in inflammatory airway disease: a key for understanding inflammation and remodeling

Transforming growth factor-beta1 in inflammatory airway disease: a key for understanding inflammation and remodeling

http://www.wikidata.org/entity/Q26823406

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Intermittent or sustained systemic inflammation and the preterm brain Versican in inflammation and tissue remodeling: the impact on lung disorders Kaempferol Inhibits Endoplasmic Reticulum Stress-Associated Mucus Hypersecretion in Airway Epithelial Cells And Ovalbumin-Sensitized Mice Immunomodulatory oligonucleotide IMT504: Effects on mesenchymal stem cells as a first-in-class immunoprotective/immunoregenerative therapy Fibrosis-related biomarkers and risk of total and cause-specific mortality: the cardiovascular health study TGF- β: an important mediator of allergic disease and a molecule with dual activity in cancer development.Identification and association of the single nucleotide polymorphisms, C-509T, C+466T and T+869C, of the TGF-β1 gene in patients with asthma and their influence on the mRNA expression level of TGF-β1 A novel human anti-VCAM-1 monoclonal antibody ameliorates airway inflammation and remodelling.ICON: chronic rhinosinusitis.Key mediators in the immunopathogenesis of allergic asthma Tgf-β1 inhibits Cftr biogenesis and prevents functional rescue of ΔF508-Cftr in primary differentiated human bronchial epithelial cells.Hypoxia down-regulates expression of secretory leukocyte protease inhibitor in bronchial epithelial cells via TGF-β1 Involvement of B2 receptor in bradykinin-induced proliferation and proinflammatory effects in human nasal mucosa-derived fibroblasts isolated from chronic rhinosinusitis patients.Differential Expression and Release of Activin A and Follistatin in Chronic Rhinosinusitis with and without Nasal Polyps Prevention of Influenza Virus-Induced Immunopathology by TGF-β Produced during Allergic Asthma.Mast cells in airway diseases and interstitial lung disease Low-dose memantine attenuated methadone dose in opioid-dependent patients: a 12-week double-blind randomized controlled trial.Immunization against TGF-β1 reduces collagen deposition but increases sustained inflammation in a murine asthma model Repair and Remodeling of airway epithelium after injury in Chronic Obstructive Pulmonary Disease A HuR/TGF-β1 feedback circuit regulates airway remodeling in airway smooth muscle cells.Dendritic cells in the periphery control antigen-specific natural and induced regulatory T cells.Transforming growth factor-beta 1 pathways in inflammatory airway diseases.Disease-modifying genes and monogenic disorders: experience in cystic fibrosis.Regulatory T cells and immune regulation of allergic diseases: roles of IL-10 and TGF-β.Transforming growth factor-β superfamily in obstructive lung diseases. more suspects than TGF-β alone.Immunopathology of chronic rhinosinusitis.Cellular and molecular mechanisms of chronic rhinosinusitis and potential therapeutic strategies: review on cytokines, nuclear factor kappa B and transforming growth factor beta.Subtyping Chronic Obstructive Pulmonary Disease Using Peripheral Blood Proteomics.Chronic inflammatory airway diseases: the central role of the epithelium revisited.Fibrotic Tissue and Middle Turbinate Exhibit Similar Mechanical Properties. Is Fibrosis a Solution in Nasal Polyposis?Mechanisms of immune regulation in allergic diseases: the role of regulatory T and B cells.Neuropilin-1 Is Expressed on Lymphoid Tissue Residing LTi-like Group 3 Innate Lymphoid Cells and Associated with Ectopic Lymphoid Aggregates Vitamin D attenuates myofibroblast differentiation and extracellular matrix accumulation in nasal polyp-derived fibroblasts through smad2/3 signaling pathway.The role of TGFβ‑HGF‑Smad4 axis in regulating the proliferation of mouse airway progenitor cells.Immunoregulatory effects of bone marrow-derived mesenchymal stem cells in the nasal polyp microenvironment.Autophagy and airway fibrosis: Is there a link?AVE 0991, a non-peptide mimic of angiotensin-(1-7) effects, attenuates pulmonary remodelling in a model of chronic asthma.Systems approaches to modeling chronic mucosal inflammation.Tolerogenic CX3CR1+ B cells suppress food allergy-induced intestinal inflammation in mice.Increased epidermal growth factor in nasopharyngeal aspirates from infants with recurrent wheeze.

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Transforming growth factor-beta1 in inflammatory airway disease: a key for understanding inflammation and remodeling

http://www.wikidata.org/entity/Q26823406

description

2012 nî lūn-bûn

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2012 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

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2012 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2012年の論文

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2012年論文

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2012年論文

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2012年论文

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name

Transforming growth factor-bet ...... ng inflammation and remodeling

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Transforming growth factor-bet ...... ng inflammation and remodeling

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Transforming growth factor-bet ...... ng inflammation and remodeling

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Transforming growth factor-bet ...... ng inflammation and remodeling

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Transforming growth factor-bet ...... ng inflammation and remodeling

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Transforming growth factor-bet ...... ng inflammation and remodeling

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J.1398-9995.2012.02880.X

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Transforming growth factor-bet ...... ng inflammation and remodeling

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C. Bachert

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G. H. Hu

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K. Van Crombruggen

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S. L. Hong

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P2860

Anti-IL-5 treatment reduces deposition of ECM proteins in the bronchial subepithelial basement membrane of mild atopic asthmatics.

Targeting lung inflammation: novel therapies for the treatment of COPD.

Inhibition of airway remodeling in IL-5-deficient mice.

Naturally occurring lung CD4(+)CD25(+) T cell regulation of airway allergic responses depends on IL-10 induction of TGF-beta.

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1193-202

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