The role of gasotransmitters NO, H2S and CO in myocardial ischaemia/reperfusion injury and cardioprotection by preconditioning, postconditioning and remote conditioning - Wikidata - awgldk - TriplyDB

The role of gasotransmitters NO, H2S and CO in myocardial ischaemia/reperfusion injury and cardioprotection by preconditioning, postconditioning and remote conditioning

The role of gasotransmitters NO, H2S and CO in myocardial ischaemia/reperfusion injury and cardioprotection by preconditioning, postconditioning and remote conditioning

http://www.wikidata.org/entity/Q26825733

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Protection by the NO-Donor SNAP and BNP against Hypoxia/Reoxygenation in Rat Engineered Heart Tissue Subcellular Energetics and Metabolism: Potential Therapeutic Applications.Cardiac NO signalling in the metabolic syndrome.Ischaemic preconditioning preferentially increases protein S-nitrosylation in subsarcolemmal mitochondria Role of Hydrogen Sulfide in Ischemia-Reperfusion Injury.Association between Daily Hydrogen Sulfide Exposure and Incidence of Emergency Hospital Visits: A Population-Based Study A combined experimental and DFT investigation on the structure and CO-releasing properties of mono and binuclear fac-Re(I)(CO)3 complexes with 5-pyridin-2-ylmethylene-amino uracils.Specific Mechanisms Underlying Right Heart Failure: The Missing Upregulation of Superoxide Dismutase-2 and Its Decisive Role in Antioxidative Defense.Hydrogen sulfide and PKG in ischemia-reperfusion injury: sources, signaling, accelerators and brakes.Pre- and posttreatment with hydrogen sulfide prevents ventilator-induced lung injury by limiting inflammation and oxidation.Additive cardioprotection by pharmacological postconditioning with hydrogen sulfide and nitric oxide donors in mouse heart: S-sulfhydration vs. S-nitrosylation.Exogenous Nitric Oxide Protects Human Embryonic Stem Cell-Derived Cardiomyocytes against Ischemia/Reperfusion Injury Reciprocal regulation of eNOS, H2S and CO-synthesizing enzymes in human atheroma: Correlation with plaque stability and effects of simvastatin.The application of remote ischemic conditioning in cardiac surgery Crosstalk of mitochondria with NADPH oxidase via reactive oxygen and nitrogen species signalling and its role for vascular function.Nitrite-Nitric Oxide Signaling and Cardioprotection.Gasotransmitter Heterocellular Signaling.Effect of hypercholesterolaemia on myocardial function, ischaemia-reperfusion injury and cardioprotection by preconditioning, postconditioning and remote conditioning.New aspects of p66Shc in ischaemia reperfusion injury and other cardiovascular diseases.Antioxidant effects of resveratrol in the cardiovascular system.Targeting the NO/superoxide ratio in adipose tissue: relevance to obesity and diabetes management.Targeting vascular (endothelial) dysfunction.Targeting the ROS-HIF-1-endothelin axis as a therapeutic approach for the treatment of obstructive sleep apnea-related cardiovascular complications.Utility of Cardiac Troponin to Predict Drug Overdose Mortality.Carbon disulfide. Just toxic or also bioregulatory and/or therapeutic?Redox regulation of gasotransmission in the vascular system: A focus on angiogenesis.Carbon monoxide promotes gastric wound healing in mice via the protein kinase C pathway.Anti-apoptotic properties of carbon monoxide in porcine oocyte during in vitro aging Effects of Remote Ischemic Preconditioning on Heme Oxygenase-1 Expression and Cutaneous Wound Repair.Uncaging carbon disulfide. Delivery platforms for potential pharmacological applications: a mechanistic approach.Pharmacology of the 'gasotransmitters' NO, CO and H2S: translational opportunities.Multiple mechanisms mediating carbon monoxide inhibition of the voltage-gated K+ channel Kv1.5.Pre- and postconditioning the heart with hydrogen sulfide (H2S) against ischemia/reperfusion injury in vivo: a systematic review and meta-analysis.Gasotransmitter hydrogen sulfide signaling in neuronal health and disease.Precision gas therapy using intelligent nanomedicine.Role of inducible nitric oxide synthase in myocardial ischemia-reperfusion injury in sleep-deprived rats.International Union of Basic and Clinical Pharmacology. CII: Pharmacological Modulation of H2S Levels: H2S Donors and H2S Biosynthesis Inhibitors.AP39, a mitochondria-targeting hydrogen sulfide (H2 S) donor, protects against myocardial reperfusion injury independently of salvage kinase signalling.Exposure to cigarette smoke abrogates the beneficial effect of ischemic postconditioning.NO Signaling in the Cardiovascular System and Exercise.

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The role of gasotransmitters NO, H2S and CO in myocardial ischaemia/reperfusion injury and cardioprotection by preconditioning, postconditioning and remote conditioning

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2015 nî lūn-bûn

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Efstathios K Iliodromitis

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Ioanna Andreadou

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Péter Ferdinandy

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Tienush Rassaf

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P2860

Hydrogen sulfide and nitric oxide are mutually dependent in the regulation of angiogenesis and endothelium-dependent vasorelaxation

Nitric oxide and peroxynitrite in health and disease

Hydrogen sulfide mediates the vasoactivity of garlic

Interaction of risk factors, comorbidities, and comedications with ischemia/reperfusion injury and cardioprotection by preconditioning, postconditioning, and remote conditioning

The IUPHAR/BPS Guide to PHARMACOLOGY: an expert-driven knowledgebase of drug targets and their ligands

Potential cardioprotective actions of no-releasing aspirin

Hydrogen sulfide is an endogenous modulator of leukocyte-mediated inflammation

Cardioprotective effects of nitric oxide-aspirin in myocardial ischemia-reperfused rats

Anti-apoptotic and anti-inflammatory effects of hydrogen sulfide in a rat model of regional myocardial I/R

The therapeutic potential of carbon monoxide

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10.1111/BPH.12811

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Andreas Papapetropoulos

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reperfusion injury

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4369266

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