Breast cancer epithelial-to-mesenchymal transition: examining the functional consequences of plasticity
about
Aggressiveness Niche: Can It Be the Foster Ground for Cancer Metastasis Precursors?Stem cell technology in breast cancer: current status and potential applicationsEpithelial-to-endothelial transition and cancer stem cells: two cornerstones of vasculogenic mimicry in malignant tumorsDesmoplasia and chemoresistance in pancreatic cancerThe Roles of Epithelial-to-Mesenchymal Transition (EMT) and Mesenchymal-to-Epithelial Transition (MET) in Breast Cancer Bone Metastasis: Potential Targets for Prevention and TreatmentIntratumoral heterogeneity: Clonal cooperation in epithelial-to-mesenchymal transition and metastasisPy2T murine breast cancer cells, a versatile model of TGFβ-induced EMT in vitro and in vivoStructure-function analyses of the human SIX1–EYA2 complex reveal insights into metastasis and BOR syndromeFibronectin induces epithelial-mesenchymal transition in human breast cancer MCF-7 cells via activation of calpainEMT cells increase breast cancer metastasis via paracrine GLI activation in neighbouring tumour cells.E-cadherin loss alters cytoskeletal organization and adhesion in non-malignant breast cells but is insufficient to induce an epithelial-mesenchymal transition.p66ShcA promotes breast cancer plasticity by inducing an epithelial-to-mesenchymal transition.miR-200c targets a NF-κB up-regulated TrkB/NTF3 autocrine signaling loop to enhance anoikis sensitivity in triple negative breast cancerBeta-elemene blocks epithelial-mesenchymal transition in human breast cancer cell line MCF-7 through Smad3-mediated down-regulation of nuclear transcription factors.Loss of N-Myc interactor promotes epithelial-mesenchymal transition by activation of TGF-β/SMAD signaling.Modeling TGF-β in early stages of cancer tissue dynamics.Special AT-rich sequence-binding protein-1 participates in the maintenance of breast cancer stem cells through regulation of the Notch signaling pathway and expression of Snail1 and Twist1.Do circulating tumor cells, exosomes, and circulating tumor nucleic acids have clinical utility? A report of the association for molecular pathologyTWIST1-Induced miR-424 Reversibly Drives Mesenchymal Programming while Inhibiting Tumor Initiation.Loss of giant obscurins from breast epithelium promotes epithelial-to-mesenchymal transition, tumorigenicity and metastasis.Mathematical Modelling of Molecular Pathways Enabling Tumour Cell Invasion and Migration.CX3CL1 promotes breast cancer via transactivation of the EGF pathwayCancer drug pan-resistance: pumps, cancer stem cells, quiescence, epithelial to mesenchymal transition, blocked cell death pathways, persisters or what?DDR2 facilitates hepatocellular carcinoma invasion and metastasis via activating ERK signaling and stabilizing SNAIL1Targeting triple-negative breast cancer cells with the histone deacetylase inhibitor panobinostat.The Alternative Splicing of Cytoplasmic Polyadenylation Element Binding Protein 2 Drives Anoikis Resistance and the Metastasis of Triple Negative Breast Cancer.Clinically relevant morphological structures in breast cancer represent transcriptionally distinct tumor cell populations with varied degrees of epithelial-mesenchymal transition and CD44+CD24- stemness.HRD1 suppresses the growth and metastasis of breast cancer cells by promoting IGF-1R degradation.EZH2 inhibition decreases p38 signaling and suppresses breast cancer motility and metastasisPrimary cancer cell culture: mammary-optimized vs conditional reprogramming.The collagen receptor discoidin domain receptor 2 stabilizes SNAIL1 to facilitate breast cancer metastasisNodal promotes invasive phenotypes via a mitogen-activated protein kinase-dependent pathway.A novel role for p115RhoGEF in regulation of epithelial plasticity.Critical research gaps and translational priorities for the successful prevention and treatment of breast cancer.Fatty acid synthase mediates the epithelial-mesenchymal transition of breast cancer cells.MicroRNA-510 promotes cell and tumor growth by targeting peroxiredoxin1 in breast cancer.A mammosphere formation RNAi screen reveals that ATG4A promotes a breast cancer stem-like phenotype.DDR2 overexpression in urothelial carcinoma indicates an unfavorable prognosis: a large cohort study.Functional roles of alternative splicing factors in human disease.Cytokeratin 18 is necessary for initiation of TGF-β1-induced epithelial-mesenchymal transition in breast epithelial cells.
P2860
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P2860
Breast cancer epithelial-to-mesenchymal transition: examining the functional consequences of plasticity
description
2011 nî lūn-bûn
@nan
2011 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Breast cancer epithelial-to-me ...... nal consequences of plasticity
@ast
Breast cancer epithelial-to-me ...... nal consequences of plasticity
@en
Breast cancer epithelial-to-me ...... nal consequences of plasticity
@nl
type
label
Breast cancer epithelial-to-me ...... nal consequences of plasticity
@ast
Breast cancer epithelial-to-me ...... nal consequences of plasticity
@en
Breast cancer epithelial-to-me ...... nal consequences of plasticity
@nl
prefLabel
Breast cancer epithelial-to-me ...... nal consequences of plasticity
@ast
Breast cancer epithelial-to-me ...... nal consequences of plasticity
@en
Breast cancer epithelial-to-me ...... nal consequences of plasticity
@nl
P2860
P3181
P356
P1476
Breast cancer epithelial-to-me ...... nal consequences of plasticity
@en
P2093
David J Drasin
Heide L Ford
P2860
P2888
P3181
P356
10.1186/BCR3037
P407
P577
2011-11-01T00:00:00Z
P5875
P6179
1003277853