Food reward functions as affected by obesity and bariatric surgery - Wikidata - awgldk - TriplyDB

Food reward functions as affected by obesity and bariatric surgery

Food reward functions as affected by obesity and bariatric surgery

http://www.wikidata.org/entity/Q26861264

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The central GLP-1: implications for food and drug reward Roux-en-Y gastric bypass: effects on feeding behavior and underlying mechanisms Obese patients after gastric bypass surgery have lower brain-hedonic responses to food than after gastric banding.Gut-Brain Cross-Talk in Metabolic Control.Neural responsivity to food cues in fasted and fed states pre and post gastric bypass surgery Reversible hyperphagia and obesity in rats with gastric bypass by central MC3/4R blockade.Effect of diet on preference and intake of sucrose in obese prone and resistant rats.Metabolic adaptation following massive weight loss is related to the degree of energy imbalance and changes in circulating leptin.Striatal dopamine homeostasis is altered in mice following Roux-en-Y gastric bypass surgery.Bariatric surgery: prevalence, predictors, and mechanisms of diabetes remission.Changes in microglial activation within the hindbrain, nodose ganglia, and the spinal cord following subdiaphragmatic vagotomy.Clinical review: Regulation of food intake, energy balance, and body fat mass: implications for the pathogenesis and treatment of obesity Roux-en-Y gastric bypass surgery of morbidly obese patients induces swift and persistent changes of the individual gut microbiota Appetite and body weight regulation after bariatric surgery.Biological mechanisms that promote weight regain following weight loss in obese humans Roux-en-Y gastric bypass surgery is effective in fibroblast growth factor-21 deficient mice.Effects of preoperative exposure to a high-fat versus a low-fat diet on ingestive behavior after gastric bypass surgery in rats.A comparison of functional brain changes associated with surgical versus behavioral weight loss.Current and emerging concepts on the role of peripheral signals in the control of food intake and development of obesity.Lipids and obesity: Also a matter of taste?Hedonic Changes in Food Choices Following Roux-en-Y Gastric Bypass.Ghrelin, CCK, GLP-1, and PYY(3-36): Secretory Controls and Physiological Roles in Eating and Glycemia in Health, Obesity, and After RYGB.Are peptide conjugates the golden therapy against obesity?

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Food reward functions as affected by obesity and bariatric surgery

http://www.wikidata.org/entity/Q26861264

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2011 nî lūn-bûn

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2011 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

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2011 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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Food reward functions as affected by obesity and bariatric surgery

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Food reward functions as affected by obesity and bariatric surgery

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Food reward functions as affected by obesity and bariatric surgery

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Food reward functions as affected by obesity and bariatric surgery

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Food reward functions as affected by obesity and bariatric surgery

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International Journal of Obesity

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Food reward functions as affected by obesity and bariatric surgery

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A C Shin

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H-R Berthoud

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Obesity exacerbates chemically induced neurodegeneration

Ghrelin controls hippocampal spine synapse density and memory performance

Evidence for sugar addiction: behavioral and neurochemical effects of intermittent, excessive sugar intake

Ghrelin modulates the activity and synaptic input organization of midbrain dopamine neurons while promoting appetite

IL-6 mediated degeneration of forebrain GABAergic interneurons and cognitive impairment in aged mice through activation of neuronal NADPH oxidase

Reward deficiency syndrome: a biogenetic model for the diagnosis and treatment of impulsive, addictive, and compulsive behaviors

Leptin receptor signaling in midbrain dopamine neurons regulates feeding

Genetically determined differences in brain response to a primary food reward.

Meal-induced hormone responses in a rat model of Roux-en-Y gastric bypass surgery.

Ghrelin administration into tegmental areas stimulates locomotor activity and increases extracellular concentration of dopamine in the nucleus accumbens.

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10.1038/IJO.2011.147

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35 Suppl 3

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2011-09-01T00:00:00Z

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221934083

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bariatric surgery

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