Programmed necrosis in acute kidney injury - Wikidata - awgldk - TriplyDB

Programmed necrosis in acute kidney injury

Programmed necrosis in acute kidney injury

http://www.wikidata.org/entity/Q26865577

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TWEAK and the progression of renal disease: clinical translation Role of necroptosis in the pathogenesis of solid organ injury The restrained expression of NF-kB in renal tissue ameliorates folic acid induced acute kidney injury in mice A Role for Tubular Necroptosis in Cisplatin-Induced AKI Calpain: a molecule to induce AIF-mediated necroptosis in RGC-5 following elevated hydrostatic pressure Autophagy and its link to type II diabetes mellitus Baicalin ameliorates H2O2 induced cytotoxicity in HK-2 cells through the inhibition of ER stress and the activation of Nrf2 signaling Magnolin protects against contrast-induced nephropathy in rats via antioxidation and antiapoptosis.Regulated cell death in AKI.Adhesion of annexin 7 deficient erythrocytes to endothelial cells A combinatorial approach of Proteomics and Systems Biology in unravelling the mechanisms of acute kidney injury (AKI): involvement of NMDA receptor GRIN1 in murine AKI.P2X7 receptor inhibition protects against ischemic acute kidney injury in mice.miR-21 contributes to xenon-conferred amelioration of renal ischemia-reperfusion injury in mice.Renal telocytes contribute to the repair of ischemically injured renal tubules TRPC6 May Protect Renal Ischemia-Reperfusion Injury Through Inhibiting Necroptosis of Renal Tubular Epithelial Cells Tumor Necrosis Factor-α: Life and Death of Hepatocytes During Liver Ischemia/Reperfusion Injury.Two independent pathways of regulated necrosis mediate ischemia-reperfusion injury Protein kinase C-α interaction with iHSP70 in mitochondria promotes recovery of mitochondrial function after injury in renal proximal tubular cells The RIP1-kinase inhibitor necrostatin-1 prevents osmotic nephrosis and contrast-induced AKI in mice.Tubular epithelial cells in renal clear cell carcinoma express high RIPK1/3 and show increased susceptibility to TNF receptor 1-induced necroptosis The protective effect of baicalin against renal ischemia-reperfusion injury through inhibition of inflammation and apoptosis.Extensive podocyte loss triggers a rapid parietal epithelial cell response.Progress in the development of animal models of acute kidney injury and its impact on drug discovery.Necroptosis in immunity and ischemia-reperfusion injury.Acute kidney injury in acute liver failure: a review.Immune system modulation of kidney regeneration--mechanisms and implications.Mechanisms of maladaptive repair after AKI leading to accelerated kidney ageing and CKD.Molecular determinants of acute kidney injury.Necroinflammation in Kidney Disease.The effects and regulatory mechanism of RIP3 on RGC-5 necroptosis following elevated hydrostatic pressure.The in vivo evidence for regulated necrosis.Resveratrol alleviates the cytotoxicity induced by the radiocontrast agent, ioxitalamate, by reducing the production of reactive oxygen species in HK-2 human renal proximal tubule epithelial cells in vitro Synergistic effect of apoptosis and necroptosis inhibitors in cisplatin-induced nephrotoxicity.Postischemic microvasculopathy and endothelial progenitor cell-based therapy in ischemic AKI: update and perspectives.Toxic response of graphene nanoplatelets in vivo and in vitro.Ischemia-reperfusion induces renal tubule pyroptosis via the CHOP-caspase-11 pathway.Ischemia/Reperfusion.Cathelicidin PR-39 peptide inhibits hypoxia/reperfusion-induced kidney cell apoptosis by suppression of the endoplasmic reticulum-stress pathway.Late intervention with the small molecule BB3 mitigates postischemic kidney injury.Minocycline and doxycycline, but not other tetracycline-derived compounds, protect liver cells from chemical hypoxia and ischemia/reperfusion injury by inhibition of the mitochondrial calcium uniporter.

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P2860

Programmed necrosis in acute kidney injury

http://www.wikidata.org/entity/Q26865577

description

2012 nî lūn-bûn

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2012 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

2012 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

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name

Programmed necrosis in acute kidney injury

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Programmed necrosis in acute kidney injury

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Programmed necrosis in acute kidney injury

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type

label

Programmed necrosis in acute kidney injury

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Programmed necrosis in acute kidney injury

@en

Programmed necrosis in acute kidney injury

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prefLabel

Programmed necrosis in acute kidney injury

@ast

Programmed necrosis in acute kidney injury

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Programmed necrosis in acute kidney injury

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Nephrology Dialysis Transplantation

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Programmed necrosis in acute kidney injury

@en

P2093

F. De Zen

http://www.w3.org/2001/XMLSchema#string

J. Weinberg

http://www.w3.org/2001/XMLSchema#string

S. Krautwald

http://www.w3.org/2001/XMLSchema#string

U. Kunzendorf

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P2860

Fas triggers an alternative, caspase-8-independent cell death pathway using the kinase RIP as effector molecule

Linear ubiquitination prevents inflammation and regulates immune signalling

Mixed lineage kinase domain-like protein mediates necrosis signaling downstream of RIP3 kinase

The mitochondrial phosphatase PGAM5 functions at the convergence point of multiple necrotic death pathways

cIAPs block Ripoptosome formation, a RIP1/caspase-8 containing intracellular cell death complex differentially regulated by cFLIP isoforms

Receptor interacting protein kinase-3 determines cellular necrotic response to TNF-alpha

Phosphorylation-driven assembly of the RIP1-RIP3 complex regulates programmed necrosis and virus-induced inflammation

Many stimuli pull the necrotic trigger, an overview

Cyclophilin D-dependent mitochondrial permeability transition regulates some necrotic but not apoptotic cell death

Loss of cyclophilin D reveals a critical role for mitochondrial permeability transition in cell death

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10.1093/NDT/GFS373

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9

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27

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Andreas Linkermann

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2012-09-01T00:00:00Z

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230784045

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22942173

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