New experimental models of diabetic nephropathy in mice models of type 2 diabetes: efforts to replicate human nephropathy - Wikidata - awgldk - TriplyDB

New experimental models of diabetic nephropathy in mice models of type 2 diabetes: efforts to replicate human nephropathy

New experimental models of diabetic nephropathy in mice models of type 2 diabetes: efforts to replicate human nephropathy

http://www.wikidata.org/entity/Q26996084

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Animal models of diabetic macrovascular complications: key players in the development of new therapeutic approaches Chemical Exchange Saturation Transfer (CEST) Agents: Quantum Chemistry and MRI.A novel mouse model of advanced diabetic kidney disease.Neural stem cells in the diabetic brain Balanced regulation of the CCN family of matricellular proteins: a novel approach to the prevention and treatment of fibrosis and cancer.Glomerulopathy in the KK.Cg-A(y) /J mouse reflects the pathology of diabetic nephropathy.The Extract of Litsea japonica Reduced the Development of Diabetic Nephropathy via the Inhibition of Advanced Glycation End Products Accumulation in db/db Mice.Tofogliflozin, a novel sodium-glucose co-transporter 2 inhibitor, improves renal and pancreatic function in db/db mice.Effect of insulin on ACE2 activity and kidney function in the non-obese diabetic mouse.Altered renal lipid metabolism and renal lipid accumulation in human diabetic nephropathy.Tubulointerstitial disease in diabetic nephropathy.Animal models of chronic kidney disease: useful but not perfect.Pharmacokinetic and pharmacodynamic drug evaluation of tofogliflozin for the treatment of type 2 diabetes.GS-E3D, a new pectin lyase-modified red ginseng extract, inhibited diabetes-related renal dysfunction in streptozotocin-induced diabetic rats.Sirt6 deficiency exacerbates podocyte injury and proteinuria through targeting Notch signaling.The effect of resveratrol on the expression of AdipoR1 in kidneys of diabetic nephropathy.Urine RAS components in mice and people with type 1 diabetes and chronic kidney disease.Rhein lysinate protects renal function in diabetic nephropathy of KK/HlJ mice.ORAI channels are critical for receptor-mediated endocytosis of albumin.Monitoring p53 by MDM2 and MDMX is required for endocrine pancreas development and function in a spatio-temporal manner.Combining streptozotocin and unilateral nephrectomy is an effective method for inducing experimental diabetic nephropathy in the 'resistant' C57Bl/6J mouse strain.Kidney disease models: tools to identify mechanisms and potential therapeutic targets.

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P2860

New experimental models of diabetic nephropathy in mice models of type 2 diabetes: efforts to replicate human nephropathy

http://www.wikidata.org/entity/Q26996084

description

2012 nî lūn-bûn

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2012 թուականի Փետրուարին հրատարակուած գիտական յօդուած

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2012 թվականի փետրվարին հրատարակված գիտական հոդված

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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name

New experimental models of dia ...... to replicate human nephropathy

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New experimental models of dia ...... to replicate human nephropathy

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New experimental models of dia ...... to replicate human nephropathy

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New experimental models of dia ...... to replicate human nephropathy

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New experimental models of dia ...... to replicate human nephropathy

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New experimental models of dia ...... to replicate human nephropathy

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Experimental Diabetes Research

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Daniel Batlle

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Marta Riera

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María José Soler

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Creative Commons Attribution 3.0 Unported

P2860

Mouse models of diabetic nephropathy.

Transgenic overexpression of GLUT1 in mouse glomeruli produces renal disease resembling diabetic glomerulosclerosis.

Diabetic kidney disease in the db/db mouse.

Prevention of diabetic nephropathy in db/db mice with glycated albumin antagonists. A novel treatment strategy.

Obese, a new mutation in the house mouse.

Phenotypes of mouse diabetes and rat fatty due to mutations in the OB (leptin) receptor.

Abnormal splicing of the leptin receptor in diabetic mice.

BTBR Ob/Ob mutant mice model progressive diabetic nephropathy.

Glomerular-specific alterations of VEGF-A expression lead to distinct congenital and acquired renal diseases.

Long-term prevention of renal insufficiency, excess matrix gene expression, and glomerular mesangial matrix expansion by treatment with monoclonal antitransforming growth factor-beta antibody in db/db diabetic mice.

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10.1155/2012/616313

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2012

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223990514

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22461787

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type-2 diabetes

genitourinary system

maturity-onset diabetes of the young

chronic kidney disease

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3291115

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