Pathophysiology and Treatments of Oxidative Injury in Ischemic Stroke: Focus on the Phagocytic NADPH Oxidase 2 - Wikidata - awgldk - TriplyDB

Pathophysiology and Treatments of Oxidative Injury in Ischemic Stroke: Focus on the Phagocytic NADPH Oxidase 2

Pathophysiology and Treatments of Oxidative Injury in Ischemic Stroke: Focus on the Phagocytic NADPH Oxidase 2

http://www.wikidata.org/entity/Q27005956

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Update on Inflammatory Biomarkers and Treatments in Ischemic Stroke Platelet-neutrophil interactions under thromboinflammatory conditions NADPH Oxidase Activity in Cerebral Arterioles Is a Key Mediator of Cerebral Small Vessel Disease-Implications for Prevention.5-HMF attenuates striatum oxidative damage via Nrf2/ARE signaling pathway following transient global cerebral ischemia Anti-Inflammatory Targets for the Treatment of Reperfusion Injury in Stroke.Down-regulation of NOX4 by betulinic acid protects against cerebral ischemia-reperfusion in mice.A single primary blast-induced traumatic brain injury in rodent model causes cell-type dependent increase in NADPH oxidase isoforms in vulnerable brain regions.Scutellarin protects oxygen/glucose-deprived astrocytes and reduces focal cerebral ischemic injury Mitochondrial targeting as a novel therapy for stroke

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Pathophysiology and Treatments of Oxidative Injury in Ischemic Stroke: Focus on the Phagocytic NADPH Oxidase 2

http://www.wikidata.org/entity/Q27005956

description

2015 nî lūn-bûn

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2015 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2015 թվականի օգոստոսին հրատարակված գիտական հոդված

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年论文

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Pathophysiology and Treatments ...... the Phagocytic NADPH Oxidase 2

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Antioxidants & Redox Signaling

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Pathophysiology and Treatments ...... the Phagocytic NADPH Oxidase 2

@en

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François Mach

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Nicolas Vuilleumier

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Priscila Camillo Teixeira

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P2860

Effect of antioxidant vitamin supplementation on cardiovascular outcomes: a meta-analysis of randomized controlled trials

Heart Disease and Stroke Statistics--2010 Update: A Report From the American Heart Association

Hydroxyl radical formation is greater in striatal core than in penumbra in a rat model of ischemic stroke

Binding of beta 2-glycoprotein I to anionic phospholipids facilitates processing and presentation of a cryptic epitope that activates pathogenic autoreactive T cells

p53 opens the mitochondrial permeability transition pore to trigger necrosis

Vitamin E therapy results in a reduction in HDL function in individuals with diabetes and the haptoglobin 2-1 genotype

Vitamins E and C in the prevention of cardiovascular disease in men: the Physicians' Health Study II randomized controlled trial

Sex differences in stroke recovery.

Oxidation-specific epitopes and immunological responses: Translational biotheranostic implications for atherosclerosis

Neuroprotection after stroke by targeting NOX4 as a source of oxidative stress

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460-89

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scholarly article

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10.1089/ARS.2013.5778

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5

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23

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Fabrizio Montecucco

Federico Carbone

Vincent Braunersreuther

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2015-08-10T00:00:00Z

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260872588

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24635113

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pathophysiology

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4545676

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